THE STUDY OF DIBARYON SYSTEMS WITH STRANGENESS ON CHIRAL QUARK MODEL

2005 ◽  
Vol 20 (01) ◽  
pp. 69-77
Author(s):  
HOURONG PANG ◽  
JIALUN PING ◽  
XIAOHUA WU

We expand Salamanca SU (2) chiral quark model to SU (3) one and calculate promising dibaryon candidates with strangeness S=-3,-6 in the framework of resonating group method. We find that, besides ΩΩ, the mass of NΩ state is about 23–38 MeV lower than its threshold, it might appear as a bound state or a narrow resonance. The effect of K and η exchanges on the masses of strangeness and nonstrangeness systems has been studied and found to be negligible for nonstrangeness systems. However this effect brings some changes (about tens of MeV) on the masses of strangeness systems. We have also studied the sensitivities of binding energy and root mean square radius to the mass of s-quark.

2014 ◽  
Vol 26 ◽  
pp. 1460071
Author(s):  
W. L. WANG ◽  
F. HUANG ◽  
Z. Y. ZHANG ◽  
B. S. ZOU

We report our recent work on a dynamical investigation of the [Formula: see text] and [Formula: see text] states in a chiral quark model. Our results show that the [Formula: see text] interaction is attractive, which consequently results in a [Formula: see text] bound state with the binding energy of about 5-42 MeV, unlike the case of [Formula: see text] state, which has a repulsive interaction and thus is unbound.


2010 ◽  
Vol 25 (16) ◽  
pp. 1325-1332 ◽  
Author(s):  
W. L. WANG ◽  
F. HUANG ◽  
Z. Y. ZHANG ◽  
F. LIU

The ωϕ states with spin S=0,1 and 2 are dynamically studied in a chiral SU(3) quark model by solving the resonating group method (RGM) equation. It is found that the interactions of ωϕ systems are attractive, while no ωϕ bound state or resonance state is obtained due to the insufficiency of the strength of ωϕ attractions.


2010 ◽  
Vol 19 (02) ◽  
pp. 281-289
Author(s):  
S. T. LV ◽  
H. Y. CAO ◽  
X. LU

With the resonating group method, we study the non-local Σ - N interaction and spin–isospin dependence of Σ - N in the chiral SU (3) quark model. Meanwhile, the binding energy of hypernucleus [Formula: see text] is calculated based on the non-local Σ - N interaction in the union of channels with (S = 0, T = 1/2), (S = 1, T = 1/2) and (S = 0, T = 3/2). The result shows that there theoretically exists bound state of [Formula: see text]. Furthermore, we analyze the effect of various potentials and relative motion energy between hyperon and core on the binding energy in the model.


2014 ◽  
Vol 26 ◽  
pp. 1460120 ◽  
Author(s):  
L. R. DAI ◽  
J. LIU ◽  
L. YUAN

The mixing of scalar mesons is introduced into the baryon-baryon system in the chiral SU(3) quark model to further dynamically investigate the Di-omega state by using the same parameters as those in reasonably describing the experimental hyperon-nucleon and nucleon-nucleon scattering data. Two different mixings of scalar mesons, the ideal mixing and 19° mixing, are discussed, and compared with no mixing. The results show that it is still deeply bound state if 19° mixing is adopted, the same as those of no mixing. However, for ideal mixing, the binding energy is reduced quite a lot, yet it is still a bound state.


2005 ◽  
Vol 14 (07) ◽  
pp. 995-1015 ◽  
Author(s):  
T. INOUE ◽  
V. E. LYUBOVITSKIJ ◽  
TH. GUTSCHE ◽  
AMAND FAESSLER

We study the recently discovered Θ+ baryon in the context of the perturbative chiral quark model. The basic configuration of the Θ+ is a pentaquark bound state, where the single particle wave functions are the ground state solutions of a confining potential. We classify the resulting pentaquark multiplets as the JP=1/2- and 3/2- flavor SU (3) antidecuplet. The full mass spectrum of the multiplets is determined by including the meson and gluon cloud contributions, which induce flavor SU (3) breaking. The resulting 3/2- antidecuplet is about 185 MeV heavier than the 1/2- one, mainly because of the semi-perturbative gluon effects. We assign the observed Θ+ baryon as a member of the 1/2- antidecuplet and discuss in particular its relation to the recent experimental signal for a Ξ-- baryon.


2012 ◽  
Vol 27 (27) ◽  
pp. 1250161 ◽  
Author(s):  
M. T. LI ◽  
W. L. WANG ◽  
Y. B. DONG ◽  
Z. Y. ZHANG

We perform a systematic study of the bound state problem of [Formula: see text] and [Formula: see text] systems by using effective interaction in our chiral quark model. Our results show that both the interactions of [Formula: see text] and [Formula: see text] states are attractive, which consequently result in [Formula: see text] and [Formula: see text] bound states.


2002 ◽  
Vol 11 (01) ◽  
pp. 67-70
Author(s):  
NAZIH EL-NOHY

The bases of the translation invariant shell model are used to construct the ground-state wave functions of 3 T , 5 He and 6 Li . For 3 T the bases used correspond to the number of quanta of excitation N up to ten. For 5 He and 6 Li the bases used correspond to the number of quanta of excitation N up to six. The model is applied to calculate the binding energy and the root mean square radius for 3 T , 5 He and 6 Li nuclei. The residual interactions used consist of central, tensor, spin-orbit and quadratic spin-orbit terms with Gaussian radial dependence. The parameters of these interactions are chosen in such away that they represent the long range attraction and the short range repulsion of nucleon interactions. It was found that this potential is more suitable for calculating the characteristics of these nuclei, and better than other potentials, such as our previous potentials which were represented by the parameters of long range attraction forces only. For 3T we obtained good agreement between calculated and experimental values of both the ground state binding energy and the root mean square radius. For 5 He and 6 Li nuclei we obtained an acceptable improvement with these calculations over other potentials.


2016 ◽  
Vol 15 ◽  
pp. CIN.S38044 ◽  
Author(s):  
Merlin Lopus ◽  
D. Meshach Paul ◽  
R. Rajasekaran

Tumor suppressor gene, STK11, encodes for serine-threonine kinase, which has a critical role in regulating cell growth and apoptosis. Mutations of the same lead to the inactivation of STK11, which eventually causes different types of cancer. In this study, we focused on identifying those driver mutations through analyzing structural variations of mutants, viz., D194N, E199K, L160P, and Y49D. Native and the mutants were analyzed to determine their geometrical deviations such as root-mean-square deviation, root-mean-square fluctuation, radius of gyration, potential energy, and solvent-accessible surface area using conformational sampling technique. Additionally, the global minimized structure of native and mutants was further analyzed to compute their intramolecular interactions and distribution of secondary structure. Subsequently, simulated thermal denaturation and docking studies were performed to determine their structural variations, which in turn alter the formation of active complex that comprises STK11, STRAD, and MO25. The deleterious effect of the mutants would result in a comparative loss of enzyme function due to variations in their binding energy pertaining to spatial conformation and flexibility. Hence, the structural variations in binding energy exhibited by the mutants, viz., D194N, E199K, L160P, and Y49D, to that of the native, consequently lead to pathogenesis.


2021 ◽  
Vol 21 (1) ◽  
Author(s):  
M. A. F. Yahaya ◽  
A. R. Abu Bakar ◽  
J. Stanslas ◽  
N. Nordin ◽  
M. Zainol ◽  
...  

Abstract Background Neuroinflammation has been identified to be the key player in most neurodegenerative diseases. If neuroinflammation is left to be unresolved, chronic neuroinflammation will be establish. Such situation is due to the overly-activated microglia which have the tendency to secrete an abundance amount of pro-inflammatory cytokines into the neuron microenvironment. The abundance of pro-inflammatory cytokines will later cause toxic and death to neurons. Toll-like receptor 4 (TLR4)/MD-2 complex found on the cell surface of microglia is responsible for the attachment of LPS and activation of nuclear factor-κB (NF-κB) downstream signalling pathway. Albeit vitexin has been shown to possess anti-inflammatory property, however, little is known on its ability to bind at the binding site of TLR4/MD-2 complex of microglia as well as to be an antagonist for LPS. Results The present study reveals that both vitexin and donepezil are able to bind at the close proximity of LPS binding site located at the TLR4/MD-2 complex with the binding energy of − 4.35 and − 9.14 kcal/mol, respectively. During molecular dynamic simulations, both vitexin and donepezil formed stable complex with TLR4/MD-2 throughout the 100 ns time length with the root mean square deviation (RMSD) values of 2.5 Å and 4.0 Å, respectively. The root mean square fluctuation (RMSF) reveals that both compounds are stable. Interestingly, the radius of gyration (rGyr) for donepezil shows notable fluctuations when compare with vitexin. The MM-GBSA results showed that vitexin has higher binding energy in comparison with donepezil. Conclusions Taken together, the findings suggest that vitexin is able to bind at the binding site of TLR4/MD-2 complex with more stability than donepezil throughout the course of 100 ns simulation. Hence, vitexin has the potential to be an antagonist candidate for LPS.


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