How Robust Is the p Factor? Using Multitrait-Multimethod Modeling to Inform the Meaning of General Factors of Youth Psychopathology

2021 ◽  
pp. 216770262110551
Author(s):  
Ashley L. Watts ◽  
Bridget A. Makol ◽  
Isabella M. Palumbo ◽  
Andres De Los Reyes ◽  
Thomas M. Olino ◽  
...  

We used multitrait-multimethod (MTMM) modeling to examine general factors of psychopathology in three samples of youths ( Ns = 2,119, 303, and 592) for whom three informants reported on the youth’s psychopathology (e.g., child, parent, teacher). Empirical support for the p-factor diminished in multi-informant models compared with mono-informant models: The correlation between externalizing and internalizing factors decreased, and the general factor in bifactor models essentially reflected externalizing. Widely used MTMM-informed approaches for modeling multi-informant data cannot distinguish between competing interpretations of the patterns of effects we observed, including that the p factor reflects, in part, evaluative consistency bias or that psychopathology manifests differently across contexts (e.g., home vs. school). Ultimately, support for the p factor may be stronger in mono-informant designs, although it does not entirely vanish in multi-informant models. Instead, the general factor of psychopathology in any given mono-informant model likely reflects a complex mix of variances, some substantive and some methodological.

2021 ◽  
Author(s):  
Ashley L. Watts ◽  
Bridget Makol ◽  
Isabella Palumbo ◽  
Andres De Los Reyes ◽  
Thomas M Olino ◽  
...  

We used multitrait-multimethod (MTMM) modeling to examine general factors of psychopathology in three samples of youth (ns = 2119, 303, 592) for whom three informants reported on the youth’s psychopathology (e.g., child, parent, teacher). Empirical support for the p-factor diminished in multi-informant models compared with mono-informant models: the correlation between externalizing and internalizing factors decreased and the general factor in bifactor models essentially reflected externalizing. Widely used MTMM-informed approaches for modeling multi-informant data cannot distinguish between competing interpretations of the patterns of effects we observed, including that the p-factor reflects, in part, evaluative consistency bias or that psychopathology manifests differently across contexts (e.g., home vs. school). Ultimately, support for the p-factor may be stronger in mono-informant designs, although it is does not entirely vanish in multi-informant models. Instead, the general factor of psychopathology in any given mono-informant model likely reflects a complex mix of variances, some substantive and some methodological.


2019 ◽  
Author(s):  
Ashley L. Watts ◽  
Holly Poore ◽  
Irwin Waldman

We advanced several “riskier tests” of the validity of bifactor models of psychopathology, which included that the general and specific factors should be reliable and well-represented by their indicators, and that including a general factor should improve the correlated factor model’s external validity. We compared bifactor and correlated factors models using data from a community sample of youth (N=2498) whose parents provided ratings on psychopathology and external criteria (i.e., temperament, aggression, antisociality). Bifactor models tended to yield either general or specific factors that were unstable and difficult to interpret. The general factor appeared to reflect a differentially-weighted amalgam of psychopathology rather than a liability for psychopathology broadly construed. With rare exceptions, bifactor models did not explain additional variance in psychopathology symptom dimensions or external criteria compared with correlated factors models. Together, our findings call into question the validity of bifactor models of psychopathology, and the p-factor more broadly.


2020 ◽  
Vol 16 (1) ◽  
pp. 75-98 ◽  
Author(s):  
Gregory T. Smith ◽  
Emily A. Atkinson ◽  
Heather A. Davis ◽  
Elizabeth N. Riley ◽  
Joshua R. Oltmanns

An important advance in understanding and defining mental disorders has been the development of empirical approaches to mapping dimensions of dysfunction and their interrelatedness. Such empirical approaches have consistently observed intercorrelations among the many forms of psychopathology, leading to the identification of a general factor of psychopathology (the p factor). In this article, we review empirical support for p, including evidence for the stability and criterion validity of p. Further, we discuss the strong relationship between p and both the general factor of personality and the general factor of personality disorder, substantive interpretations of p, and the potential clinical utility of p. We posit that proposed substantive interpretations of p do not explain the full range of symptomatology typically included in p. The most plausible explanation is that p represents an index of impairment that has the potential to inform the duration and intensity of a client's mental health treatment.


2019 ◽  
Author(s):  
Cassandra M Brandes ◽  
Kathrin Herzhoff ◽  
Avante J Smack ◽  
Jennifer L Tackett

Research across age groups has consistently indicated that psychopathology has a general factor structure, such that there is a broad latent dimension (or p factor) capturing variance common to all mental disorders, as well as specific internalizing and externalizing factors. This research has found that the p factor overlaps substantially with trait negative emotionality (or neuroticism). However, less is known about the psychological substance of the specific factors of the general psychopathology model, or how lower-order facets of neuroticism may relate to each psychopathology factor. We investigated the structure of neuroticism and psychopathology, as well as associations between these domains in a sample of 695 pre-adolescent children using multi-method assessments. We found that both psychopathology and neuroticism may be well-characterized by bifactor models, and that there was substantial overlap between psychopathology (p) and neuroticism (n) general factors, as well as between specific factors (Internalizing with Fear, Externalizing with Irritability).


2019 ◽  
Vol 7 (6) ◽  
pp. 1266-1284 ◽  
Author(s):  
Cassandra M. Brandes ◽  
Kathrin Herzhoff ◽  
Avanté J. Smack ◽  
Jennifer L. Tackett

Research across age groups has consistently indicated that psychopathology has a general factor structure such that a broad latent dimension (or p factor) captures variance common to all mental disorders as well as specific internalizing and externalizing factors. This research has found that the p factor overlaps substantially with trait negative emotionality (or neuroticism). However, less is known about the psychological substance of the specific factors of the general psychopathology model or how lower-order facets of neuroticism may relate to each psychopathology factor. We investigated the structure of neuroticism and psychopathology as well as associations between these domains using multimethod assessments in a sample of 695 preadolescent children. We found that both psychopathology and neuroticism may be well characterized by bifactor models and that there was substantial overlap between psychopathology (p) and neuroticism (n) general factors as well as between specific factors (Internalizing with Fear, Externalizing with Irritability).


Author(s):  
Darren Haywood ◽  
Frank D. Baughman ◽  
Barbara A. Mullan ◽  
Karen R. Heslop

Recently, structural models of psychopathology, that address the diagnostic stability and comorbidity issues of the traditional nosological approach, have dominated much of the psychopathology literature. Structural approaches have given rise to the p-factor, which is claimed to reflect an individual’s propensity toward all common psychopathological symptoms. Neurocognitive abilities are argued to be important to the development and maintenance of a wide range of disorders, and have been suggested as an important driver of the p-factor. However, recent evidence argues against p being an interpretable substantive construct, limiting conclusions that can be drawn from associations between p, the specific factors of a psychopathology model, and neurocognitive abilities. Here, we argue for the use of the S-1 bifactor approach, where the general factor is defined by neurocognitive abilities, to explore the association between neurocognitive performance and a wide range of psychopathological symptoms. We use simulation techniques to give examples of how S-1 bifactor models can be used to examine this relationship, and how the results can be interpreted.


2021 ◽  
Author(s):  
Ashley Lauren Greene ◽  
Ashley L. Watts ◽  
Miriam K. Forbes ◽  
Roman Kotov ◽  
Robert Krueger ◽  
...  

Confirmatory factor analysis (CFA) and its bifactor models are popular in empirical investigations of the factor structure of psychological constructs. CFA offers straightforward hypothesis testing but has notable pitfalls, such as the imposition of strict assumptions (i.e., simple structure) that obscure unmodeled complexity. Due to the limitations of bifactor CFAs, they have yielded anomalous results across samples and studies that suggest model misspecification (e.g., evaporating specific factors and unexpected loadings). We propose the use of exploratory factor analysis (EFA) to evaluate the structural validity of CFA solutions—either before or after the estimation of more restrictive CFA models—to (1) identify model misspecifications that may drive anomalous estimates and (2) confirm CFA models by examining whether hypothesized structures emerge with limited researcher input. We evaluated the degree to which predominant factor structures were invariant across contexts along the exploratory-confirmatory continuum and demonstrate how poor methodological choices can distort results and impede theory development. All models fit well, but there were numerous differences in replicability and substantive interpretability. Several similarities emerged between bifactor CFA and EFA models, including evidence of overextraction, the collapse of specific factors onto the general factor, and subsequent shifts in how the general factor was defined. We situate these methodological shortcomings within the broader literature on structural models of psychopathology, articulate implications for theories (such as the p-factor) that are borne out of factor analysis, outline several remedies for problems encountered when performing exploratory bifactor analysis, and propose alternative specifications for confirmatory bifactor models.


2021 ◽  
Author(s):  
Darren Haywood ◽  
Frank Baughman ◽  
Barbara Mullan ◽  
Karen R. Heslop

Recently, structural models of psychopathology, that address the diagnostic stability and comorbidity issues of the nosological approach, have dominated much of the literature. Structural approaches have given rise to the p factor, which is claimed to reflect an individual’s propensity toward all common psychopathological symptoms. Neurocognitive abilities are argued to be important to the development and maintenance of a wide range of disorders, and have between suggested as an important driver of the p factor. However, recent evidence argues against p being an interpretable substantive construct, limiting conclusions that can be drawn from associations between p and neurocognitive abilities. Here, we argue for the use of the S-1 bifactor approach, where the general factor is defined by neurocognitive abilities, to explore the association. We use simulation techniques to give examples of how S-1 bifactor models can be used to examine the relationship, and how the results can be interpreted.


Assessment ◽  
2021 ◽  
pp. 107319112110602
Author(s):  
Manuel Heinrich ◽  
Christian Geiser ◽  
Pavle Zagorscak ◽  
G. Leonard Burns ◽  
Johannes Bohn ◽  
...  

Symmetrical bifactor models are frequently applied to diverse symptoms of psychopathology to identify a general P factor. This factor is assumed to mark shared liability across all psychopathology dimensions and mental disorders. Despite their popularity, however, symmetrical bifactor models of P often yield anomalous results, including but not limited to nonsignificant or negative specific factor variances and nonsignificant or negative factor loadings. To date, these anomalies have often been treated as nuisances to be explained away. In this article, we demonstrate why these anomalies alter the substantive meaning of P such that it (a) does not reflect general liability to psychopathology and (b) differs in meaning across studies. We then describe an alternative modeling framework, the bifactor-( S−1) approach. This method avoids anomalous results, provides a framework for explaining unexpected findings in published symmetrical bifactor studies, and yields a well-defined general factor that can be compared across studies when researchers hypothesize what construct they consider “transdiagnostically meaningful” and measure it directly. We present an empirical example to illustrate these points and provide concrete recommendations to help researchers decide for or against specific variants of bifactor structure.


2019 ◽  
Vol 7 (6) ◽  
pp. 1285-1303 ◽  
Author(s):  
Ashley L. Watts ◽  
Holly E. Poore ◽  
Irwin D. Waldman

We advanced several “riskier tests” of the validity of bifactor models of psychopathology, which included that the general and specific psychopathology factors should be reliable and well represented by their respective indicators and that including a general factor should improve on the correlated factor model’s external validity. We compared bifactor and correlated factors models of psychopathology using data from a community sample of youth ( N = 2,498) whose parents provided ratings on psychopathology and theoretically relevant external criteria (i.e., personality, aggression, antisociality). Bifactor models tended to yield either general or specific factors that were unstable and difficult to interpret. The general factor appeared to reflect a differentially weighted amalgam of psychopathology rather than a liability for psychopathology broadly construed. With rare exceptions, bifactor models did not explain additional variance in first-order psychopathology symptom dimensions or external criteria compared with correlated factors models. Together, our findings call into question the validity of bifactor models of psychopathology and the p factor more broadly.


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