scholarly journals Swallowing dysfunction in patients hospitalised due to a COPD exacerbation: correspondence

2021 ◽  
pp. 00490-2021
Author(s):  
Nicolas Terzi ◽  
Hélène Prigent ◽  
Frédéric Lofaso
2021 ◽  
pp. 00173-2021
Author(s):  
M. Gonzalez Lindh ◽  
C. Janson ◽  
M. Blom Johansson ◽  
M. Jonsson ◽  
E. Mälberg ◽  
...  

ObjectivesThis cross-sectional study aimed to investigate the prevalence of self-reported and clinically screened swallowing dysfunction (dysphagia) in COPD patients with severe exacerbations and to identify any associated factors. Findings were then compared to a control group.MethodsParticipants included 30 patients hospitalized due to a COPD exacerbation. The control group consisted of 30 adults hospitalized with acute cardiac symptoms. Data were derived from spirometry, the 150mL timed water swallow test, a cookie swallow test and a dyspnea questionnaire (mMRC). Scores from the Eating Assessment Tool (EAT-10) were calculated to assess patient perception of swallowing dysfunction.ResultsSelf-reported swallowing dysfunction and clinical signs thereof was more common in COPD patients than in the control group (67% versus 23% and 80% versus 37%, respectively, p≤0.001). Clinical signs of swallowing dysfunction in the AECOPD group were associated with self-reported swallowing dysfunction (p=0.02) and xerostomia (p=0.04). Dyspnea (MRC≥2) was more common among the COPD patients (90% versus 47%, p<0.001). There was a significant negative correlation between lung function and self-reported dysphagia (r=−0.39, p=0.03), but not between lung function and clinically screened dysphagia (r=−0.23, p=0.21).ConclusionCOPD patients hospitalized with an acute exacerbation experienced significantly more self-reported and clinically screened swallowing dysfunction compared to a control group of patients with cardiac symptoms. Both patient groups experienced dyspnea, but it was twice as common in the AECOPD group. Both groups also experienced xerostomia.


2019 ◽  
Vol 4 (4) ◽  
pp. 648-655
Author(s):  
William G. Pearson ◽  
Jacline V. Griffeth ◽  
Alexis M. Ennis

Purpose Rehabilitation of pharyngeal swallowing dysfunction requires a thorough understanding of the functional anatomy underlying the performance goals of pharyngeal swallowing. These goals include the safe and efficient transfer of a bolus through the hypopharynx into the esophagus. Penetration or aspiration of a bolus threatens swallowing safety. Bolus residue indicates swallowing inefficiency. Several primary mechanics, or elements of the swallowing mechanism, underlie these performance goals, with some elements contributing to both goals. These primary mechanics include velopharyngeal port closure, hyoid movement, laryngeal elevation, pharyngeal shortening, tongue base retraction, and pharyngeal constriction. Each element of the swallowing mechanism is under neuromuscular control and is therefore, in principle, a potential target for rehabilitation. Secondary mechanics of pharyngeal swallowing, those movements dependent on primary mechanics, include opening the upper esophageal sphincter and epiglottic inversion. Conclusion Understanding the functional anatomy of pharyngeal swallowing underlying swallowing performance goals will facilitate anatomically informed critical thinking in the rehabilitation of pharyngeal swallowing dysfunction.


JMS SKIMS ◽  
2010 ◽  
Vol 13 (1) ◽  
pp. 15-19
Author(s):  
Bashir Ahmed Shah ◽  
Muzafar Ahmed Naik ◽  
Sajjad Rajab ◽  
Syed Muddasar ◽  
Ghulam Nabi Dhobi ◽  
...  

Objective: To study the significance of serum magnesium levels during COPD exacerbation and stability.Materials & Methods: The patient population consisted of all patients of COPD admitted as acute exacerbation as defined by the Anthonisens criteria, from June 2006 to may 2008. Same patients one month post discharge presenting to the OPD for routine check up as stable COPD served as controls. Results: A total number of 77 patients of COPD presenting as acute exacerbation were included in the study. The incidence of Hypomagnesaemia was 33.8% at admission; 5% at discharge and 4% at one month of post discharge in COPD patients. The mean serum magnesium levels were significantly lower in cases than controls (1.88±0.67mg/ dl V/S 2.3±0.36mg/dl; p<0.0001). Also, hypomagnesemia was present in higher number of cases (22/77, 33.8%) compared to controls, 3/75, 4.0%; (p<0.0001). Patients of COPD with acute exacerbation and hypomagnesemia, had longer duration of symptoms and had advanced stage III of COPD (p<0.001); and had raised mean corpuscle volume (p<0.045) and longer hospital stay (p<0.008).  Conclusion: We conclude COPD exacerbation is associated with hypomagnesemia. The duration of symptoms of more than 8 days, advanced stage of COPD (stage III) and raised MCV were associated with hypomagnesemia. We recommend to monitor serum magnesium levels in COPD patients with acute exacerbation at the time of admission and during their stay in the hospital.J Med Sci.2010;13(1);15-19


Odontology ◽  
2021 ◽  
Author(s):  
Maya Izumi ◽  
Kazuo Sonoki ◽  
Yuko Ohta ◽  
Masayo Fukuhara ◽  
Masaharu Nagata ◽  
...  

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