scholarly journals Chapter 7: Does Staying Where You Grew Up Mean You Don’t Develop? Following Jannis and Noel from Young to Middle Adulthood

2021 ◽  
pp. 171-194
Author(s):  
Ramona Bullik ◽  
Barbara Keller ◽  
Christopher F. Silver
Keyword(s):  
2010 ◽  
Vol 31 (3) ◽  
pp. 158-165 ◽  
Author(s):  
Thomas Boll ◽  
Tom Michels ◽  
Dieter Ferring ◽  
Sigrun-Heide Filipp

Despite its importance for basic and applied psychology, only a few longitudinal studies have examined whether parental differential treatment (PDT) is a persistent or a transient phenomenon, these studies being confined to childhood or adolescence. Based on latent state-trait theory, the present study identified the amount of variance in three dimensions of perceived PDT in middle adulthood attributable to stable interindividual differences (trait variance) and to intraindividual changes (state variance). At two occasions of measurement (2 years apart), 709 middle-aged adults rated how often they and a sibling currently received parental recognition, nurture, and demand to assume filial responsibility. Tests of latent state-trait models for these three dimensions of PDT by structural equation modeling revealed that trait variance represented the largest proportion of the systematic variance in all observed indicators of perceived maternal and paternal differential treatment. Yet there was a considerable increase in state variance for the dimension of differential parental demand for assuming responsibility. Results are discussed with respect to the conditions accounting for the high overall stability of actual and/or perceived PDT in adulthood, and different approaches for determining their role are proposed.


1986 ◽  
Vol 31 (1) ◽  
pp. 29-30
Author(s):  
Florence L. Denmark
Keyword(s):  

2021 ◽  
Vol 21 (1) ◽  
Author(s):  
Jaehong Yoon ◽  
Ja Young Kim ◽  
Ji-Hwan Kim ◽  
Seung-Sup Kim

Abstract Background We sought to examine the association between childhood experience of parental death (CEPD) and adulthood suicidal ideation, and the mediating role of adulthood SES in the association. Methods We analyzed a nationally representative dataset of 8609 adults from the Korea Welfare Panel Study, which is a longitudinal cohort dataset in South Korea. CEPD was measured using a question: “During your childhood (0-17 years old), have you experienced the death of parents?” We classified responses of CEPD during 2006–2011 into ‘yes,’ and the others into ‘no.’ Suicidal ideation over the past year was assessed annually during 2012–2019. As a potential mediator, adulthood educational attainment and household income in 2011 were included in the analysis. Logistic regression was applied to examine the association of CEPD with adulthood suicidal ideation across age groups (early adulthood, 19–39 years old; middle adulthood, 40–59 years old; late adulthood, ≥60 years old), after excluding people who reported lifetime suicidal ideation in 2011. Causal mediation analysis using a parametric regression model was applied to examine the mediating role of adulthood SES in the association between CEPD and adulthood suicidal ideation. Results After adjusting for potential confounders including childhood SES, CEPD was significantly associated with adulthood suicidal ideation among the late adulthood group (OR: 1.43; 95% CI: 1.13–1.81), while the association was not statistically significant among the early; and middle adulthood groups. In mediation analysis of adulthood household income, both indirect association (ORNIE: 1.05; 95% CI: 1.02–1.09) and direct association (ORNDE: 1.37; 95% CI: 1.09–1.73) were statistically significant among the late adulthood group. In the mediation analysis of adulthood education attainment among the late adulthood, only a direct association was statistically significant (ORNDE: 1.43; 95% CI: 1.14–1.80). Conclusions These results suggest that CEPD could be a risk factor for adulthood suicidal ideation. Furthermore, the findings imply that income security policy might be necessary to reduce suicide among the late adulthood group.


BMC Medicine ◽  
2021 ◽  
Vol 19 (1) ◽  
Author(s):  
Joshua A. Bell ◽  
Diana L. Santos Ferreira ◽  
Abigail Fraser ◽  
Ana Luiza G. Soares ◽  
Laura D. Howe ◽  
...  

Abstract Background Males experience higher rates of coronary heart disease (CHD) than females, but the circulating traits underpinning this difference are poorly understood. We examined sex differences in systemic metabolites measured at four life stages, spanning childhood to middle adulthood. Methods Data were from the Avon Longitudinal Study of Parents and Children (7727 offspring, 49% male; and 6500 parents, 29% male). Proton nuclear magnetic resonance (1H-NMR) spectroscopy from a targeted metabolomics platform was performed on EDTA-plasma or serum samples to quantify 229 systemic metabolites (including lipoprotein-subclass-specific lipids, pre-glycaemic factors, and inflammatory glycoprotein acetyls). Metabolites were measured in the same offspring once in childhood (mean age 8 years), twice in adolescence (16 years and 18 years) and once in early adulthood (25 years), and in their parents once in middle adulthood (50 years). Linear regression models estimated differences in metabolites for males versus females on each occasion (serial cross-sectional associations). Results At 8 years, total lipids in very-low-density lipoproteins (VLDL) were lower in males; levels were higher in males at 16 years and higher still by 18 years and 50 years (among parents) for medium-or-larger subclasses. Larger sex differences at older ages were most pronounced for VLDL triglycerides—males had 0.19 standard deviations (SD) (95% CI = 0.12, 0.26) higher at 18 years, 0.50 SD (95% CI = 0.42, 0.57) higher at 25 years, and 0.62 SD (95% CI = 0.55, 0.68) higher at 50 years. Low-density lipoprotein (LDL) cholesterol, apolipoprotein-B, and glycoprotein acetyls were generally lower in males across ages. The direction and magnitude of effects were largely unchanged when adjusting for body mass index measured at the time of metabolite assessment on each occasion. Conclusions Our results suggest that males begin to have higher VLDL triglyceride levels in adolescence, with larger sex differences at older ages. Sex differences in other CHD-relevant metabolites, including LDL cholesterol, show the opposite pattern with age, with higher levels among females. Such life course trends may inform causal analyses with clinical endpoints in specifying traits which underpin higher age-adjusted CHD rates commonly seen among males.


2021 ◽  
pp. 016402752110273
Author(s):  
Markus Wettstein ◽  
Hans-Werner Wahl ◽  
Vera Heyl

Although stress is a risk factor for various diseases in later life, its role for sensory abilities in the second half of life has rarely been empirically addressed. We examined if perceived stress at baseline predicts self-reported difficulties with vision and hearing 3 years later. We also explored whether chronological age is a moderator of associations between stress and sensory difficulties. Our sample was derived from the German Ageing Survey and consisted of n = 5,085 individuals aged 40–95 years ( M = 64.01 years, SD = 10.84 years). Controlling for baseline self-reported sensory functioning, socio-demographic indicators, self-rated health and chronic diseases, greater perceived stress at baseline predicted greater self-reported difficulties with vision and hearing 3 years later. The effect of stress did not vary by age. Our findings suggest that, from middle adulthood to advanced old age, stress is a risk factor for increases in self-perceived problems with vision and hearing.


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