Acute and Chronic Childhood Lead Poisoning: Criticism of the Statement

PEDIATRICS ◽  
1972 ◽  
Vol 49 (3) ◽  
pp. 474-475
Author(s):  
Jane S. Lin-Fu

I read the AAP's recent statement, "Acute and Chronic Childhood Lead Poisoning,"1 with disappointment verging on alarm. The statement recommends that "the major emphasis . . . be placed on the testing of dwellings for lead-pigment paints . . . in order to identify high-risk areas." Yet many such areas, or "lead belts," have long been so correctly identified that 20 to 40% of young children screened from these areas have been shown to have blood lead values of 40 µg/100 ml or more.2

PEDIATRICS ◽  
1971 ◽  
Vol 48 (3) ◽  
pp. 490-491
Author(s):  
Reginald S. Lourie ◽  
Paul F. Wehrle

The otherwise excellent and timely statement on acute and chronic childhood lead poisoning of the Academy's Committee on Environmental Hazards and Subcommittee on Accidental Poisoning of the Committee on Accident Prevention1 has one serious omission. After calling attention to pica as a major factor in lead poisoning in young children, this fundamental cause for most cases of plumbism is disregarded. A basic part of the problem would appear to be how it attacks the reasons why the children eat the paint on the walls in the first place.


PEDIATRICS ◽  
1996 ◽  
Vol 97 (6) ◽  
pp. 916-917
Author(s):  
Mary Beth Miller ◽  
Steven C. Curry ◽  
Donald B. Kunkel ◽  
Patricia Arreola ◽  
Ernest Arvizu ◽  
...  

Lead compounds are used as coloring agents for numerous products. Two cases of children with elevated blood lead concentrations encountered by the authors suggested that pool cue chalk may serve as a source of environmental lead. The objective of this study was to determine lead content of various brands and colors of pool cue chalk. Atomic absorption analyses were conducted of 23 different types of pool cue chalk for lead content. Three of 23 types of pool cue chalk contained more than 7000 ppm (mg/kg) lead: one manufacturer's green and tangerine chalk and another manufacturer's green chalk. It was concluded that some brands of pool cue chalk contain relatively large amounts of lead and could contribute to childhood lead poisoning.


1997 ◽  
Vol 3 (4) ◽  
pp. 241-248 ◽  
Author(s):  
Carol H. Rubin ◽  
Emilio Esteban ◽  
Robert Jones ◽  
Gary Noonan ◽  
Elena Gurvich ◽  
...  

2019 ◽  
Vol 25 ◽  
pp. S51-S57 ◽  
Author(s):  
Shelley A. Bruce ◽  
Krista Y. Christensen ◽  
Marjorie J. Coons ◽  
Jeffrey A. Havlena ◽  
Jon G. Meiman ◽  
...  

PEDIATRICS ◽  
1993 ◽  
Vol 92 (1) ◽  
pp. 176-183
Author(s):  

Knowledge of the extent and seriousness of childhood lead poisoning has vastly expanded since the last statement regarding lead poisoning by the American Academy of Pediatrics in 1987.1 Blood lead levels once thought to be safe have been shown to be associated with IQ deficits, behavior disorders, slowed growth, and impaired hearing.2 In fact, lead poisoning is, according to the Department of Health and Human Services, "the most important environmental health problem for young children."3 The rapid development of the scientific database requires recognition by physicians of the significance of effects at lower levels and a change in clinical practice. During the last 30 years the Centers for Disease Control and Prevention (CDC) has revised downward the definition of the blood level at which lead poisoning occurs from 60 µg/dL whole blood in the early 1960s, to 30 µg/dL in 1975, and 25 µg/dL in 1985. The 1991 CDC statement "Preventing Lead Poisoning in Young Children" recommended lowering the community intervention level to 10 µg/dL and setting several action levels (Table 1).2 In 1987 the American Academy of Pediatrics stated that lead levels greater than 25 µg/dL were unacceptable for children.1 The Academy now recognizes that impairment of cognitive function begins to occur at levels greater than 10 µg/dL, even though clinical symptoms are not seen. In the late 1970s, the average blood lead level for US children was 16 µg/dL.4 The mean blood lead level for US children has declined since 1976 due to the phaseout of lead in gasoline5 and the reduction of lead in food, and it is now between 4 and 6 µg/dL.6


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