scholarly journals Study on the Respiration of Crop Plants : (8) Fffect of hydrogen-sulfide and lower fatty acids on the respiration of root in rice plant

1959 ◽  
Vol 27 (2) ◽  
pp. 155-160 ◽  
Author(s):  
Noboru YAMADA ◽  
Yasuo OTA
Life ◽  
2021 ◽  
Vol 11 (2) ◽  
pp. 149
Author(s):  
Thomas Geisberger ◽  
Jessica Sobotta ◽  
Wolfgang Eisenreich ◽  
Claudia Huber

Thiophene was detected on Mars during the Curiosity mission in 2018. The compound was even suggested as a biomarker due to its possible origin from diagenesis or pyrolysis of biological material. In the laboratory, thiophene can be synthesized at 400 °C by reacting acetylene and hydrogen sulfide on alumina. We here show that thiophene and thiophene derivatives are also formed abiotically from acetylene and transition metal sulfides such as NiS, CoS and FeS under simulated volcanic, hydrothermal conditions on Early Earth. Exactly the same conditions were reported earlier to have yielded a plethora of organic molecules including fatty acids and other components of extant metabolism. It is therefore tempting to suggest that thiophenes from abiotic formation could indicate sites and conditions well-suited for the evolution of metabolism and potentially for the origin-of-life on extraterrestrial planets.


2017 ◽  
Vol 42 (4) ◽  
pp. 1635-1644 ◽  
Author(s):  
Zhu-lin Luo ◽  
Jian-dong Ren ◽  
Zhu Huang ◽  
Tao Wang ◽  
Ke Xiang ◽  
...  

Background: This study aimed to investigate whether exogenous hydrogen sulfide (H2S) can protect the RAW264.7 macrophages against the inflammation induced by free fatty acids (FFA) by blunting NLRP3 inflammasome activation via a specific TLR4/NF-κB pathway. Methods: RAW264.7 macrophages were exposed to increasing concentrations of FFA for up to 3 days to induce FFA-induced inflammation. The cells were pretreated with NaHS (a donor of H2S) before exposure to FFA. Cell viability, cell apoptosis, TLR4, NF-κB, NLRP3 inflammasome, IL-1β, IL-18 and cleaved caspase-3 expression were measured by a combination of MTT assay, ELISA, and immunoblotting. Results: H2S attenuated FFA-induced cell apoptosis, and reduced the expression of NLRP3, ASC, pro-caspase-1, caspase-1, IL- 1β, IL-18 and caspase-3. In addition, H2S inhibited the FFA-induced activation of TLR4 and NF-κB. Furthermore, NLRP3 inflammasome activation was regulated by the TLR4 and NF-κB pathway. Conclusion: The present study demonstrated for the first time that H2S appears to suppress FFA-induced macrophage inflammation and apoptosis by inhibiting the TLR4/ NF-κB pathway and its downstream NLRP3 inflammasome activation. Thus H2S might possess potential in the treatment of diseases resulting from FFA overload like insulin resistance and type diabetes.


2016 ◽  
Vol 11 (10) ◽  
pp. 1262-1267 ◽  
Author(s):  
Takeshi Furuhashi ◽  
Takemichi Nakamura ◽  
Lena Fragner ◽  
Valentin Roustan ◽  
Verena Schön ◽  
...  

1968 ◽  
Vol 14 (1) ◽  
pp. 1-6 ◽  
Author(s):  
A. Tanaka ◽  
Ranjit P. Mulleriyawa ◽  
T. Yasu

1954 ◽  
Vol 22 (3-4) ◽  
pp. 53-54
Author(s):  
Noboru YAMADA ◽  
Yoshio MURATA ◽  
Akio OSADA ◽  
Junichiro IYAMA

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