Modern approaches and prospective directions in treatment of acute sensorineural hearing loss following acoustic trauma

2020 ◽  
Vol 85 (5) ◽  
pp. 88
Author(s):  
M.S. Kuznecov ◽  
M.V. Morozova ◽  
V.V. Dvorjanchikov ◽  
L.A. Glaznikov ◽  
V.L. Pastushenkov ◽  
...  
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Acoustic Trauma ◽  
Sensorineural Hearing

scholarly journals Characteristics of sensorineural hearing loss secondary to inner ear acoustic trauma

2008 ◽  
Vol 136 (5-6) ◽  
pp. 221-225
Author(s):  
Slobodan Spremo ◽  
Zdenko Stupar
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Hearing Threshold ◽  
Acoustic Trauma ◽  
Sensorineural Hearing ◽  
Cochlear Damage ◽  
Type 3 ◽  
Trauma Group

INTRODUCTION Cochlear damage secondary to exposure to acoustic trauma is the consequence of the acoustic energy effects on the hearing cells in Korti's organ. OBJECTIVE The objective was to assess the correlation between the degree of sensorineural hearing loss and the type of audiogram registered in acoustic trauma exposed patients. METHOD We analyzed 262 audiograms of patients exposed to acoustic trauma in correlation to 146 audiograms of patients with cochlear damage and hearing loss not related to acoustic trauma. "A" group consisted of acoustic trauma cases, while "B" group incorporated cases with hearing loss secondary to cochlear ischaemia or degeneration. All audiograms were subdivided with regard to the mean hearing loss into three groups: mild (21-40 dB HL), moderate (41-60 dB HL) and severe (over 60 dB HL) hearing loss. Based on audiogram configuration five types of audiogram were defined: type 1 flat; type 2 hearing threshold slope at 2 kHz, type 3 hearing threshold slope at 4 kHz; type 4 hearing threshold notch at 2 kHz; type 5 notch at 4 kHz. RESULTS Mild hearing loss was recorded in 163 (62.2%) ears in the acoustic trauma group, while in 78 (29.8%) ears we established moderate hearing loss with the maximum threshold shift at frequencies ranging from 4 kHz to 8 kHz. The least frequent was profound hearing loss, obtained in 21 (8%) audiograms in the acoustic trauma group. Characteristic audiogram configurations in the acoustic trauma patient group were: type 1 (N=66; 25.2%), type 2 (N=71; 27.1%), and type 3 (N=68; 25.9%). Audiogram configurations were significanly different in the acoustic trauma group in comparison to the cochlear ischaemia group of patients (p=0.0005). CONCLUSION Cochlear damage concomitant to acoustic trauma could be assessed by the audiogram configuration. Preserved hearing acuity at low and mild frequency range indicates the limited damage to the hearing cells in Korti's organ in the apical cochlear turn.

Ears

2020 ◽  
Author(s):  
Peter Pruitt ◽  
Thomas Osborne Stair
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Acoustic Trauma ◽  
Tympanic Membrane Perforation ◽  
Sensorineural Hearing ◽  
Hearing Disorders ◽  
High Frequency Hearing Loss ◽  
Membrane Perforation ◽  
Acute Hearing Loss ◽  
Conductive Hearing

As the structure of the ear is made of little more than cartilage, a limited blood supply, and a thin layer of skin, trauma in this area can easily manifest from a variety of causes. Some common examples of trauma involve laceration, piercing (intentional or otherwise), infection causing chondritis, blunt trauma causing necrosis, rupture of the tympanic membrane, perforation of the ear drum, and acoustic trauma that may result in hearing disorders such as tinnitus and high-frequency hearing loss. Acute hearing loss shows in two forms: conductive hearing loss and sensorineural hearing loss, the latter of which is caused by damage to the anatomic or neurologic structures of the ear dedicated to hearing. Sensorineural hearing loss generally has a poor prognosis and mandates prompt referral to an otolaryngologist.  This review contains 4 figures, 13 tables, and 32 references. Keywords: Ear, auricular canal, trauma, otitis media, otitis externa, hearing loss, mastoiditis, cerumen, impaction

scholarly journals How Drill-Generated Acoustic Trauma effects Hearing Functions in an Ear Surgery?

2012 ◽  
Vol 3 (3) ◽  
pp. 127-132 ◽  
Author(s):  
Mustafa Paksoy ◽  
Arif Sanli ◽  
Umit Hardal ◽  
Sermin Kibar ◽  
Gokhan Altin ◽  
...  
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Noise Exposure ◽  
Bone Conduction ◽  
Acoustic Trauma ◽  
Sensorineural Hearing ◽  
Radical Mastoidectomy ◽  
Ear Surgery ◽  
Hearing Thresholds ◽  
Mastoid Surgery

ABSTRACT Objective In otology, a wide variety of devices are used that have significant noise output, both operated ear and the patient. We aimed to determine hearing damages due to drill-generated acoustic trauma in ear surgery. We want to find how degree drill-generated acoustic trauma is responsible from sensorineural hearing loss in ear surgery. Materials and methods We designed a retrospective study about 100 patients who underwent radical or modiphied radical mastoidectomy and tympanoplasty. The audiometric testing was done both pre and postoperatively to detect any significant hearing loss in the immediate postoperative period. The data were analyzed using the Wilcoxon sign and Mann-Whitney U tests. This study proposes that hearing loss is caused by drill noise conducted to the operated ear by vibrations of temporal bone. Results A sensorineural hearing loss soon after mastoid surgery is seen due to the noise generated by the drill. Mean pure-tone thresholds obtained was significantly more in mastoidectomy applied patients when compared to tympanoplasty . Mean bone conduction (BC) hearing levels impaired 6,6 dB in 1 kHz, 5.5 dB in 0.5 kHz, 5 dB in 4.kHz and 3.1 dB in 2 kHz in mastoidectomy groups but improved 5.5 dB in 0.5 kHz, 2.2 dB in 1 kHz, 2.7 dB in 2 kHz in tympanoplasty groups. Statistically significant differences were observed at the 0.5-1 and 4 kHz frequencies pre and postoperative in the hearing thresholds of BC changing in mastoidectomy group, however, the averages of ranks of all pre and postoperative measurement of hearing levels show differences between mastoidectomy and tympanoplasty groups was significant in statistically at independent groups (p < 0.05). Conclusion We conclude that drill-generated noise during mastoid surgery has been incriminated as a cause of sensorineural hearing loss. Drilling during mastoid surgery may result in temporary or permanent noise-induced hearing loss. Possible noise disturbance to the inner ear can only be avoided by minimizing the duration of harmful noise exposure and carefull using burr to near the cochlear structures. How to cite this article Paksoy M, Sanli A, Hardal U, Kibar S, Altin G, Erdogan BA, Bekmez ZE. How Drill-Generated Acoustic Trauma effects Hearing Functions in an Ear Surgery? Int J Head and Neck Surg 2012;3(3):127-132.

Transient Sensorineural Hearing Loss after Overuse of Portable Headphone Cassette Radios

1985 ◽  
Vol 93 (5) ◽  
pp. 622-625 ◽  
Author(s):  
Phillip C. Lee ◽  
Craig W. Senders ◽  
Bruce J. Gantz ◽  
Steven R. Otto
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Occupational Safety ◽  
Noise Exposure ◽  
Maximum Level ◽  
Acoustic Trauma ◽  
Sensorineural Hearing ◽  
Maximum Output ◽  
Health Administration ◽  
Listening Levels

Noise-induced sensorineural hearing loss has been associated with Industry for many years. One conservative estimate suggests that 10 million Americans may have industry-related, noise-induced hearing loss. Acoustic trauma from any source, whether associated with work or recreations, is detrimental to hearing. The Occupational Safety and Health Administration has set industrial standards for noise levels, with current standards limiting noise exposure to 95 dBA for 2 hours daily. To date, however, there are no recreational standards. Many portable headphone cassette radios produce peak outputs of more than 100 dBA. Temporary threshold shifts could result from listening levels near the maximum output. Permanent sensorineural loss may result with repeated exposure. A pilot study was conducted in which 16 volunteers listened to headphone sets for 3 hours at their usual maximum level. Six volunteers showed transient shifts of 10 dB, and one volunteer showed a transient shift of approximately 30 dB. These shifts returned to normal within 24 hours. As expected, transient shifts frequently occur with recreational use. Therefore, recreational warnings and standards should be established.

Sensorineural hearing loss in chronic otitis media

1989 ◽  
Vol 103 (2) ◽  
pp. 158-163 ◽  
Author(s):  
F. Cusimano ◽  
V. C. Cocita ◽  
A. D'Amico
Keyword(s):  
Cardiovascular Disease ◽  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Otitis Media ◽  
Statistical Study ◽  
Age Of Onset ◽  
Chronic Otitis Media ◽  
Acoustic Trauma ◽  
Sensorineural Hearing ◽  
Ototoxic Drugs

AbstractA statistical study was done on the sensorineural component in hearing loss, using 595 patients suffering from Chronic Otitis Media (COM); of these, 195 with monolateral COM were taken into consideration. They presented criteria of valuation which excluded other possible causes of sensorineural hearing loss, such as exposure to acoustic trauma, ototoxic drugs, cardiovascular disease, past head injury and hereditary causes. The contralateral (healthy) ear served as a control. We determined the average sensorineural component in the hearing losses in relation to the age of onset and duration of the disease, examining it in relation to other eventual aural complications such as cholesteatoma.On the basis of the data obtained, we believe that the sensorineural component in hearing loss does not change with respect to the age of onset of COM, but the duration of COM does exert a significant influence.

scholarly journals Spinal anesthesia causing hearing loss: are we aware?

2014 ◽  
Vol 13 (3) ◽  
pp. 251-254
Author(s):  
Hasme Zam Hashim ◽  
Irfan Mohamad ◽  
Rosdan Salim ◽  
Saedah Ali
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Inner Ear ◽  
Spinal Anesthesia ◽  
Medical Science ◽  
Acoustic Trauma ◽  
Sensorineural Hearing ◽  
Noisy Environment ◽  
Genetic Syndromes ◽  
Ear Infection

Sensorineural hearing loss can be attributed to many factors. Acoustic trauma, noisy environment, genetic syndromes, inner ear infection and tumors are the known well-established causes. Some of them are treatable but many of those are nonreversible. Recent literatures have shown some data that suggest this type of hearing loss also occurring post anesthesia, particularly in spinal anesthesia cases. Others claim that this hearing loss is temporary and clinically not significant. DOI: http://dx.doi.org/10.3329/bjms.v13i3.19136 Bangladesh Journal of Medical Science Vol.13(3) 2014 p.251-254

Imaging of the Temporal Bone

2020 ◽  
Author(s):  
Peter Pruitt
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Acoustic Trauma ◽  
Tympanic Membrane Perforation ◽  
Sensorineural Hearing ◽  
Hearing Disorders ◽  
High Frequency Hearing Loss ◽  
Membrane Perforation ◽  
Acute Hearing Loss ◽  
Conductive Hearing

As the structure of the ear is made of little more than cartilage, a limited blood supply, and a thin layer of skin, trauma in this area can easily manifest from a variety of causes. Some common examples of trauma involve laceration, piercing (intentional or otherwise), infection causing chondritis, blunt trauma causing necrosis, rupture of the tympanic membrane, perforation of the ear drum, and acoustic trauma that may result in hearing disorders such as tinnitus and high-frequency hearing loss. Acute hearing loss shows in two forms: conductive hearing loss and sensorineural hearing loss, the latter of which is caused by damage to the anatomic or neurologic structures of the ear dedicated to hearing. Sensorineural hearing loss generally has a poor prognosis and mandates prompt referral to an otolaryngologist.  This review contains 4 figures, 13 tables, and 32 references. Keywords: Ear, auricular canal, trauma, otitis media, otitis externa, hearing loss, mastoiditis, cerumen, impaction

Ears

2020 ◽  
Author(s):  
Peter Pruitt ◽  
Thomas Osborne Stair
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Acoustic Trauma ◽  
Tympanic Membrane Perforation ◽  
Sensorineural Hearing ◽  
Hearing Disorders ◽  
High Frequency Hearing Loss ◽  
Membrane Perforation ◽  
Acute Hearing Loss ◽  
Conductive Hearing

As the structure of the ear is made of little more than cartilage, a limited blood supply, and a thin layer of skin, trauma in this area can easily manifest from a variety of causes. Some common examples of trauma involve laceration, piercing (intentional or otherwise), infection causing chondritis, blunt trauma causing necrosis, rupture of the tympanic membrane, perforation of the ear drum, and acoustic trauma that may result in hearing disorders such as tinnitus and high-frequency hearing loss. Acute hearing loss shows in two forms: conductive hearing loss and sensorineural hearing loss, the latter of which is caused by damage to the anatomic or neurologic structures of the ear dedicated to hearing. Sensorineural hearing loss generally has a poor prognosis and mandates prompt referral to an otolaryngologist.  This review contains 4 figures, 13 tables, and 32 references. Keywords: Ear, auricular canal, trauma, otitis media, otitis externa, hearing loss, mastoiditis, cerumen, impaction

scholarly journals Analysis of Pharmacokinetics in the Cochlea of the Inner Ear

2021 ◽  
Vol 12 ◽  
Author(s):  
Seishiro Sawamura ◽  
Genki Ogata ◽  
Kai Asai ◽  
Olga Razvina ◽  
Takeru Ota ◽  
...  
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Inner Ear ◽  
Analytical Techniques ◽  
Acoustic Trauma ◽  
Therapeutic Agents ◽  
Therapeutic Strategies ◽  
Clinical Impact ◽  
Sensorineural Hearing ◽  
Global Population

Hearing loss affects &gt;5% of the global population and therefore, has a great social and clinical impact. Sensorineural hearing loss, which can be caused by different factors, such as acoustic trauma, aging, and administration of certain classes of drugs, stems primarily from a dysfunction of the cochlea in the inner ear. Few therapeutic strategies against sensorineural hearing loss are available. To develop effective treatments for this disease, it is crucial to precisely determine the behavior of ototoxic and therapeutic agents in the microenvironment of the cochlea in live animals. Since the 1980s, a number of studies have addressed this issue by different methodologies. However, there is much less information on pharmacokinetics in the cochlea than that in other organs; the delay in ontological pharmacology is likely due to technical difficulties with accessing the cochlea, a tiny organ that is encased with a bony wall and has a fine and complicated internal structure. In this review, we not only summarize the observations and insights obtained in classic and recent studies on pharmacokinetics in the cochlea but also describe relevant analytical techniques, with their strengths, limitations, and prospects.

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