CONDITION OF THE AUDITORY ANALYZER IN PATIENTS WITH ACUTE SENSORINEURAL HEARING LOSS AFTER ACOUSTIC TRAUMA

2019 ◽  
pp. 44-45
Author(s):  
S. S. Arifov ◽  
S. S. Orifov
2008 ◽  
Vol 136 (5-6) ◽  
pp. 221-225
Author(s):  
Slobodan Spremo ◽  
Zdenko Stupar

INTRODUCTION Cochlear damage secondary to exposure to acoustic trauma is the consequence of the acoustic energy effects on the hearing cells in Korti's organ. OBJECTIVE The objective was to assess the correlation between the degree of sensorineural hearing loss and the type of audiogram registered in acoustic trauma exposed patients. METHOD We analyzed 262 audiograms of patients exposed to acoustic trauma in correlation to 146 audiograms of patients with cochlear damage and hearing loss not related to acoustic trauma. "A" group consisted of acoustic trauma cases, while "B" group incorporated cases with hearing loss secondary to cochlear ischaemia or degeneration. All audiograms were subdivided with regard to the mean hearing loss into three groups: mild (21-40 dB HL), moderate (41-60 dB HL) and severe (over 60 dB HL) hearing loss. Based on audiogram configuration five types of audiogram were defined: type 1 flat; type 2 hearing threshold slope at 2 kHz, type 3 hearing threshold slope at 4 kHz; type 4 hearing threshold notch at 2 kHz; type 5 notch at 4 kHz. RESULTS Mild hearing loss was recorded in 163 (62.2%) ears in the acoustic trauma group, while in 78 (29.8%) ears we established moderate hearing loss with the maximum threshold shift at frequencies ranging from 4 kHz to 8 kHz. The least frequent was profound hearing loss, obtained in 21 (8%) audiograms in the acoustic trauma group. Characteristic audiogram configurations in the acoustic trauma patient group were: type 1 (N=66; 25.2%), type 2 (N=71; 27.1%), and type 3 (N=68; 25.9%). Audiogram configurations were significanly different in the acoustic trauma group in comparison to the cochlear ischaemia group of patients (p=0.0005). CONCLUSION Cochlear damage concomitant to acoustic trauma could be assessed by the audiogram configuration. Preserved hearing acuity at low and mild frequency range indicates the limited damage to the hearing cells in Korti's organ in the apical cochlear turn.


2020 ◽  
Author(s):  
Peter Pruitt ◽  
Thomas Osborne Stair

As the structure of the ear is made of little more than cartilage, a limited blood supply, and a thin layer of skin, trauma in this area can easily manifest from a variety of causes. Some common examples of trauma involve laceration, piercing (intentional or otherwise), infection causing chondritis, blunt trauma causing necrosis, rupture of the tympanic membrane, perforation of the ear drum, and acoustic trauma that may result in hearing disorders such as tinnitus and high-frequency hearing loss. Acute hearing loss shows in two forms: conductive hearing loss and sensorineural hearing loss, the latter of which is caused by damage to the anatomic or neurologic structures of the ear dedicated to hearing. Sensorineural hearing loss generally has a poor prognosis and mandates prompt referral to an otolaryngologist.  This review contains 4 figures, 13 tables, and 32 references. Keywords: Ear, auricular canal, trauma, otitis media, otitis externa, hearing loss, mastoiditis, cerumen, impaction


2012 ◽  
Vol 3 (3) ◽  
pp. 127-132 ◽  
Author(s):  
Mustafa Paksoy ◽  
Arif Sanli ◽  
Umit Hardal ◽  
Sermin Kibar ◽  
Gokhan Altin ◽  
...  

ABSTRACT Objective In otology, a wide variety of devices are used that have significant noise output, both operated ear and the patient. We aimed to determine hearing damages due to drill-generated acoustic trauma in ear surgery. We want to find how degree drill-generated acoustic trauma is responsible from sensorineural hearing loss in ear surgery. Materials and methods We designed a retrospective study about 100 patients who underwent radical or modiphied radical mastoidectomy and tympanoplasty. The audiometric testing was done both pre and postoperatively to detect any significant hearing loss in the immediate postoperative period. The data were analyzed using the Wilcoxon sign and Mann-Whitney U tests. This study proposes that hearing loss is caused by drill noise conducted to the operated ear by vibrations of temporal bone. Results A sensorineural hearing loss soon after mastoid surgery is seen due to the noise generated by the drill. Mean pure-tone thresholds obtained was significantly more in mastoidectomy applied patients when compared to tympanoplasty . Mean bone conduction (BC) hearing levels impaired 6,6 dB in 1 kHz, 5.5 dB in 0.5 kHz, 5 dB in 4.kHz and 3.1 dB in 2 kHz in mastoidectomy groups but improved 5.5 dB in 0.5 kHz, 2.2 dB in 1 kHz, 2.7 dB in 2 kHz in tympanoplasty groups. Statistically significant differences were observed at the 0.5-1 and 4 kHz frequencies pre and postoperative in the hearing thresholds of BC changing in mastoidectomy group, however, the averages of ranks of all pre and postoperative measurement of hearing levels show differences between mastoidectomy and tympanoplasty groups was significant in statistically at independent groups (p < 0.05). Conclusion We conclude that drill-generated noise during mastoid surgery has been incriminated as a cause of sensorineural hearing loss. Drilling during mastoid surgery may result in temporary or permanent noise-induced hearing loss. Possible noise disturbance to the inner ear can only be avoided by minimizing the duration of harmful noise exposure and carefull using burr to near the cochlear structures. How to cite this article Paksoy M, Sanli A, Hardal U, Kibar S, Altin G, Erdogan BA, Bekmez ZE. How Drill-Generated Acoustic Trauma effects Hearing Functions in an Ear Surgery? Int J Head and Neck Surg 2012;3(3):127-132.


2020 ◽  
Vol 85 (5) ◽  
pp. 88
Author(s):  
M.S. Kuznecov ◽  
M.V. Morozova ◽  
V.V. Dvorjanchikov ◽  
L.A. Glaznikov ◽  
V.L. Pastushenkov ◽  
...  

2019 ◽  
Vol 8 (1) ◽  
pp. 39-42
Author(s):  
Alexander Zinchuk ◽  
Nadiya Prykuda

Introduction. Lyme borreliosis (LB) is endemic spirochetosis with a transmission mechanism, which is often marked by a durable, recurrent course with multiple organ damages. LB is characterized by the appearance of migrating erythema at the site of tick bite. Durable persistence of the agent promotes disease progressing and occurrence of serious complications related to the nervous system, the heart and the locomotive apparatus. Among infrequent and especially dangerous complications of LB is damage to the hearing apparatus with the development of sensorineural deafness – hearing loss, which develops within 24 hours due to function impairment of the internal ear or auditory nerve. That infectious diseases account for 12.8-13% of numerous causes of sensorineural deafness development. Most frequently, the disease caused by B. burgdorferi is mentioned. Aim. To analyze the clinical case of Lyme borreliosis complicated by the sensorineural hearing loss. Materials and methods. The analysis of clinical-laboratory and instrumental data of the patient with Lyme borreliosis, whose course of the disease was complicated by sensorineural hearing loss. The patient received care at Lviv Regional Infectious Diseases Clinical Hospital (LRIDCH). Results. The article presents a clinical case of Lyme borreliosis, complicated by sensorineural hearing loss, which appeared on the 15th day from the onset of erythema annulare in the patient. Hearing recovery in the patient could not be achieved via administration of etiotropic treatment (2.0 g/per day of ceftriaxone for 28 days), which was likely associated with late visit of the patient for medical aid. Conclusions. Sensorineural hearing loss is one of the rare, but severe lesions of auditory analyzer in LB. Hearing improvement after antibiotic therapy is possible in certain cases in early treatment.


1985 ◽  
Vol 93 (5) ◽  
pp. 622-625 ◽  
Author(s):  
Phillip C. Lee ◽  
Craig W. Senders ◽  
Bruce J. Gantz ◽  
Steven R. Otto

Noise-induced sensorineural hearing loss has been associated with Industry for many years. One conservative estimate suggests that 10 million Americans may have industry-related, noise-induced hearing loss. Acoustic trauma from any source, whether associated with work or recreations, is detrimental to hearing. The Occupational Safety and Health Administration has set industrial standards for noise levels, with current standards limiting noise exposure to 95 dBA for 2 hours daily. To date, however, there are no recreational standards. Many portable headphone cassette radios produce peak outputs of more than 100 dBA. Temporary threshold shifts could result from listening levels near the maximum output. Permanent sensorineural loss may result with repeated exposure. A pilot study was conducted in which 16 volunteers listened to headphone sets for 3 hours at their usual maximum level. Six volunteers showed transient shifts of 10 dB, and one volunteer showed a transient shift of approximately 30 dB. These shifts returned to normal within 24 hours. As expected, transient shifts frequently occur with recreational use. Therefore, recreational warnings and standards should be established.


1989 ◽  
Vol 103 (2) ◽  
pp. 158-163 ◽  
Author(s):  
F. Cusimano ◽  
V. C. Cocita ◽  
A. D'Amico

AbstractA statistical study was done on the sensorineural component in hearing loss, using 595 patients suffering from Chronic Otitis Media (COM); of these, 195 with monolateral COM were taken into consideration. They presented criteria of valuation which excluded other possible causes of sensorineural hearing loss, such as exposure to acoustic trauma, ototoxic drugs, cardiovascular disease, past head injury and hereditary causes. The contralateral (healthy) ear served as a control. We determined the average sensorineural component in the hearing losses in relation to the age of onset and duration of the disease, examining it in relation to other eventual aural complications such as cholesteatoma.On the basis of the data obtained, we believe that the sensorineural component in hearing loss does not change with respect to the age of onset of COM, but the duration of COM does exert a significant influence.


2014 ◽  
Vol 13 (3) ◽  
pp. 251-254
Author(s):  
Hasme Zam Hashim ◽  
Irfan Mohamad ◽  
Rosdan Salim ◽  
Saedah Ali

Sensorineural hearing loss can be attributed to many factors. Acoustic trauma, noisy environment, genetic syndromes, inner ear infection and tumors are the known well-established causes. Some of them are treatable but many of those are nonreversible. Recent literatures have shown some data that suggest this type of hearing loss also occurring post anesthesia, particularly in spinal anesthesia cases. Others claim that this hearing loss is temporary and clinically not significant. DOI: http://dx.doi.org/10.3329/bjms.v13i3.19136 Bangladesh Journal of Medical Science Vol.13(3) 2014 p.251-254


2020 ◽  
Author(s):  
Peter Pruitt

As the structure of the ear is made of little more than cartilage, a limited blood supply, and a thin layer of skin, trauma in this area can easily manifest from a variety of causes. Some common examples of trauma involve laceration, piercing (intentional or otherwise), infection causing chondritis, blunt trauma causing necrosis, rupture of the tympanic membrane, perforation of the ear drum, and acoustic trauma that may result in hearing disorders such as tinnitus and high-frequency hearing loss. Acute hearing loss shows in two forms: conductive hearing loss and sensorineural hearing loss, the latter of which is caused by damage to the anatomic or neurologic structures of the ear dedicated to hearing. Sensorineural hearing loss generally has a poor prognosis and mandates prompt referral to an otolaryngologist.  This review contains 4 figures, 13 tables, and 32 references. Keywords: Ear, auricular canal, trauma, otitis media, otitis externa, hearing loss, mastoiditis, cerumen, impaction


2020 ◽  
Author(s):  
Peter Pruitt ◽  
Thomas Osborne Stair

As the structure of the ear is made of little more than cartilage, a limited blood supply, and a thin layer of skin, trauma in this area can easily manifest from a variety of causes. Some common examples of trauma involve laceration, piercing (intentional or otherwise), infection causing chondritis, blunt trauma causing necrosis, rupture of the tympanic membrane, perforation of the ear drum, and acoustic trauma that may result in hearing disorders such as tinnitus and high-frequency hearing loss. Acute hearing loss shows in two forms: conductive hearing loss and sensorineural hearing loss, the latter of which is caused by damage to the anatomic or neurologic structures of the ear dedicated to hearing. Sensorineural hearing loss generally has a poor prognosis and mandates prompt referral to an otolaryngologist.  This review contains 4 figures, 13 tables, and 32 references. Keywords: Ear, auricular canal, trauma, otitis media, otitis externa, hearing loss, mastoiditis, cerumen, impaction


Sign in / Sign up

Export Citation Format

Share Document