scholarly journals How Drill-Generated Acoustic Trauma effects Hearing Functions in an Ear Surgery?

2012 ◽  
Vol 3 (3) ◽  
pp. 127-132 ◽  
Author(s):  
Mustafa Paksoy ◽  
Arif Sanli ◽  
Umit Hardal ◽  
Sermin Kibar ◽  
Gokhan Altin ◽  
...  
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Noise Exposure ◽  
Bone Conduction ◽  
Acoustic Trauma ◽  
Sensorineural Hearing ◽  
Radical Mastoidectomy ◽  
Ear Surgery ◽  
Hearing Thresholds ◽  
Mastoid Surgery

ABSTRACT Objective In otology, a wide variety of devices are used that have significant noise output, both operated ear and the patient. We aimed to determine hearing damages due to drill-generated acoustic trauma in ear surgery. We want to find how degree drill-generated acoustic trauma is responsible from sensorineural hearing loss in ear surgery. Materials and methods We designed a retrospective study about 100 patients who underwent radical or modiphied radical mastoidectomy and tympanoplasty. The audiometric testing was done both pre and postoperatively to detect any significant hearing loss in the immediate postoperative period. The data were analyzed using the Wilcoxon sign and Mann-Whitney U tests. This study proposes that hearing loss is caused by drill noise conducted to the operated ear by vibrations of temporal bone. Results A sensorineural hearing loss soon after mastoid surgery is seen due to the noise generated by the drill. Mean pure-tone thresholds obtained was significantly more in mastoidectomy applied patients when compared to tympanoplasty . Mean bone conduction (BC) hearing levels impaired 6,6 dB in 1 kHz, 5.5 dB in 0.5 kHz, 5 dB in 4.kHz and 3.1 dB in 2 kHz in mastoidectomy groups but improved 5.5 dB in 0.5 kHz, 2.2 dB in 1 kHz, 2.7 dB in 2 kHz in tympanoplasty groups. Statistically significant differences were observed at the 0.5-1 and 4 kHz frequencies pre and postoperative in the hearing thresholds of BC changing in mastoidectomy group, however, the averages of ranks of all pre and postoperative measurement of hearing levels show differences between mastoidectomy and tympanoplasty groups was significant in statistically at independent groups (p < 0.05). Conclusion We conclude that drill-generated noise during mastoid surgery has been incriminated as a cause of sensorineural hearing loss. Drilling during mastoid surgery may result in temporary or permanent noise-induced hearing loss. Possible noise disturbance to the inner ear can only be avoided by minimizing the duration of harmful noise exposure and carefull using burr to near the cochlear structures. How to cite this article Paksoy M, Sanli A, Hardal U, Kibar S, Altin G, Erdogan BA, Bekmez ZE. How Drill-Generated Acoustic Trauma effects Hearing Functions in an Ear Surgery? Int J Head and Neck Surg 2012;3(3):127-132.

scholarly journals Does Drilling Induce Hearing Loss after Modified Radical Mastoidectomy? A Retrospective Study

2020 ◽  
Vol 42 (3) ◽  
pp. 38-41
Author(s):  
Yogesh Neupane ◽  
Bijaya Kharel ◽  
Heempali Dutta
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Bone Conduction ◽  
Sensorineural Hearing ◽  
Radical Mastoidectomy ◽  
Modified Radical Mastoidectomy ◽  
The Mean ◽  
Sensory Neural ◽  
Mastoid Surgery ◽  
Sensory Neural Hearing Loss

Introduction Incidence of sensory neural hearing loss following mastoid surgery varies from 1.2 – 4.5%.There are various causes for postoperative sensorineural hearing loss during mastoid surgery. This study aims to identify whether there is any correlation between drilling and postoperative sensory neural hearing loss. MethodsA retrospective study was conducted in the Department of ENT from January 2018 to June 2019. A total number of 68 patients above five years of age who underwent modified radical mastoidectomy for chronic otitis media squamous were included. Revision surgery, preoperative sensorineural hearing loss, injury to the ossicular chain during surgery, patients with lack of follow up or doubtful reports in mentally challenged were excluded from the study. The average bone conduction threshold was calculated from 500, 1000, 2000, 4000 Hz and compared using the Wilcoxon signed-rank test. ResultsThere were 43 males and 25 females in the study with a median age of 23.5 years (16-55). The mean preoperative bone conduction threshold in the four frequencies of 500 Hz, 1kHz, 2kHz, 4kHz were -2.06dB, -2.06dB, 3.31dB, 4.63 dB respectively and the mean postoperative bone conduction thresholds were 1.03, 1.32, 5.29, 4.04 respectively. There was a decline of mean of 3.09 dB and 3.38dB only at the low-frequencies (500Hz and 1kHz) BC threshold respectively which were statistically significant, whereas at higher frequency there was no decline in average postoperative BC threshold. ConclusionThere is no definite role of drill in inducing hearing loss and if present other causes of hearing loss should be sought in postoperative sensorineural hearing loss.

Sensorineural Hearing Loss in Chronic Ear Surgery

1977 ◽  
Vol 86 (1) ◽  
pp. 3-8 ◽  
Author(s):  
G. D. L. Smyth
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Clinical Evidence ◽  
Bone Conduction ◽  
Ossicular Chain ◽  
Sensorineural Hearing ◽  
Inflammatory Factors ◽  
Speech Discrimination ◽  
Ear Surgery ◽  
Cochlear Damage

The author's series of 3000 consecutive operations of tympanoplasty from 1960 to 1975 were reviewed in regard to the occurrence of sensorineural hearing loss as a consequence of the surgical procedure. Worsening of bone conduction thresholds by 10 dB through the frequencies 500 to 4000 cps, or a 10% reduction in speech discrimination scores were considered significant. Whereas in transcanal tympanoplasty the incidence of cochlear damage was greater in ears when initially the ossicular chain was incomplete, by contrast in combined approach tympanoplasty the risk was greater when the chain was intact initially. It was concluded that cochlear trauma was usually due to 1) the hydraulic effect of excessive stapes manipulation during the removal of disease, and 2) the development of a perilymph fistula. The unpredictable predisposing threat of cochlear fragility due to genetic and inflammatory factors was emphasized and the poor results of tympanoplasty in tympanosclerosis were underlined. The current methods of treating sensorineural hearing loss after tympanoplasty were enumerated and discussed. It was concluded that although those aimed at improving labyrinthine circulation had theoretical backing, there is as yet little experimental or clinical evidence to support the claims of their protagonists.

scholarly journals Towards a differential diagnosis of cochlear synaptopathy and outer-hair-cell deficits in mixed sensorineural hearing loss pathologies

2019 ◽  
Author(s):  
Viacheslav Vasilkov ◽  
Sarah Verhulst
Keyword(s):  
Hearing Loss ◽  
Differential Diagnosis ◽  
Hair Cell ◽  
Sensorineural Hearing Loss ◽  
Noise Exposure ◽  
Outer Hair Cell ◽  
Cell Damage ◽  
Sensorineural Hearing ◽  
Inner Hair Cell ◽  
Hearing Thresholds

AbstractDamage to the auditory periphery is more widespread than predicted by the gold-standard clinical audiogram. Noise exposure, ototoxicity and aging can destroy cochlear inner-hair-cell afferent synapses and result in a degraded subcortical representation of sound while leaving hearing thresholds unaffected. Damaged afferent synapses, i.e. cochlear synaptopathy, can be quantified using histology, but a differential diagnosis in living humans is difficult: histology cannot be applied and existing auditory evoked potential (AEP) metrics for synaptopathy become insensitive when other sensorineural hearing impairments co-exist (e.g., outer-hair-cell damage associated with elevated hearing thresholds). To develop a non-invasive diagnostic method which quantifies synaptopathy in humans and animals with normal or elevated hearing thresholds, we employ a computational model approach in combination with human AEP and psychoacoustics. We propose the use of a sensorineural hearing loss (SNHL) map which comprises two relative AEP-based metrics to quantify the respective degrees of synaptopathy and OHC damage and evaluate to which degree our predictions of AEP alterations can explain individual data-points in recorded SNHL maps from male and female listeners with normal or elevated audiometric thresholds. We conclude that SNHL maps can offer a more precise diagnostic tool than existing AEP methods for individual assessment of the synaptopathy and OHC-damage aspect of sensorineural hearing loss.Significance StatementHearing loss ranks fourth in global causes for disability and risk factors include noise exposure, ototoxicity and aging. The most vulnerable parts of the cochlea are the inner-hair-cell afferent synapses and their damage (cochlear synaptopathy) results in a degraded subcortical representation of sound. While synaptopathy can be estimated reliably using histology, it cannot be quantified this way in living humans. Secondly, other co-existing sensorineural hearing deficits (e.g., outer-hair-cell damage) can complicate a differential diagnosis. To quantify synaptopathy in humans and animals with normal or elevated hearing thresholds, we adopt a theoretical and interdisciplinary approach. Sensitive diagnostic metrics for synaptopathy are crucial to assess its prevalence in humans, study its impact on sound perception and yield effective hearing restoration strategies.

scholarly journals Sensorineural hearing loss in tubotympanic variety of chronic suppurative otitis media

2020 ◽  
Vol 22 (1) ◽  
pp. 26-30
Author(s):  
Dhiman Pramanik ◽  
Mesbah Uddin Ahmed ◽  
Abm Luthful Kabir ◽  
Mohammad Harun Or Rashid ◽  
Mohammad Anamul Haque ◽  
...  
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Otitis Media ◽  
Noise Exposure ◽  
Bone Conduction ◽  
Sensorineural Hearing ◽  
Chronic Suppurative Otitis Media ◽  
Threshold Elevation ◽  
Suppurative Otitis Media ◽  
Bone Conduction Threshold

Objectives: To determine whether chronic suppurative otitis media(CSOM) can cause Sensorineural hearing loss(SNHL) and to note its degree and its relation to duration of disease. Methods: This was a cross sectional study which was carried out in the departments of Otolaryngology and Head-Neck Surgery of Dhaka Medical College Hospital during the period of April’2012 to March’2013. A total number of 76 patients having unilateral CSOM were included in this study age ranging from 6 to 59 years after exclusion of other possible causes of SNHL such as meningitis, head injury, previous ear surgery and chronic noise exposure. The use of unilateral CSOM cases provided a contralateral ear to serve as a control. Bone conduction threshold elevation between diseased and control ear was considered as the indicator of inner ear damage. Results: In this series, CSOM was seen to be associated with sensorineural hearing loss. Significant threshold elevation was observed in relation to disease duration. Here bone conduction measurement in all frequencies (500Hz,1000Hz,2000Hz and 4000Hz) in diseased and uninvolved contralateral side showed elevation of bone conduction in diseased side and this elevation was significantly higher (p<0.001). The average bone conduction loss at 4kHz was higher than the average at the speech frequency range. Conclusion: This study suggests that CSOM can cause significant bone conduction threshold elevation and it should be considered when managing this problem. Early detection and prompt treatment may limit this potential handicap. Bangladesh J Otorhinolaryngol; April 2016; 22(1): 26-30

Transient Sensorineural Hearing Loss after Overuse of Portable Headphone Cassette Radios

1985 ◽  
Vol 93 (5) ◽  
pp. 622-625 ◽  
Author(s):  
Phillip C. Lee ◽  
Craig W. Senders ◽  
Bruce J. Gantz ◽  
Steven R. Otto
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Occupational Safety ◽  
Noise Exposure ◽  
Maximum Level ◽  
Acoustic Trauma ◽  
Sensorineural Hearing ◽  
Maximum Output ◽  
Health Administration ◽  
Listening Levels

Noise-induced sensorineural hearing loss has been associated with Industry for many years. One conservative estimate suggests that 10 million Americans may have industry-related, noise-induced hearing loss. Acoustic trauma from any source, whether associated with work or recreations, is detrimental to hearing. The Occupational Safety and Health Administration has set industrial standards for noise levels, with current standards limiting noise exposure to 95 dBA for 2 hours daily. To date, however, there are no recreational standards. Many portable headphone cassette radios produce peak outputs of more than 100 dBA. Temporary threshold shifts could result from listening levels near the maximum output. Permanent sensorineural loss may result with repeated exposure. A pilot study was conducted in which 16 volunteers listened to headphone sets for 3 hours at their usual maximum level. Six volunteers showed transient shifts of 10 dB, and one volunteer showed a transient shift of approximately 30 dB. These shifts returned to normal within 24 hours. As expected, transient shifts frequently occur with recreational use. Therefore, recreational warnings and standards should be established.

Sensorineural Hearing Loss After Radiotherapy and Chemoradiotherapy: A Single, Blinded, Randomized Study

2006 ◽  
Vol 24 (12) ◽  
pp. 1904-1909 ◽  
Author(s):  
Wong Kein Low ◽  
Song Tar Toh ◽  
Joseph Wee ◽  
Stephanie M.C. Fook-Chong ◽  
De Yun Wang
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Randomized Study ◽  
Bone Conduction ◽  
Hearing Threshold ◽  
Sensorineural Hearing ◽  
First Year ◽  
Radiotherapy Group ◽  
Hearing Thresholds ◽  
Radiotherapy Alone

Purpose The synergistic ototoxicity of radiation and cisplatin (CDDP) has not been adequately studied. This study investigated whether the use of concurrent and postradiotherapy CDDP in patients with nasopharyngeal carcinoma (NPC) resulted in a difference in postradiotherapy sensorineural hearing when compared with the use of radiotherapy alone. Patients and Methods Newly diagnosed patients were randomly assigned to the radiotherapy or chemoradiotherapy groups. Bone conduction hearing thresholds were performed before treatment and at 1 week, 6 months, 1 year, and 2 years after completion of radiotherapy. Statistical analysis was performed using the Mann-Whitney U test. Results Hearing thresholds averaged over 0.5, 1, and 2 kHz were found to be poorer in the chemoradiotherapy group (58 patients) compared with the radiotherapy group (57 patients) at 1 year (P = .001) and 2 years (P = .03) after radiotherapy. Hearing thresholds at 4 kHz were significantly worse for patients in the chemoradiotherapy arm at all of the postradiotherapy time points studied and were more severely affected than the thresholds at lower speech frequencies. In the radiotherapy group, deterioration of median hearing thresholds, which occurred in the immediate post-treatment period, improved within the first year but deteriorated again at 2 years. In the chemoradiotherapy group, median hearing threshold deterioration, which started immediately after radiotherapy, stabilized by 1 year. Conclusion Patients with NPC who received radiotherapy and concurrent/adjuvant chemotherapy using CDDP experienced greater sensorineural hearing loss compared with patients treated with radiotherapy alone, especially to high-frequency sounds in the speech range. Normal inner ear tissue tolerance, which was once defined only for radiotherapy patients alone, should be redefined in chemoradiotherapy patients.

scholarly journals Sensorineural component in chronic suppurative otitis media

1970 ◽  
Vol 15 (2) ◽  
pp. 69-74
Author(s):  
Ahmed Raquib ◽  
Ahmmad Taous ◽  
Rojibul Haque
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Otitis Media ◽  
Noise Exposure ◽  
Bone Conduction ◽  
Sensorineural Hearing ◽  
Chronic Suppurative Otitis Media ◽  
Threshold Elevation ◽  
Suppurative Otitis Media ◽  
Bone Conduction Threshold

A cross sectional study was conducted at the Department of Otolaryngology and Head-Neck surgery, Dhaka Medical College Hospital, Dhaka during the period of July 2005 to June 2007 to determine whether chronic suppurative otitis media (CSOM) can cause sensorineural hearing loss (SNHL) and to note its degree and its relation to duration of disease. 130 patients with unilateral CSOM were included in the study age ranging from 11 to 50 years after exclusion of other possible causes of SNHL such as meningitis, head injury, previous ear surgery, and chronic noise exposure. The use of unilateral CSOM cases provided a contralateral ear to serve as a control thus excluded common variables, such as presbycusis, parenteral ototoxic medications, metabolic disorders, and the effect of sex or genetics. Bone conduction threshold elevation between diseased and control ear was considered as the indicator of inner ear damage. In this series, CSOM is seen to be associated with sensorineural hearing loss. The study showed a bone conduction threshold elevation from 4.1dB to 10.7dB across frequency ranges. Significant threshold elevation was observed in relation to the disease duration. The average bone conduction loss at 4 kHz was higher than the average at the speech frequency range. This study suggests that CSOM can cause significant bone conduction threshold elevation and it should be considered when managing this problem. Early detection and prompt treatment may limit this potential handicap. Key words: CSOM; Sensorineural hearing loss; Bone conduction DOI: 10.3329/bjo.v15i2.5060 Bangladesh J Otorhinolaryngol 2009; 15(2): 69-74

scholarly journals Characteristics of sensorineural hearing loss secondary to inner ear acoustic trauma

2008 ◽  
Vol 136 (5-6) ◽  
pp. 221-225
Author(s):  
Slobodan Spremo ◽  
Zdenko Stupar
Keyword(s):  
Hearing Loss ◽  
Sensorineural Hearing Loss ◽  
Hearing Threshold ◽  
Acoustic Trauma ◽  
Sensorineural Hearing ◽  
Cochlear Damage ◽  
Type 3 ◽  
Trauma Group

INTRODUCTION Cochlear damage secondary to exposure to acoustic trauma is the consequence of the acoustic energy effects on the hearing cells in Korti's organ. OBJECTIVE The objective was to assess the correlation between the degree of sensorineural hearing loss and the type of audiogram registered in acoustic trauma exposed patients. METHOD We analyzed 262 audiograms of patients exposed to acoustic trauma in correlation to 146 audiograms of patients with cochlear damage and hearing loss not related to acoustic trauma. "A" group consisted of acoustic trauma cases, while "B" group incorporated cases with hearing loss secondary to cochlear ischaemia or degeneration. All audiograms were subdivided with regard to the mean hearing loss into three groups: mild (21-40 dB HL), moderate (41-60 dB HL) and severe (over 60 dB HL) hearing loss. Based on audiogram configuration five types of audiogram were defined: type 1 flat; type 2 hearing threshold slope at 2 kHz, type 3 hearing threshold slope at 4 kHz; type 4 hearing threshold notch at 2 kHz; type 5 notch at 4 kHz. RESULTS Mild hearing loss was recorded in 163 (62.2%) ears in the acoustic trauma group, while in 78 (29.8%) ears we established moderate hearing loss with the maximum threshold shift at frequencies ranging from 4 kHz to 8 kHz. The least frequent was profound hearing loss, obtained in 21 (8%) audiograms in the acoustic trauma group. Characteristic audiogram configurations in the acoustic trauma patient group were: type 1 (N=66; 25.2%), type 2 (N=71; 27.1%), and type 3 (N=68; 25.9%). Audiogram configurations were significanly different in the acoustic trauma group in comparison to the cochlear ischaemia group of patients (p=0.0005). CONCLUSION Cochlear damage concomitant to acoustic trauma could be assessed by the audiogram configuration. Preserved hearing acuity at low and mild frequency range indicates the limited damage to the hearing cells in Korti's organ in the apical cochlear turn.

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