On the question of inflammation of the lumbosacral plexus

1929 ◽  
Vol 25 (9) ◽  
pp. 937-944
Author(s):  
G. V. Pervushin
Keyword(s):  
Nervous System ◽  
Brachial Plexus ◽  
Peripheral Nervous System ◽  
Lumbosacral Plexus

Until recently, neuropathologists have been paying great attention to diseases of the peripheral nervous system. Among these diseases, there are often cases of damage to the brachial and lumbosacral plexus. Inflammations of the brachial plexus are observed more often than the lumbosacral plexus, and a number of works are devoted to their study (Dricheue, Erb, Oppenheim, Flaubert, Dejerine - Klumpke, Dejerine, Gysi, Bernehardt, Unger, Kramer, etc.). The question of inflammation of the lumbosacral plexus is poor in literature and, in our opinion, has not been sufficiently developed to date.

Peripheral Nervous SystemUpper Extremity

2013 ◽  
Author(s):  
Adam Fisch
Keyword(s):  
Nervous System ◽  
Brachial Plexus ◽  
Median Nerve ◽  
Peripheral Nervous System ◽  
Ulnar Nerve ◽  
Radial Nerve ◽  
Upper Extremities ◽  
Cervical Plexus

Chapter 3 discusses how to draw the peripheral nervous system (upper extremities), including the brachial plexus, median nerve, ulnar nerve, radial nerve, and the cervical plexus.

Neurosurgery of the peripheral nervous system: entrapment syndromes of the brachial plexus

2000 ◽  
Vol 53 (1) ◽  
pp. 82-85 ◽  
Author(s):  
Eduardo Fernandez ◽  
Roberto Pallini ◽  
Enrico Marchese ◽  
Liverana Lauretti ◽  
Paolo Palma ◽  
...  
Keyword(s):  
Nervous System ◽  
Brachial Plexus ◽  
Peripheral Nervous System ◽  
Entrapment Syndromes

Peripheral Nervous SystemLower Extremity

2013 ◽  
Author(s):  
Adam Fisch
Keyword(s):  
Nervous System ◽  
Peripheral Nervous System ◽  
Lower Extremities ◽  
Lumbosacral Plexus

Chapter 4 discusses how to draw the peripheral nervous system (lower extremities), including the lumbosacral plexus, the leg and foot, and the thigh.

scholarly journals Differential diagnosis of flaccid palsy of the upper extremities in children first months after birth (Literature review)

2021 ◽  
Vol 9 (1) ◽  
pp. 115-126
Author(s):  
Olga E. Agranovich ◽  
Galina A. Ikoeva ◽  
Elena L. Gabbasova ◽  
Ekaterina V. Petrova ◽  
Vladimir M. Kenis ◽  
...  
Keyword(s):  
Nervous System ◽  
Differential Diagnosis ◽  
Brachial Plexus ◽  
Peripheral Nervous System ◽  
Motor Neurons ◽  
Peripheral Nerves ◽  
Upper Extremities ◽  
Adequate Treatment ◽  
Traumatic Origin ◽  
Children First

This article analyzes the literature related to flaccid paresis and paralysis of the upper extremities in children during the first months of life. This pathology is a heterogeneous group of diseases with different etiopathogenesis. There are various courses of flaccid paresis and paralysis of the upper extremities in children: damage to the spinal cord, brachial plexus, peripheral nervous system to the level of the brachial plexus, and isolated damage to peripheral nerves. According to the time of occurrence, flaccid paresis and paralysis can be divided into three groups: antenatal, intranatal, and postnatal pathology. The main mechanism of occurrence of this pathology is intranatal trauma. More rare causes of flaccid paresis and paralysis of the upper extremities are antenatal conditions of dysplastic and traumatic origin, postnatal damage to the peripheral nervous system due to trauma or infection. Congenital contractures of the upper extremities combined with flaccid paralysis are connected with genetically determined diseases of the lower motor neurons and congenital myopathies, intrauterine injuries of the brachial plexus peripheral nerves. This article discusses the issues of topical and differential diagnosis of this pathology, the clinical picture suitable for each period of the childs life, and the prognosis of the disease. This research will be useful not only for neurologists, but also for specialists of related specialties: orthopedists, physiotherapists, and neonatologists for making correct the diagnosis, providing adequate treatment, and predicting its results.

Positioning in Anesthesiology

2002 ◽  
Vol 97 (1) ◽  
pp. 75-81 ◽  
Author(s):  
Michel W. Coppieters ◽  
Marc Van De Velde ◽  
Karel H. Stappaerts
Keyword(s):  
Nervous System ◽  
Brachial Plexus ◽  
Range Of Motion ◽  
Peripheral Nervous System ◽  
Shoulder Girdle ◽  
Shoulder Abduction ◽  
Neutral Position ◽  
Cumulative Impact ◽  
Experimental Findings ◽  
The Impact

Background Stretch-induced neuropathy of the brachial plexus and median nerve in conventional perioperative care remains a relatively frequent and poorly understood complication. Guidelines for positioning have been formulated, although the protective effect of most recommendations remains unexamined. The similarity between the stipulated potentially dangerous positions and the components of the brachial plexus tension test (BPTT) justified the analysis of the BPTT to quantify the impact of various arm and neck positions on the peripheral nervous system. Methods Four variations of the BPTT in three different shoulder positions were performed in 25 asymptomatic male participants. The impact of arm and neck positions on the peripheral nervous system was evaluated by analyzing the maximal available range of motion, pain intensity, and type of elicited symptoms during the BPTT. Results Cervical contralateral lateral flexion, lateral rotation of the shoulder and fixation of the shoulder girdle in a neutral position in combination with shoulder abduction, and wrist extension all significantly reduced the available range of motion. Elbow extension also challenged the nervous system substantially. A cumulative impact could be observed when different components were simultaneously added, and a neutralizing effect was noted when an adjacent region allowed for unloading of the nervous system. Conclusions The experimental findings support the experientially based guidelines for positioning. Especially when simultaneously applied, submaximal joint positions easily load the nervous system, which may substantially compromise vital physiologic processes in and around the nerve. Therefore, even when the positioning of all upper limb joints is carefully considered, complete prevention of perioperative neuropathy seems almost inconceivable.

Neurorehabilitation approaches for disorders of the peripheral nervous system

2015 ◽  
pp. 224-237
Author(s):  
William Huynh ◽  
Michael Lee ◽  
Matthew Kiernan
Keyword(s):  
Nervous System ◽  
Peripheral Nervous System ◽  
Functional Independence ◽  
Spinal Nerve ◽  
Neurological Deficits ◽  
Nerve Lesion ◽  
Lumbosacral Plexus ◽  
System P ◽  
Different Levels

Disorders of the peripheral nervous system comprise both traumatic and non-traumatic aetiologies that may involve different levels in the spinal cord, emerging spinal nerve roots, brachial or lumbosacral plexus, to the peripheral nerves. Lesions can be focal, as in most cases of traumatic aetiology, or multifocal or generalized as commonly seen in non-traumatic causes. Current rehabilitative strategies aim at reversing the underlying cause of the nerve lesion, whilst at the same time, treating the symptoms, and preventing complications. More specifically, the emphasis of rehabilitation is to maximize functional independence, locomotion, prevent physical deformity, facilitate integration into society, and overall, improve quality of life. This chapter provides a basic understanding of the pathophysiology of neurological deficits associated with lesions of the peripheral nervous system, followed by a systematic discussion of the general principles. The aim is to provide the healthcare professional with a template and practical approach to the delivery of such therapy.

Glucuronyl 3-sulfate occurs exclusively on distal unmyelinated terminals of selected sensory neurons in the peripheral nervous system

1995 ◽  
Vol 53 ◽  
pp. 958-959
Author(s):  
S.S. Spicer ◽  
B.A. Schulte
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Cerebral Cortex ◽  
Peripheral Nervous System ◽  
Sensory Neurons ◽  
Sensory Nerves ◽  
Tissue Antigens ◽  
The Central Nervous System ◽  
The Brain ◽  
Leu 7

Generation of monoclonal antibodies (MAbs) against tissue antigens has yielded several (VC1.1, HNK- 1, L2, 4F4 and anti-leu 7) which recognize the unique sugar epitope, glucuronyl 3-sulfate (Glc A3- SO4). In the central nervous system, these MAbs have demonstrated Glc A3-SO4 at the surface of neurons in the cerebral cortex, the cerebellum, the retina and other widespread regions of the brain.Here we describe the distribution of Glc A3-SO4 in the peripheral nervous system as determined by immunostaining with a MAb (VC 1.1) developed against antigen in the cat visual cortex. Outside the central nervous system, immunoreactivity was observed only in peripheral terminals of selected sensory nerves conducting transduction signals for touch, hearing, balance and taste. On the glassy membrane of the sinus hair in murine nasal skin, just deep to the ringwurt, VC 1.1 delineated an intensely stained, plaque-like area (Fig. 1). This previously unrecognized structure of the nasal vibrissae presumably serves as a tactile end organ and to our knowledge is not demonstrable by means other than its selective immunopositivity with VC1.1 and its appearance as a densely fibrillar area in H&E stained sections.

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