Social Crowding and Individualss Risk Taking: A Life-History Theory Perspective

2018 ◽  
Author(s):  
Uwe mname Messer ◽  
Alexander mname Leischnig
Author(s):  
Steven L. Neuberg ◽  
Andreana C. Kenrick

How does being discriminated against affect one’s health, and through what mechanisms? Most research has focused on two causal pathways, highlighting how discrimination increases psychological stress and exposure to neighborhood hazards. This chapter advances an alternative, complementary set of mechanisms through which stigma and discrimination may shape health. Grounded in evolutionary biology’s life history theory, the framework holds that discrimination alters aspects of the physical and social ecologies in which people live (e.g., sex ratio, unpredictable extrinsic causes of mortality). These discriminating ecologies pull for specific behaviors and physiological responses (e.g., risk-taking, sexual activity, offspring care, fat storage) that are active, strategic, and rational given the threats and opportunities afforded by these ecologies but that also have downstream implications for health. This framework generates a wide range of nuanced insights and unique hypotheses about the discrimination-health relationship, and suggests specific approaches to intervention while pointing to complex ethical issues.


2019 ◽  
Vol 42 ◽  
Author(s):  
Boris Kotchoubey

Abstract Life History Theory (LHT) predicts a monotonous relationship between affluence and the rate of innovations and strong correlations within a cluster of behavioral features. Although both predictions can be true in specific cases, they are incorrect in general. Therefore, the author's explanations may be right, but they do not prove LHT and cannot be generalized to other apparently similar processes.


Author(s):  
Paul W Turke

Abstract The severity of COVID-19 is age-related, with the advantage going to younger age groups. Five reasons are presented. The first two are well-known, are being actively researched by the broader medical community, and therefore are discussed only briefly here. The third, fourth, and fifth reasons derive from evolutionary life history theory, and potentially fill gaps in current understanding of why and how young and old age groups respond differently to infection with SARS-CoV-2. Age of onset of generalized somatic aging, and the timing of its progression, are identified as important causes of these disparities, as are specific antagonistic pleiotropic tradeoffs in immune system function.


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