Oscillations or Cuff-Arterial Pulses?

2010 ◽  
Vol 44 (1) ◽  
pp. 6-6 ◽  
Author(s):  
Jiri Jilek
Keyword(s):  
1971 ◽  
Vol 27 (6) ◽  
pp. 708-709 ◽  
Author(s):  
Alvan R. Feinstein ◽  
◽  
Elliot Hochstein ◽  
Aldo A. Luisada ◽  
Joseph K. Perloff ◽  
...  

1975 ◽  
Vol 353 (1) ◽  
pp. 67-81 ◽  
Author(s):  
Thomas Kenner
Keyword(s):  

1965 ◽  
Vol 208 (5) ◽  
pp. 968-983 ◽  
Author(s):  
John W. Remington

The carotid and the subclavian arterial-systems were treated as though composed of a series of homogeneous distensible tubes joined in series or in parallel. With minor corrections, the contour of the aortic arch pulse was taken as that of a single transient incident wave entering the proximal end of these systems. Pulse height changes were localized to the junctions between coupled tubes of dissimilar wave impedance values. The amount of pressure change at these junctions was calculated by comparing arterial pulses recorded from various sites in these systems with the aortic pulse. An attempt was made to keep a strict accounting of all reflected waves arising either from the junction points or from peripheral drainage beds. The pulse constructions provided evidence for appreciable junctional positive reflections which alter the contour of the peripheral pulses. Because returning reflected waves from these sites were reflected negatively at each junction, their amplitude on entering the aorta was relatively small. The good-to-excellent reconstructions of the actual pulse contours suggest that the method of analysis used has promise, and that the basic assumptions on which the calculation of reflection coefficients was based did not impose errors of critical importance.


1999 ◽  
Vol 4 (4) ◽  
pp. 140-141 ◽  
Author(s):  
Sandra Hutto Faria

Author(s):  
Asawari Meshram ◽  
Vaishali Tembhare ◽  
Seema Singh ◽  
Ranjana Sharma ◽  
Ruchira Ankar ◽  
...  

Chiari Malformation is a rare condition. A condition known as Chiari malformation occurs when brain tissue spreads into the spinal canal. When a portion of your skull is excessively small or malformed, it presses on your brain and forces it downward. Chiari malformation is a rare occurrence, although the increased use of imaging testing has resulted in more diagnosis. Case Presentation: A 18-year-old boy was admitted to the hospital with the following symptoms: Tingling sensation, numbness over left hand since 2 to 3 months. Neck bend toward right side, pain in left hand since 6 month. Difficulty during eating by hand since 2 to 3 month. On physical examination, indicated a bright attentive person with pale conjunctiva and no symptoms of icterus. He had a tachycardia, bilateral pitting pedal edema and a swollen abdomen with shifting dullness, all of which pointed to as cites. He had a history of intermittent abdominal pain. On admission he complaint of new onset of dyspnea on exertion, fatigue and abdominal swelling. The rest of all physical examination was normal, with no skin changes and an intact arterial pulses in all four extremities. Conclusion: The primary focus of this case study is on professional management and outstanding nursing care, which may provide the holistic care that Chiari Syndrome necessitates while also effectively managing the challenging case. After a full recovery, the patient's comprehensive health care team collaborates to help the patient regain his or her previous level of independence and satisfaction.


2019 ◽  
Vol 39 (01) ◽  
pp. 006-019 ◽  
Author(s):  
Theodore Warkentin

AbstractRelatively little scientific attention has been given to the small subset of critically ill patients with circulatory shock who develop ischaemic limb losses (symmetrical peripheral gangrene [SPG]). The clinical picture consists of acral (distal extremity) tissue necrosis involving lower limbs in a largely symmetrical fashion and with detectable arterial pulses; in one-third of patients the upper extremities are also affected (potential for four-limb amputations). The laboratory picture includes thrombocytopenia, coagulopathy, and normoblastemia (circulating nucleated red blood cells). The explanation for limb losses is microvascular thrombosis caused by disseminated intravascular coagulation usually secondary to cardiogenic or septic shock. A common myth is that vasopressors cause the ischaemic limb injury. However, the more likely explanation is failure of the natural anticoagulant systems (protein C and antithrombin) to downregulate thrombin generation in the microvasculature. This is because more than 90% of patients with SPG have preceding ‘shock liver’, which occurs 2 to 5 days (median, 3 days) prior to ischaemic limb injury, with impaired hepatic production of protein C and antithrombin.


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