scholarly journals Does Hypovitaminosis A in Mothers Cause Congenital Malformation of the Eye?

2020 ◽  
Vol 3 (11) ◽  
pp. 04-07
Author(s):  
Shreya Shah ◽  
Mehul Shah ◽  
Ashit Desai ◽  
Mrugesha Vaghela ◽  
Deeksha Thorat
Keyword(s):  
Congenital Malformation ◽  
Hypovitaminosis A

scholarly journals Does hypovitaminosis A in mothers cause congenital malformation of eye?

2020 ◽  
Author(s):  
MEHUL SHAH ◽  
SHREYA SHAH ◽  
Ashit Desai ◽  
Ronuaq Khanna ◽  
Chetan Rathod
Keyword(s):  
Cohort Study ◽  
Congenital Malformation ◽  
Retrospective Cohort ◽  
Night Blindness ◽  
Pediatric Patients ◽  
Clinical Observation ◽  
Vitamin A Deficiency ◽  
Pediatric Ophthalmology ◽  
History Of ◽  
Hypovitaminosis A

Abstract Introduction: Congenital malformation is important cause of child hood blindness, we would like to study factors responsible for it.Methods: This was retrospective cohort study We collected data from our hospital EMR from 2008 to 2019.WE retrieved total number of Pediatric patients outdoor and children with congenital malformed eyes. We also explored history of night blindness in mothers during pregnancy we exported data in excel sheet , type of anomalies calculated using descriptive analyses of SPSS 22.Results: Our cohort consisted of 1435(1.42%) children out of 100946 of total outdoor of pediatric ophthalmology. We found various anomalies in our analyses, out of total 423(29.47%) had history of night blindness to mother during pregnancy suggesting vitamin A deficiency. Conclusion: Hypovitaminosis A in mother has influence maldevelopment and congenital anomalies of eye based on clinical observation.

The Effect of Vitamin A on the Intermittent Nitrogen Dioxide Gas Exposure in Hamsters

1976 ◽  
Vol 34 ◽  
pp. 176-177
Author(s):  
J.C.S. Kim ◽  
M.G. Jourden ◽  
E.S. Carlisle
Keyword(s):  
Electron Microscopy ◽  
Vitamin A ◽  
Nitrogen Dioxide ◽  
Chronic Exposure ◽  
Light And Electron Microscopy ◽  
Specific Effect ◽  
Deficient Diet ◽  
Intermittent Exposure ◽  
Hypovitaminosis A ◽  
Gas Exposure

Chronic exposure to nitrogen dioxide in rodents has shown that injury reaches a maximum after 24 hours, and a reparative adaptive phase follows (1). Damage occurring in the terminal bronchioles and proximal portions of the alveolar ducts in rats has been extensively studied by both light and electron microscopy (1).The present study was undertaken to compare the response of lung tissue to intermittent exposure to 10 ppm of nitrogen dioxide gas for 4 hours per week, while the hamsters were on a vitamin A deficient diet. Ultrastructural observations made from lung tissues obtained from non-gas exposed, hypovitaminosis A animals and gas exposed animals fed a regular commercially prepared diet have been compared to elucidate the specific effect of vitamin A on nitrogen dioxide gas exposure. The interaction occurring between vitamin A and nitrogen dioxide gas has not previously been investigated.

CLINICAL AND PARA-CLINICAL CHARACTERISTICS OF SICK NEWBORN BABIES TREATED AT PEDIATRIC DEPARTMENT OF HUE UNIVERSITY HOSPITAL

2011 ◽  
pp. 112-117
Author(s):  
Thi Kieu Nhi Nguyen
Keyword(s):  
Respiratory Distress ◽  
Congenital Malformation ◽  
Clinical Characteristics ◽  
Descriptive Study ◽  
Neonatal Unit ◽  
University Hospital ◽  
Pediatric Department ◽  
Sick Newborn ◽  
Observational Descriptive Study

Objectives: 1. Describe neonatal classification of WHO. 2. Identify some principal clinical and paraclinical signs of term, preterm, post term babies. Patients and method: an observational descriptive study of 233 newborns hospitalized in neonatal unit at Hue university‘ s hospital was done during 12 months from 01/01/2009 to 31/12/2009 for describing neonatal classification and identifying principal clinical and paraclinical signs. Results: Premature (16.74%); Term babies (45.5%); Post term (37.76%); Premature: asphyxia (43.59%), hypothermia (25.64%), vomit (30.77%), jaundice (61.54%), congenital malformation (17.95%); CRP > 10mg/l (53.85%); anemia Hb < 15g/dl (12.82%). Term babies: poor feeding (21.7%); fever (24.53%); CRP > 10mg/l (53.77%); Hyperleucocytes/ Leucopenia (35.85%). Post term: respiratory distress (34%); lethargy (29.55%); vomit (26.14%); polycuthemia (1.14%); hypoglycemia (22.73%). Conclusion: each of neonatal type classified by WHO presente different clinical and paraclinical. Signs. The purpose of this research is to help to treat neonatal pathology more effectively.

scholarly journals 958: Congenital malformation rates in a contemporary cohort with type 1 and type 2 diabetes mellitus

2020 ◽  
Vol 222 (1) ◽  
pp. S595
Author(s):  
Robert Martin ◽  
Elaine Duryea ◽  
Anne Ambia ◽  
Angela R. Seasely ◽  
Donald D. McIntire ◽  
...  
Keyword(s):  
Diabetes Mellitus ◽  
Type 2 Diabetes ◽  
Type 2 Diabetes Mellitus ◽  
Congenital Malformation

Congenital malformation syndromes and elevation of amniotic fluid α1-fetoprotein

Teratology ◽  
1981 ◽  
Vol 24 (2) ◽  
pp. 125-130 ◽  
Author(s):  
Uta Burck ◽  
Karsten R. Held ◽  
Hans-J. Kitschke ◽  
Martin Carstensen
Keyword(s):  
Amniotic Fluid ◽  
Congenital Malformation ◽  
Malformation Syndromes

scholarly journals Bilateral Choanal Atresia and Endoscopic Surgery: A Chance for CHARGE Patients

2021 ◽  
Vol 10 (13) ◽  
pp. 2951
Author(s):  
Maria Baldovin ◽  
Diego Cazzador ◽  
Claudia Zanotti ◽  
Giuliana Frasson ◽  
Athanasios Saratziotis ◽  
...  
Keyword(s):  
Congenital Malformation ◽  
Endoscopic Surgery ◽  
Granulation Tissue ◽  
Choanal Atresia ◽  
Tissue Formation ◽  
Endoscopic Repair ◽  
Granulation Tissue Formation ◽  
Charge Association ◽  
Cohort Data

Bilateral choanal atresia (CA) is a rare congenital malformation frequently associated with other anomalies. CHARGE association is closely linked to bilateral CA. The aim of this study was to describe the outcomes of the endoscopic repair in bilateral CA, and to assess the role of postoperative nasal stenting in two cohorts of CHARGE-associated and non-syndromic CA. Thirty-nine children were retrospectively analyzed (16 patients had CHARGE-associated CA). The rate of postoperative neochoanal restenosis was 31.3% in the CHARGE population, and 47.8% in the non-syndromic CA cohort. Data on postoperative synechiae and granulation tissue formation, need for endonasal toilette and dilation procedures, and number of procedures per patient were presented. Stent positioning led to a higher number of postoperative dilation procedures per patient in the non-syndromic cohort (p = 0.018), and to a higher rate of restenosis both in the CHARGE-associated, and non-syndromic CA populations. Children with CHARGE-associated and non-syndromic bilateral CA benefitted from endonasal endoscopic CA correction. The postoperative application of an endonasal stent should be carefully evaluated.

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