Adherence to cardiovascular prevention strategies in patients with rheumatoid arthritis

2015 ◽  
Vol 44 (6) ◽  
pp. 443-448 ◽  
Author(s):  
DF van Breukelen-van der Stoep ◽  
J Zijlmans ◽  
D van Zeben ◽  
B Klop ◽  
GJM van de Geijn ◽  
...  
Keyword(s):  
Rheumatoid Arthritis ◽  
Cardiovascular Prevention ◽  
Prevention Strategies

scholarly journals Lipid management in rheumatoid arthritis: a position paper of the Working Group on Cardiovascular Pharmacotherapy of the European Society of Cardiology

2019 ◽  
Vol 6 (2) ◽  
pp. 104-114 ◽  
Author(s):  
Ivana Hollan ◽  
Nicoletta Ronda ◽  
Patrick Dessein ◽  
Stefan Agewall ◽  
George Karpouzas ◽  
...  
Keyword(s):  
Rheumatoid Arthritis ◽  
Cardiovascular Prevention ◽  
Optimal Strategies ◽  
Position Paper ◽  
Related Factors ◽  
Lipid Management ◽  
Expert Opinions ◽  
Cardiovascular Pharmacotherapy ◽  
European Society Of Cardiology ◽  
Lipid Parameters

Abstract Rheumatoid arthritis (RA) is associated with increased cardiovascular morbidity, partly due to alterations in lipoprotein quantity, quality and cell cholesterol trafficking. Although cardiovascular disease significantly contributes to mortality excess in RA, cardiovascular prevention has been largely insufficient. Because of limited evidence, optimal strategies for lipid management (LM) in RA have not been determined yet, and recommendations are largely based on expert opinions. In this position paper, we describe abnormalities in lipid metabolism and introduce a new algorithm for estimation of cardiovascular risk (CVR) and LM in RA. The algorithm stratifies patients according to RA-related factors impacting CVR (such as RA activity and severity and medication). We propose strategies for monitoring of lipid parameters and treatment of dyslipidaemia in RA (including lifestyle, statins and other lipid-modifying therapies, and disease modifying antirheumatic drugs). These opinion-based recommendations are meant to facilitate LM in RA until more evidence is available.

Modeling the impact of cardiovascular prevention strategies

2012 ◽  
Vol 30 (1) ◽  
pp. 51-52
Author(s):  
François Gueyffier ◽  
Ivanny Marchant ◽  
Bo Carlberg
Keyword(s):  
Cardiovascular Prevention ◽  
Prevention Strategies ◽  
The Impact

scholarly journals What connection is there between intestinal microbiota and heart disease?

2020 ◽  
Vol 22 (Supplement_L) ◽  
pp. L117-L120
Author(s):  
Andrea Poli
Keyword(s):  
Intestinal Microbiota ◽  
Cardiovascular Prevention ◽  
Vascular Wall ◽  
Short Chain Fatty Acids ◽  
Prevention Strategies ◽  
Vascular Protection ◽  
Food Components ◽  
Trimethylamine Oxide ◽  
Protection Activity ◽  
Specific Food

Abstract Information on the correlation between intestinal microbiota and cardiovascular risk is growing. Some species of the microbiota influence the metabolism of specific food components (such as carnitine, choline, phosphatidyl-choline), synthesizing the precursor of trimethylamine oxide, a molecule with documented harmful activity on the vascular wall. Other strains, on the other hand, metabolize dietary fibre by synthesizing short-chain fatty acids, which have a significant anti-inflammatory activity, or produce secondary metabolites originating from molecules present in food (such as enterodiol, which derives from lignin), characterized by a vascular protection activity. Prebiotic effects from plant compounds (such as berberine or resveratrol) are also documented, which would induce favourable changes in the composition of the microbiota. The possibility of influencing the composition and activity of the intestinal microbiota will probably represent, in the future, an important component of cardiovascular prevention strategies.

scholarly journals Cardiovascular Prevention Strategies in Breast Cancer

2019 ◽  
Vol 1 (2) ◽  
pp. 322-325 ◽  
Author(s):  
Alexandra B. Yin ◽  
Abenaa M. Brewster ◽  
Ana Barac ◽  
Whittney Thoman ◽  
Kevin C. Oeffinger ◽  
...  
Keyword(s):  
Breast Cancer ◽  
Cardiovascular Prevention ◽  
Prevention Strategies

scholarly journals Rheumatoid arthritis versus diabetes as a risk factor for cardiovascular disease: a cross-sectional study, the CARRÉ Investigation

2008 ◽  
Vol 68 (9) ◽  
pp. 1395-1400 ◽  
Author(s):  
V P van Halm ◽  
M J L Peters ◽  
A E Voskuyl ◽  
M Boers ◽  
W F Lems ◽  
...  
Keyword(s):  
Rheumatoid Arthritis ◽  
Cardiovascular Disease ◽  
Risk Factor ◽  
Cardiovascular Risk ◽  
Cardiovascular Prevention ◽  
Arterial Disease ◽  
Population Based ◽  
Cross Sectional Study ◽  
Prevalence Odds Ratio ◽  
Cross Sectional

Objectives:Patients with rheumatoid arthritis (RA) have an increased cardiovascular risk, but the magnitude of this risk is not known precisely. A study was undertaken to investigate the associations between RA and type 2 diabetes (DM2), a well-established cardiovascular risk factor, on the one hand, and cardiovascular disease (CVD) on the other.Methods:The prevalence of CVD (coronary, cerebral and peripheral arterial disease) was determined in 353 randomly selected outpatients with RA (diagnosed between 1989 and 2001, aged 50–75 years; the CARRÉ study) and in participants of a population-based cohort study on diabetes and CVD (the Hoorn study). Patients with RA with normal fasting glucose levels from the CARRÉ study (RA, n = 294) were compared with individuals from the Hoorn study with normal glucose metabolism (non-diabetic, n = 258) and individuals with DM2 (DM2, n = 194).Results:The prevalence of CVD was 5.0% (95% CI 2.3% to 7.7%) in the non-diabetic group, 12.4% (95% CI 7.5% to 17.3%) in the DM2 group and 12.9% (95% CI 8.8% to 17.0%) in those with RA. With non-diabetic individuals as the reference category, the age- and gender-adjusted prevalence odds ratio (OR) for CVD was 2.3 (95% CI 1.1 to 4.7) for individuals with DM2 and 3.1 (95% CI 1.6 to 6.1) for those with RA. There was an attenuation of the prevalences after adjustment for conventional cardiovascular risk factors (OR 2.0 (95% CI 0.9 to 4.5) and 2.7 (95% CI 1.2 to 5.9), respectively).Conclusions:The prevalence of CVD in RA is increased to an extent that is at least comparable to that of DM2. This should have implications for primary cardiovascular prevention strategies in RA.

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