scholarly journals Cycle ergometer and rebound exercises with chest physiotherapy - a useful adjunct for sputum expectoration in mild to moderately symptomatic hiv infected children.

2008 ◽  
Vol 64 (3) ◽  
Author(s):  
S.S. Maharaj ◽  
P.M. Jeena

Background: Sputum expectoration of lung secretionsin HIV  infected children with associated respiratory conditions is oftendifficult. Chest physiotherapy is often recommended to assist in thisprocess but is not always successful. A erobic exercises may have bene-ficial effects on sputum expectoration but its safety is uncertain. Aim: The primary aim of this study was to determine if cycle ergometerand rebound exercises are safe for mild to moderately symptomatic HIV  infected children and if these aerobic exercises followed by chestphysiotherapy could augment sputum expectoration.Method: Thirty six A frican 8-12 year old males performed 15 minutesof either cycle ergometer or rebound exercises. A  modified 6 minutewalking test to ensure fitness of the enrolled subjects prior to randomization was performed. Heart and respiratoryrates, blood pressure and oxygen saturation was monitored for safety. Sputum expectorated was measured in a calibratedvial at baseline, 5, 10, and 15 minutes post exercise followed by 30 minutes of conventional chest physiotherapy. R esults: Total sputum produced during and post rebounding exercise was significantly higher than cycle ergometerexercises (12.6 vs. 9.8mls p=0.0002). The quantity of sputum obtained over each time point after rebound exercise wassignificantly more than cycle ergometer (5, 10 & 15 minutes; p=0.0084, p= 0.0002, p=0.0002 respectively). There wereno significant differences in heart and respiratory rates, blood pressure and oxygen saturation of enrolled subjectsbetween these exercises and no cases reached the threshold for stopping the exercise.Conclusion: Cycle ergometer and rebound exercises are safe for mild to moderately symptomatic HIV  infected children. Rebound exercises followed by chest physiotherapy can be used as a safe adjunct to significantly increasesputum expectoration.

2018 ◽  
Vol 25 (1) ◽  
pp. 23-30
Author(s):  
Jaime Della Corte ◽  
Gabriel Andrade Paz ◽  
Juliana Brandão Pinto de Castro ◽  
Humberto Miranda

Abstract Introduction. The aim of the study was to investigate the hypotensive responses in normotensive trained individuals after strength training (ST) performed using two training methods: DeLorme and Oxford. Material and methods. Fifteen normotensive trained men (age: 25.1 ± 3.2 years; height: 1.78 ± 0.01 m; BMI: 24.78 ± 1.4 kg/m2) were examined alternately in crossover with an interval of 72 hours between the DeLorme and Oxford methods. Firstly, 10 repetition maximum (RM) loads were obtained. DeLorme (50%, 70%, and 90%) and Oxford (90%, 70%, and 50%) consisted in the manipulation of 10RM loads so that the subjects performed three sets until failure in the Smith Machine (SM) and the Leg Press 45° (LP). Blood pressure was recorded at rest, post-exercise, and until 60 min post-session at 10-min intervals. Results. Both the DeLorme and Oxford methods showed significant intra-protocol reduction in systolic blood pressure (SBP) at 40, 50, and 60 min time points compared to the rest value (F = 21.848; p = 0.0001). Similar results were noted for diastolic blood pressure (DBP) between rest value and the 60 min time point (F = 46.113; p = 0.0001). Conclusions. The DeLorme and Oxford resistance training methods provided similar hemodynamic responses and similar hypotensive effects. Therefore, these methods can be used as alternatives to manipulate training intensity-volume ratio intra-session with low hemodynamic stress.


2013 ◽  
Vol 19 (3) ◽  
pp. 633-640 ◽  
Author(s):  
Ricardo Yukio Asano ◽  
Rodrigo Alberto Vieira Browne ◽  
Rafael da Costa Sotero ◽  
Marcelo Magalhães Sales ◽  
José Fernando Vila Nova de Moraes ◽  
...  

The purposes of this study were to analyze and compare the effects of exercise performed in different intensities, above and below lactate threshold (LT) on post-exercise blood pressure (BP) and nitric oxide (NO) responses in individuals with type 2 diabetes (T2D). For this, 11 T2D underwent the following sessions: 1) control session; 2) 20-min of moderate cycling (80% LT); and 3) 20-min of high intensity cycling (120%LT) on a cycle ergometer. Plasma NO and BP measurements were carried out at rest and at 15 and 45 min of post-sessions. When compared to rest, only the exercise session performed at 120%LT elicited an increase of NO (from 7.2 to 9.5 µM, p<0.05), as well as a decrease in systolic BP (from 126.6±7.9 to 118.7±3.9 mmHg, p<0.05) during the post-exercise period. In conclusion, the results suggest that NO release and post-exercise BP decrease are intensity-dependent for individuals with T2D.


2002 ◽  
Vol 93 (3) ◽  
pp. 882-886 ◽  
Author(s):  
Johan P. A. Andersson ◽  
Mats H. Linér ◽  
Elisabeth Rünow ◽  
Erika K. A. Schagatay

This study addressed the effects of apnea in air and apnea with face immersion in cold water (10°C) on the diving response and arterial oxygen saturation during dynamic exercise. Eight trained breath-hold divers performed steady-state exercise on a cycle ergometer at 100 W. During exercise, each subject performed 30-s apneas in air and 30-s apneas with face immersion. The heart rate and arterial oxygen saturation decreased and blood pressure increased during the apneas. Compared with apneas in air, apneas with face immersion augmented the heart rate reduction from 21 to 33% ( P < 0.001) and the blood pressure increase from 34 to 42% ( P < 0.05). The reduction in arterial oxygen saturation from eupneic control was 6.8% during apneas in air and 5.2% during apneas with face immersion ( P < 0.05). The results indicate that augmentation of the diving response slows down the depletion of the lung oxygen store, possibly associated with a larger reduction in peripheral venous oxygen stores and increased anaerobiosis. This mechanism delays the fall in alveolar and arterial Po 2 and, thereby, the development of hypoxia in vital organs. Accordingly, we conclude that the human diving response has an oxygen-conserving effect during exercise.


2017 ◽  
Vol 5 (3) ◽  
pp. 259
Author(s):  
Shireen H. Ramadhan ◽  
Shamil K. Talal ◽  
Wasfiya A. Moner

Tobacco smoke is enormously harmful to human health, there’s no safe way to smoke. The primary objective of this study is to analyze the role of tobacco smoke compounds and their ability to damage the cardiovascular system and, in particular, to interfere with blood pressure (Brachial and radial pressure), heart rate and partly on the percentage blood Oxygen saturation. A new device has been manufactured which is unique for measuring the level of smoke, to obtained privies readings, the device had fixed to the first reference level آ and starting up from it. The effect of smoking has been studied on (26) male passive (26) active smoker volunteer, (14) female passive and (14) female active smoker participants. The results has been showed that the blood pressure and heart rate has been increased with increasing the number of cigarettes in both genders for passive and active smokers. However, this effect for male was more pronounced comparing with females for passive and active smokers. In the case of oxygen saturation concentration percentage, for males the change of oxygen concentration percentage was not stable. In other words, it was fluctuated with the number of cigarettes. But for females the oxygen concentration was decreased but not too much. This means that this effect was not pronounced. this study found آ that the most pronounced effect has been shown by male’s comparison to females for both passive and active smokers. As well as in nonsmoker’s males, the relation between heart rate and smoke level is inversely proportional. While in male and female smokers and female nonsmokers the relation is proportional. In all cases the relation between the smoke level and time of smoking is inversely proportional. Finally males& females are affected differently by tobacco use; the sensitivities to smoke for males are higher than in females for passive and active smokers in both brachial and radial blood pressure measurement.


Author(s):  
Gabriel Kolesny Tricot ◽  
Fabiula Isoton Isoton Novelli ◽  
Lucieli Teresa Cambri

AbstractThis study aimed to assess whether obesity and/or maximal exercise can change 24 h cardiac autonomic modulation and blood pressure in young men. Thirty-nine men (n: 20; 21.9±1.8 kg·m−2, and n: 19; 32.9±2.4 kg·m−2) were randomly assigned to perform a control (non-exercise) and an experimental day exercise (after maximal incremental test). Cardiac autonomic modulation was evaluated through frequency domain heart rate variability (HRV). Obesity did not impair the ambulatory HRV (p>0.05), however higher diastolic blood pressure during asleep time (p=0.02; group main effect) was observed. The 24 h and awake heart rate was higher on the experimental day (p<0.05; day main effect), regardless of obesity. Hypotension on the experimental day, compared to control day, was observed (p<0.05). Obesity indicators were significantly correlated with heart rate during asleep time (Rho=0.34 to 0.36) and with ambulatory blood pressure(r/Rho=0.32 to 0.53). Furthermore, the HRV threshold workload was significantly correlated with ambulatory heart rate (r/Rho=− 0.38 to−0.52). Finally, ambulatory HRV in obese young men was preserved; however, diastolic blood pressure was increased during asleep time. Maximal exercise caused heart rate increase and 24h hypotension, with decreased cardiac autonomic modulation in the first hour, regardless of obesity.


2008 ◽  
Vol 14 (3) ◽  
pp. 353-361
Author(s):  
Geraldo Andrade Capuchinho-Júnior ◽  
Ricardo Marques Dias ◽  
Sônia Regina da Silva de Carvalho

1944 ◽  
Vol 79 (1) ◽  
pp. 9-22 ◽  
Author(s):  
Frank L. Engel ◽  
Helen C. Harrison ◽  
C. N. H. Long

1. In a series of rats subjected to hemorrhage and shock a high negative correlation was found between the portal and peripheral venous oxygen saturations and the arterial blood pressure on the one hand, and the blood amino nitrogen levels on the other, and a high positive correlation between the portal and the peripheral oxygen saturations and between each of these and the blood pressure. 2. In five cats subjected to hemorrhage and shock the rise in plasma amino nitrogen and the fall in peripheral and portal venous oxygen saturations were confirmed. Further it was shown that the hepatic vein oxygen saturation falls early in shock while the arterial oxygen saturation showed no alteration except terminally, when it may fall also. 3. Ligation of the hepatic artery in rats did not affect the liver's ability to deaminate amino acids. Hemorrhage in a series of hepatic artery ligated rats did not produce any greater rise in the blood amino nitrogen than a similar hemorrhage in normal rats. The hepatic artery probably cannot compensate to any degree for the decrease in portal blood flow in shock. 4. An operation was devised whereby the viscera and portal circulation of the rat were eliminated and the liver maintained only on its arterial circulation. The ability of such a liver to metabolize amino acids was found to be less than either the normal or the hepatic artery ligated liver and to have very little reserve. 5. On complete occlusion of the circulation to the rat liver this organ was found to resist anoxia up to 45 minutes. With further anoxia irreversible damage to this organ's ability to handle amino acids occurred. 6. It is concluded that the blood amino nitrogen rise during shock results from an increased breakdown of protein in the peripheral tissues, the products of which accumulate either because they do not circulate through the liver at a sufficiently rapid rate or because with continued anoxia intrinsic damage may occur to the hepatic parenchyma so that it cannot dispose of amino acids.


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