scholarly journals A CASE REPORT REGARDING MANAGEMENT OF KAMALA THROUGH AYURVEDA

2021 ◽  
Vol 9 (11) ◽  
pp. 2866-2869
Author(s):  
Charu Sharma
Keyword(s):  
Case Report ◽  
Clinical Manifestation ◽  
Liver Diseases ◽  
Biliary Obstruction ◽  
Dietary Habits ◽  
Liver Disorder ◽  
Hepatic Damage ◽  
Bilirubin Metabolism ◽  
Chief Complaints ◽  
Loss Of Appetite

Ayurveda is traditionally skilful in treating liver diseases for centuries. Although named Jaundice as a liver disorder was not mentioned in Ayurveda literature but based on common characteristics and Pathology, Kamala can be correlated with jaundice. Jaundice is a clinical manifestation of disorders of underlying bilirubin metabolism, hepa- tocellular dysfunction, or biliary obstruction. Jaundice occurs in settings of cholestasis or inability to effectively secrete bile as well as disorders of bilirubin metabolism and hepatocellular dysfunction. Today's lifestyle with un- hygienic and poor dietary habits and alcoholic habits, etc are responsible factors to promote hepatic damage which is clinically reflected as Kamala. This paper discusses a patient seen in the OPD of Kayachikitsa Quadra Institute of Ayurveda Roorkee Haridwar. Her chief complaints Udara shool (pain in the abdomen), Kshudha Mandhya (loss of appetite), Daurbalya (weakness), Hrullas (Nausea), Mutrapitata (yellow discolouration of urine, Vibhandha (constipation) for 15 days. This patient was effectively treated by the combination of Kutaki Churna, Triphala, Trivrita Churna, Bhunimba Churna, Arogya Vardhini Vati, Phalatrikadi Kashaya, Punarnava Mandoor and Liv 52. All the symptoms showed highly significant results. Hence it can be concluded that these medicines are very effective in patients with jaundice. Keywords: Udara shool, Kshudha mandhya, Daurbalya, Hrullas, Mutrapitata, Vibhandha.

OBTURATOR HERNIA: A CASE REPORT

2016 ◽  
pp. 106-109
Author(s):  
Hoang Minh Thi Nguyen ◽  
Huu Tri Nguyen ◽  
Thanh Thao Nguyen
Keyword(s):  
Computed Tomography ◽  
Case Report ◽  
Intestinal Obstruction ◽  
Key Words ◽  
Clinical Manifestation ◽  
Elderly Women ◽  
Obturator Hernia ◽  
Abdominal Hernia ◽  
Mechanical Obstruction

Obturator hernia is a rare pelvic hernia which accounts for 1% of all abdominal hernia. Clinical manifestation is ussually unspecific. Obturator hernia is often diagnosed by computed tomography or ultrasound. We present a case of obturator hernia in an elderly women who was successfully diagnosed and treated at Hue Univeristy of Medicine and Pharmacy. Key words: obturator hernia, mechanical obstruction, intestinal obstruction, Richter obturator hernia, strangulation

scholarly journals An incidental case of biliary fascioliasis with subtle clinical findings: US and MRCP findings

2013 ◽  
Vol 47 (2) ◽  
pp. 125-127
Author(s):  
Hakan Önder ◽  
Faysal Ekici ◽  
Emin Adin ◽  
Suzan Kuday ◽  
Hatice Gümüş ◽  
...  
Keyword(s):  
Case Report ◽  
Bile Duct ◽  
Magnetic Resonance ◽  
Biliary Obstruction ◽  
Fasciola Hepatica ◽  
Magnetic Resonance Cholangiopancreatography ◽  
Clinical Findings ◽  
Imaging Studies ◽  
Laboratory Findings ◽  
Acoustic Shadow

Background. Fascioliasis is a disease caused by the trematode Fasciola hepatica. Cholangitis is a common clinical manifestation. Although fascioliasis may show various radiological and clinical features, cases without biliary dilatation are rare. Case report. We present unique ultrasound (US) and magnetic resonance cholangiopancreatography (MRCP) findings of a biliary fascioliasis case which doesn’t have biliary obstruction or cholestasis. Radiologically, curvilinear parasites compatible with juvenile and mature Fasciola hepatica within the gallbladder and common bile duct were found. The parasites appear as bright echogenic structures with no acoustic shadow on US and hypo-intense curvilinear lesions on T2 weighted MRCP images. Conclusions. Imaging studies may significantly contribute to the diagnosis of patients with subtle clinical and laboratory findings, particularly in endemic regions.

Unusual cystic hamartomatous lung lesion with clinical manifestation of subpleural bullae in a woman of reproductive age: A case report

2015 ◽  
Vol 65 (10) ◽  
pp. 558-562
Author(s):  
Kenji Yorita ◽  
Takanori Ayabe ◽  
Eiichi Chosa ◽  
Noriko Uchino ◽  
Yasuhiro Nagatomo ◽  
...  
Keyword(s):  
Case Report ◽  
Clinical Manifestation ◽  
Lung Lesion ◽  
Reproductive Age

scholarly journals Catatonia, beyond a psychiatric syndrome

2017 ◽  
Vol 11 (2) ◽  
pp. 209-212
Author(s):  
Nathália Stela Visoná de Figueiredo ◽  
Débora Bartzen Moraes Angst ◽  
Antônio de Matos Lima Neto ◽  
Michel Ferreira Machado ◽  
Maria Sheila Guimarães Rocha ◽  
...  
Keyword(s):  
Case Report ◽  
Status Epilepticus ◽  
Clinical Manifestation ◽  
Sinus Thrombosis ◽  
Cerebral Venous Sinus Thrombosis ◽  
Venous Sinus ◽  
Nonconvulsive Status Epilepticus ◽  
Diagnostic Investigation ◽  
Venous Sinus Thrombosis ◽  
Psychiatric Syndrome

ABSTRACT Although catatonia is a well-known psychiatric syndrome, there are many possible systemic and neurological etiologies. The aim of this case report was to present a case of a patient with cerebral venous sinus thrombosis and infarction in which catatonia was the clinical manifestation of a possible nonconvulsive status epilepticus. To our knowledge, only one such case has been reported in the literature, which had a simplified diagnostic investigation. It is important to correctly recognize the organic cause underlying catatonia in order to treat the patient as soon as possible thereby improving outcome. Therefore, physicians need to update their knowledge on catatonia, recognizing that it can be part of a psychiatric or neurologic condition.

How dangerous Gilbert's syndrome is

2021 ◽  
pp. 8-12
Author(s):  
Roman Petrovich Stepchenkov
Keyword(s):  
Pathological Condition ◽  
Inhibitory Effect ◽  
Unconjugated Bilirubin ◽  
Breakdown Product ◽  
Gilbert’S Syndrome ◽  
Gilbert's Syndrome ◽  
Bilirubin Metabolism ◽  
Gallbladder Dyskinesia ◽  
Conjugation Process ◽  
Loss Of Appetite

Gilbert's syndrome is a benign (functional) hyperbilirubinemia, which is based on a hereditary disorder of bilirubin metabolism, as a result of which the concentration of unbound bilirubin can increase several times. Bilirubin, being a breakdown product of hemoglobin, circulates through the bloodstream, combining with albumin molecules. Such bilirubin is called indirect. In the endoplasmic reticulum, it is conjugated; the enzyme glucuronyltransferase is responsible for this process. In Gilbert's syndrome, as a result of insufficient production of this enzyme, the conjugation process is disrupted, and, as a result, the concentration of unconjugated bilirubin increases. According to statistics, this pathological condition is observed in about 5 % of Russians. This syndrome was first described in 1901 by the French physician Augustin Nicolas Gilbert, and was subsequently named after him. The literature also contains references to this syndrome, described as «constitutional hepatic dysfunction», «familial non-hemolytic hyperbilirubinemia», «idiopathic non-conjugated hyperbilirubinemia». Gilbert's syndrome is inherited in an autosomal recessive manner; men get ill 3–4 times more often than women. A number of scientists associate this with a possible inhibitory effect of testosterone on the enzyme UDP-GT1, which breaks down bilirubin. Clinically, Gilbert's syndrome is manifested by episodes of jaundice caused by an increase in the level of unconjugated bilirubin in the blood serum. Against the background of icterus of the sclera and skin, there is increased fatigue, the appearance of a feeling of bitterness in the mouth, loss of appetite, nausea, and sometimes vomiting. The association of Gilbert's syndrome with functional disorders of the biliary tract, in particular, with gallbladder dyskinesia, is often noted.

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