scholarly journals A shallow compaction model for Holocene Mississippi Delta sediments

Author(s):  
Claudia Zoccarato ◽  
Torbjörn E. Törnqvist ◽  
Pietro Teatini ◽  
Jonathan G. Bridgeman

Abstract. The extensive loss of land elevation and the consequent exposure to flood hazards are seriously threatening the long-term survival of the Mississippi Delta. Shallow compaction of the top soil is one of the major components contributing to the relative sea level rise. In the last decades, more subsidence measurements have become available and recent studies demonstrate that compaction of Holocene strata is dominant. Here we propose a novel application aimed at modeling the present-day shallow compaction due to consolidation processes in the top soil. Soil compaction is properly computed and accounts for the large soil grain motion and the delayed dissipation of pore-water overpressure. The grain motion is described by means of a Lagrangian approach with an adaptive mesh where the grid nodes follows the accretion/compaction processes. Model calibration is obtained from stratigraphic and geochrology information collected at the Myrtle Grove Subsidence Superstation, where a nearly 40 m-deep sediment core that penetrates the entire Holocene succession allows testing model results over long (millennial) timescales. Model validation with available observations from rod surface-elevation table – marker horizon (RSET-MH) data enables the model to predict future scenarios.

2002 ◽  
Vol 38 ◽  
pp. 9-19 ◽  
Author(s):  
Guy S Salvesen

The ability of metazoan cells to undergo programmed cell death is vital to both the precise development and long-term survival of the mature adult. Cell deaths that result from engagement of this programme end in apoptosis, the ordered dismantling of the cell that results in its 'silent' demise, in which packaged cell fragments are removed by phagocytosis. This co-ordinated demise is mediated by members of a family of cysteine proteases known as caspases, whose activation follows characteristic apoptotic stimuli, and whose substrates include many proteins, the limited cleavage of which causes the characteristic morphology of apoptosis. In vertebrates, a subset of caspases has evolved to participate in the activation of pro-inflammatory cytokines, and thus members of the caspase family participate in one of two very distinct intracellular signalling pathways.


2000 ◽  
Vol 111 (1) ◽  
pp. 363-370 ◽  
Author(s):  
Katsuto Takenaka ◽  
Mine Harada ◽  
Tomoaki Fujisaki ◽  
Koji Nagafuji ◽  
Shinichi Mizuno ◽  
...  

2001 ◽  
Vol 120 (5) ◽  
pp. A747-A748
Author(s):  
S DRESNER ◽  
A IMMMANUEL ◽  
P LAMB ◽  
S GRIFFIN

2006 ◽  
Vol 175 (4S) ◽  
pp. 355-355
Author(s):  
Manuel Eisenberg ◽  
John S. Lam ◽  
Rakhee H. Goel ◽  
Allan J. Pantuck ◽  
Robert A. Figlin ◽  
...  

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