Evaluation of Maize Inbred Lines for Iranian maize mosaic virus (IMMV) Resistance

2015 ◽  
Vol 8 (2) ◽  
pp. 1-12 ◽  
Author(s):  
A. Estakhr ◽  
B. Heidari ◽  
A. Dadkhodaie ◽  
K. Izadpanah
2003 ◽  
Vol 2 (1) ◽  
pp. 1-8 ◽  
Author(s):  
I.R.P. Souza ◽  
E. Oliveira ◽  
M.A. Peres ◽  
A.C. Oliveira ◽  
A.Á.C. Purcino

1981 ◽  
Vol 32 (5) ◽  
pp. 741 ◽  
Author(s):  
DM Persley ◽  
IF Martin ◽  
RS Greber

Maize inbred lines, derived from both Australian and exotic sources, and used in a breeding programme at Kairi, Qld, were screened for resistance to a Johnson grass strain of sugarcane mosaic virus (SCMV-Jg). There was a good correlation between ratings made following manual inoculation in a glasshouse and those following exposure to natural field infection. Seven lines were highly resistant in both glasshouse and field ratings. A further nine lines showed an intermediate level of resistance following manual inoculation and developed from 0 to 7 % infection under field conditions when a susceptible line developed 99 % infection. Data obtained following the manual inoculation of plants in segregating generations of crosses between resistant KL 57 and susceptible KL 9 were consistent with resistance being controlled by a single dominant gene. Six lines (Pa 405, CI 44, Tx 601, Oh 07, Oh 7B, 38-11) that were used as sources of resistance to maize dwarf mosaic virus, strain A (MDMV-A) in the U.S.A. developed natural infection levels of less than 15% with SCMV-Jg. Only Pa 405 was highly resistant to manual inoculation. Four of 11 SCMV-Jg resistant lines were also highly resistant to manual inoculations with the sugarcane, Sabi grass and Queensland blue couch grass strains.


Plant Disease ◽  
1997 ◽  
Vol 81 (2) ◽  
pp. 195-198 ◽  
Author(s):  
A. Marçon ◽  
S. M. Kaeppler ◽  
S. G. Jensen

High plains virus (HPV) is a pathogen that causes a severe disease, especially in susceptible corn genotypes. The virus is transmitted by the eriophyid mite Aceria tosichella, which also transmits wheat streak mosaic virus (WSMV). This often results in a mixed infection by these two viruses. Genetic variability for resistance to the HPV exists among maize inbred lines but has not been characterized. The disease reaction of 30 maize inbred lines to the mixed infection and to WSMV alone was characterized in this study. Evaluation was based on symptom expression and virus titer (HPV and WSMV), as measured by enzyme-linked immunosorbent assay. All lines showed some HPV symptoms, which ranged from a few visible spots to rapid plant death. HPV-resistant inbreds include B73 and B14. Susceptible inbreds include W64A, Wf9, H100, N213, N215, and N194. Five of the six inbreds most severely affected by HPV also had a high WSMV titer, although they displayed few symptoms when inoculated with WSMV alone. Inbred N194 was one of the genotypes most susceptible to HPV, but it showed no detectable WSMV titer. Seedlings of highly susceptible genotypes often died within 2 weeks of infection.


Author(s):  
O. E. Bradfute

Maize mosaic virus (MMV) causes a severe disease of Zea mays in many tropical and subtropical regions of the world, including the southern U.S. (1-3). Fig. 1 shows internal cross striations of helical nucleoprotein and bounding membrane with surface projections typical of many plant rhabdovirus particles including MMV (3). Immunoelectron microscopy (IEM) was investigated as a method for identifying MMV. Antiserum to MMV was supplied by Ramon Lastra (Instituto Venezolano de Investigaciones Cientificas, Caracas, Venezuela).


2014 ◽  
Vol 40 (5) ◽  
pp. 838 ◽  
Author(s):  
Chao CUI ◽  
Ju-Lin GAO ◽  
Xiao-Fang YU ◽  
Zhi-Jun SU ◽  
Zhi-Gang WANG ◽  
...  

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