Transcriptome Analysis
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2021 ◽  
Vol 173 ◽  
pp. 114092
Author(s):  
Jun Yan ◽  
Hong Wang ◽  
Ying Wang ◽  
Shuang Xu ◽  
Yanhui Wan ◽  
...  

Aquaculture ◽  
2022 ◽  
Vol 548 ◽  
pp. 737550
Author(s):  
Jian Zhu ◽  
Rui-Yu Du ◽  
Qin-Qin Liu ◽  
Li Luo ◽  
Shi-Mei Lin ◽  
...  

Aquaculture ◽  
2022 ◽  
Vol 547 ◽  
pp. 737527
Author(s):  
Yipeng Ren ◽  
Siying Fu ◽  
Wenhao Dong ◽  
Baoping Pan ◽  
Wenjun Bu

2021 ◽  
Vol 173 ◽  
pp. 114141
Author(s):  
Qingyan Ruan ◽  
Jingyi Wang ◽  
Chengyu Xiao ◽  
Yinkai Yang ◽  
Enhui Luo ◽  
...  

Author(s):  
shuxia li ◽  
Zhihao Cheng ◽  
Shiman Dong ◽  
Zhibo Li ◽  
Liangping Zou ◽  
...  

Long non-coding RNAs (lncRNAs) have been considered to be important regulators of gene expression in a range of biological processes in plants. A large number of lncRNAs have been identified in plants. However, most of their biological functions still remain to be determined. Here, we identified total 3 004 lncRNAs in cassava under normal or cold-treated conditions from Iso-seq data. We further characterized a lincRNA, CRIR1, as a novel positive regulator of the plant response to cold stress. CRIR1 can be significantly induced by cold treatment. Overexpression of CRIR1 in cassava enhanced the cold tolerance of transgenic plants. Transcriptome analysis demonstrated that CRIR1 regulates a range of cold stress-related genes in a CBF-independent pathway. We further found that CRIR1 RNA can interact with MeCSP5, a homolog of the cold shock protein that acts as RNA chaperones, indicating that CRIR1 may recruit MeCSP5 to improve the translation efficiency of mRNA. In summary, our study greatly extends the repertoire of lncRNAs in plants as well as its responding to cold stress. Moreover, it reveals a sophisticated mechanism by which CRIR1 regulates plant cold stress response by modulating the expression of stress-responsive genes and increasing the translational yield.


2021 ◽  
Author(s):  
Shahan Mamoor

In these brief notes we document work using published microarray data (1, 2) to pioneer integrative transcriptome analysis comparing vulvar carcinoma to its tissue of origin, the vulva. We report the differential expression of cyclic nucleotide gated channel beta 1, encoded by CNGB1, in cancer of the vulva. CNGB1 may be of pertinence to understanding transformation and disease progression in vulvar cancer (3).


2021 ◽  
Author(s):  
Shahan Mamoor

In these brief notes we document work using published microarray data (1, 2) to pioneer integrative transcriptome analysis comparing vulvar carcinoma to its tissue of origin, the vulva. We report the differential expression of chromosome 2 open reading frame 49, encoded by C2orf49, in cancer of the vulva. C2orf49 may be of pertinence to understanding transformation and disease progression in vulvar cancer (3).


2021 ◽  
Author(s):  
Shahan Mamoor

In these brief notes we document work using published microarray data (1, 2) to pioneer integrative transcriptome analysis comparing vulvar carcinoma to its tissue of origin, the vulva. We report the differential expression of RAB8B, member RAS oncogene family, encoded by RAB8B, in cancer of the vulva. RAB8B may be of pertinence to understanding transformation and disease progression in vulvar cancer (3).


2021 ◽  
Author(s):  
Shahan Mamoor

In these brief notes we document work using published microarray data (1, 2) to pioneer integrative transcriptome analysis comparing vulvar carcinoma to its tissue of origin, the vulva. We report the differential expression of serine/threonine kinase 17a, encoded by STK17A, in cancer of the vulva. STK17A may be of pertinence to understanding transformation and disease progression in vulvar cancer (3).


2021 ◽  
Author(s):  
Shahan Mamoor

In these brief notes we document work using published microarray data (1, 2) to pioneer integrative transcriptome analysis comparing vulvar carcinoma to its tissue of origin, the vulva. We report the differential expression of carbohydrate (chondroitin 4) sulfotransferase 11, encoded by CHST11, in cancer of the vulva. CHST11 may be of pertinence to understanding transformation and disease progression in vulvar cancer (3).


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