Outer Synchronization of Drive-Response Complex-Valued Complex Networks via Intermittent Pinning Control

This paper is concerned with the outer exponential synchronization of the drive-response complex dynamical networks subject to time-varying delays. The dynamics of nodes is complex valued, the interactions among of the nodes are directed, and the two coupling matrices in the drive system and the response system are also different. The intermittent pinning control is proposed to achieve outer exponential synchronization in the aperiodical way. Some novel sufficient conditions are derived to guarantee outer exponential synchronization of the considered complex-valued complex networks by using the Lyapunov functional method. Finally, two numerical examples are presented to illustrate the effectiveness of the proposed control protocols.

HIGH-RESPONSE COMPLEX FOR ANALYZING DROPLETS AND VAPORS IN AEROSOL CLOUDS FORMED IN THE ATMOSPHEREDUE TO LIQUID DISPERSION

The high-response diagnostic complex for remote control and analyses of droplets and vapors of mazut, oil, gasoline, kerosene, diesel fuel and liquefied natural gas in the clouds and turbulent aerosolflows in the atmosphere with volume up to 107 m3 is described.

A stress-induced response complex (SIRC) shuttles miRNAs, siRNAs, and oligonucleotides to the nucleus

Although some information is available for specific subsets of miRNAs and several factors have been shown to bind oligonucleotides (ONs), no general transport mechanism for these molecules has been identified to date. In this work, we demonstrate that the nuclear transport of ONs, siRNAs, and miRNAs responds to cellular stress. Furthermore, we have identified a stress-induced response complex (SIRC), which includes Ago-1 and Ago-2 in addition to the transcription and splicing regulators YB1, CTCF, FUS, Smad1, Smad3, and Smad4. The SIRC transports endogenous miRNAs, siRNAs, and ONs to the nucleus. We show that cellular stress can significantly increase ON- or siRNA-directed splicing switch events and endogenous miRNA targeting of nuclear RNAs.