Hemostatic abnormalities and increased vascular endothelial cell markers in patients with red cell fragmentation syndrome induced by mitomycin C

1995 ◽  
Vol 50 (4) ◽  
pp. 237-243 ◽  
Author(s):  
Shozaburo Nagaya ◽  
Hideo Wada ◽  
Kouzi Oka ◽  
Motoaki Tanigawa ◽  
Shigehisa Tamaki ◽  
...  
Keyword(s):  
Endothelial Cell ◽  
Mitomycin C ◽  
Vascular Endothelial Cell ◽  
Vascular Endothelial ◽  
Red Cell ◽  
Cell Markers

Elevated plasma levels of vascular endothelial cell markers in patients with hypercholesterolemia

1993 ◽  
Vol 44 (2) ◽  
pp. 112-116 ◽  
Author(s):  
Hideo Wada ◽  
Yoshitaka Mori ◽  
Toshihiro Kaneko ◽  
Yoshihiro Wakita ◽  
Tutomu Nakase ◽  
...  
Keyword(s):  
Endothelial Cell ◽  
Plasma Levels ◽  
Vascular Endothelial Cell ◽  
Vascular Endothelial ◽  
Elevated Plasma ◽  
Cell Markers

Increased vascular endothelial cell markers in patients with disseminated intravascular coagulation

1993 ◽  
Vol 44 (2) ◽  
pp. 85-88 ◽  
Author(s):  
Hideo Wada ◽  
Kouzou Minamikawa ◽  
Yoshihiro Wakita ◽  
Tutomu Nakase ◽  
Toshihiro Kaneko ◽  
...  
Keyword(s):  
Endothelial Cell ◽  
Disseminated Intravascular Coagulation ◽  
Vascular Endothelial Cell ◽  
Vascular Endothelial ◽  
Cell Markers ◽  
Intravascular Coagulation

Increased levels of vascular endothelial cell markers in thrombotic thrombocytopenic purpura

1993 ◽  
Vol 44 (2) ◽  
pp. 101-105 ◽  
Author(s):  
Hideo Wada ◽  
Toshihiro Kaneko ◽  
Michiaki Ohiwa ◽  
Motoaki Tanigawa ◽  
Tatsuya Hayashi ◽  
...  
Keyword(s):  
Endothelial Cell ◽  
Thrombotic Thrombocytopenic Purpura ◽  
Vascular Endothelial Cell ◽  
Vascular Endothelial ◽  
Thrombocytopenic Purpura ◽  
Cell Markers

Effect of angiotensin(1-7) on the expression of E-selectin induced by angiotensinII and its mechanism in vascular endothelial cell

2010 ◽  
Vol 34 (8) ◽  
pp. S71-S71
Author(s):  
Xiaohui Shen ◽  
Zhi‑Bin Wen ◽  
Na Li ◽  
Qingmei Cheng ◽  
Xiaofan He ◽  
...  
Keyword(s):  
Endothelial Cell ◽  
Vascular Endothelial Cell ◽  
Vascular Endothelial

Alterations in Vascular Endothelial Cell-related Plasma Proteins In Thalassaemic Patients and their Correlation with Clinical Symptoms

1995 ◽  
Vol 74 (04) ◽  
pp. 1045-1049 ◽  
Author(s):  
P Butthep ◽  
A Bunyaratvej ◽  
Y Funahara ◽  
H Kitaguchi ◽  
S Fucharoen ◽  
...  
Keyword(s):  
Endothelial Cells ◽  
Endothelial Cell ◽  
Immunosorbent Assay ◽  
Plasma Proteins ◽  
Clinical Symptoms ◽  
Vascular Endothelial Cells ◽  
Vascular Endothelial Cell ◽  
Enzyme Linked Immunosorbent Assay ◽  
Vascular Endothelial ◽  
Leg Ulcers

SummaryAn increased level of plasma thrombomodulin (TM) in α- and β- thalassaemia was demonstrated using an enzyme-linked immunosorbent assay (ELISA). Nonsplenectomized patients with β-thalassaemia/ haemoglobin E (BE) had higher levels of TM than splenectomized cases (BE-S). Patients with leg ulcers (BE-LU) were found to have the highest increase in TM level. Appearance of larger platelets in all types of thalassaemic blood was observed indicating an increase in the number of younger platelets. These data indicate that injury of vascular endothelial cells is present in thalassaemic patients.

scholarly journals Uric acid promotes oxidative stress and enhances vascular endothelial cell apoptosis in rats with middle cerebral artery occlusion

2018 ◽  
Vol 38 (3) ◽  
Author(s):  
Chengfu Song ◽  
Xiangdong Zhao
Keyword(s):  
Oxidative Stress ◽  
Uric Acid ◽  
Endothelial Cell ◽  
Cell Apoptosis ◽  
Vascular Endothelial Cell ◽  
Artery Occlusion ◽  
Vascular Endothelial ◽  
Endothelial Cell Apoptosis ◽  
Related Factors ◽  
Cerebral Artery Occlusion

In patients with cerebral infarction (CI), elevated serum uric acid (UA) level may exacerbate the occurrence and development of carotid atherosclerosis (AS). Our study intended to explore the underlying mechanism. We enrolled 86 patients with CI, and divided them into four groups: Non-AS, AS-mild, AS-moderate, and AS-severe groups; the levels of UA and oxidative stress-related factors in serum were detected. The middle cerebral artery occlusion (MCAO) model was used to stimulate CI in rats, and different doses of UA were administrated. The levels of oxidative stress-related factors in serum were detected. Hematoxylin & eosin (H&E) staining was used to observe the morphological alterations, and the apoptotic cell death detection kit was used to detect apoptotic cells. Increased UA concentration and enhanced oxidative stress were found in AS patients. H&E staining results showed that UA treatment exacerbated morphological damage in rats with MCAO, promoted oxidative stress, and enhanced vascular endothelial cell apoptosis in rats with MCAO.

Sign in / Sign up

Export Citation Format

Share Document