scholarly journals Putative tumor suppressor miR-145 inhibits colon cancer cell growth by targeting oncogene friend leukemia virus integration 1 gene

Cancer ◽  
2010 ◽  
Vol 117 (1) ◽  
pp. 86-95 ◽  
Author(s):  
Jianjun Zhang ◽  
Haiyan Guo ◽  
He Zhang ◽  
Haibo Wang ◽  
Guanxiang Qian ◽  
...  
Keyword(s):  
Colon Cancer ◽  
Cell Growth ◽  
Tumor Suppressor ◽  
Leukemia Virus ◽  
Colon Cancer Cell ◽  
Cancer Cell Growth ◽  
Friend Leukemia ◽  
Putative Tumor Suppressor ◽  
Virus Integration ◽  
Friend Leukemia Virus

A Short SOX9 Peptide Mimics SOX9 Tumor Suppressor Activity and Is Sufficient to Inhibit Colon Cancer Cell Growth

2019 ◽  
Vol 18 (8) ◽  
pp. 1386-1395 ◽  
Author(s):  
Philippe Blache ◽  
Lucile Canterel-Thouennon ◽  
Muriel Busson ◽  
Pascal Verdié ◽  
Gilles Subra ◽  
...  
Keyword(s):  
Colon Cancer ◽  
Cell Growth ◽  
Tumor Suppressor ◽  
Cancer Cell ◽  
Colon Cancer Cell ◽  
Peptide Mimics ◽  
Cancer Cell Growth ◽  
Suppressor Activity ◽  
Tumor Suppressor Activity

Stachyose-induced apoptosis of Caco-2 cells via the caspase-dependent mitochondrial pathway

2015 ◽  
Vol 6 (3) ◽  
pp. 765-771 ◽  
Author(s):  
Guidong Huang ◽  
Jian Mao ◽  
Zhongwei Ji ◽  
Aisikaer Ailati
Keyword(s):  
Colon Cancer ◽  
Cell Growth ◽  
Cancer Cell ◽  
Mitochondrial Pathway ◽  
Colon Cancer Cell ◽  
Cancer Cell Growth ◽  
Induced Apoptosis

Some studies have shown that stachyose, as prebiotics, can prevent indirectly colon cancer cell growth by promoting the proliferation of probiotics or producing beneficial materials in the intestine.

What can we learn from Fli1-deficient mice, new animal models of systemic sclerosis?

2018 ◽  
Vol 3 (1) ◽  
pp. 6-13 ◽  
Author(s):  
Yoshihide Asano
Keyword(s):  
Systemic Sclerosis ◽  
Animal Models ◽  
Leukemia Virus ◽  
Environmental Influences ◽  
Epidemiological Studies ◽  
Potential Candidate ◽  
Predisposing Factor ◽  
Friend Leukemia ◽  
Virus Integration ◽  
Friend Leukemia Virus

Systemic sclerosis is a complex multifactorial disease characterized by autoimmunity, vasculopathy, and selective organ fibrosis. A series of genetic and epidemiological studies have demonstrated that environmental influences play a central role in the onset of systemic sclerosis, while genetic factors determine the susceptibility to and the severity of this disease. Therefore, the identification of predisposing factors related to environmental influences would provide us with an informative clue to better understand the pathological process of this disease. Based on this concept, the deficiency of transcription factor Friend leukemia virus integration 1, which is epigenetically suppressed in systemic sclerosis, seems to be a potential candidate acting as the predisposing factor of this disease. Indeed, Fli1-mutated mice serve as a set of useful disease models to disclose the complex pathology of systemic sclerosis. This article overviews the recent advancement in systemic sclerosis animal models associated with Friend leukemia virus integration 1 deficiency.

scholarly journals Somatostatin inhibits colon cancer cell growth through cyclooxygenase-2 downregulation

2008 ◽  
Vol 155 (2) ◽  
pp. 198-209 ◽  
Author(s):  
R Colucci ◽  
C Blandizzi ◽  
N Ghisu ◽  
T Florio ◽  
M Del Tacca
Keyword(s):  
Colon Cancer ◽  
Cell Growth ◽  
Cancer Cell ◽  
Colon Cancer Cell ◽  
Cyclooxygenase 2 ◽  
Cancer Cell Growth

scholarly journals CSBF/C10orf99, a novel potential cytokine, inhibits colon cancer cell growth through inducing G1 arrest

2014 ◽  
Vol 4 (1) ◽  
Author(s):  
Wen Pan ◽  
Yingying Cheng ◽  
Heyu Zhang ◽  
Baocai Liu ◽  
Xiaoning Mo ◽  
...  
Keyword(s):  
Colon Cancer ◽  
Cell Growth ◽  
Cancer Cell ◽  
Colon Cancer Cell ◽  
G1 Arrest ◽  
Cancer Cell Growth
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