Effect on Wall Shear Rates of Taylor Vortex Instabilities Induced by Progressive Variation of the Inner Cylinder

Author(s):  
Emna Berrich ◽  
Fethi Aloui ◽  
Jack Legrand
Keyword(s):  
Author(s):  
Emna Berrich ◽  
Fethi Aloui ◽  
Jack Legrand

In the simplest and original case of study of the Taylor–Couette TC problems, the fluid is contained between a fixed outer cylinder and a concentric inner cylinder which rotates at constant angular velocity. Much of the works done has been concerned on steady rotating cylinder(s) i.e. rotating cylinders with constant velocity and the various transitions that take place as the cylinder(s) velocity (ies) is (are) steadily increased. On this work, we concentrated our attention in the case in which the inner cylinder velocity is not constant, but oscillates harmonically (in time) clockwise and counter-clockwise while the outer cylinder is maintained fixed. Our aim is to attempt to answer the question if the modulation makes the flow more or less stable with respect to the vortices apparition than in the steady case. If the modulation amplitude is large enough to destabilise the circular Couette flow, two classes of axisymmetric Taylor vortex flow are possible: reversing Taylor Vortex Flow (RTVF) and Non-Reversing Taylor Vortex Flow (NRTVF) (Youd et al., 2003; Lopez and Marques, 2002). Our work presents an experimental investigation of the effect of oscillatory Couette-Taylor flow, i.e. both the oscillation frequency and amplitude on the apparition of RTVF and NRTVF by analysing the instantaneous and local mass transfer and wall shear rates evolutions, i.e. the impact of vortices at wall. The vortices may manifest themselves by the presence of time-oscillations of mass transfer and wall shear rates, this generally corresponds to an instability apparition even for steady rotating cylinder. On laminar CT flow, the time-evolution of wall shear rate is linear. It may be presented as a linear function of the angular velocity, i.e. the evolution is steady even if the angular velocity is not steady. At a “critical” frequency and amplitude, the laminar CT flow is disturbed and Taylor vortices appear. Comparing to a steady velocity case, oscillatory flow accelerate the instability apparition, i.e. the critical Taylor number corresponds to the transition is smaller than that of the steady case. For high oscillation amplitudes of the inner cylinder rotation, the mass transfer time-evolution has a sinusoidal evolution with non equal oscillation amplitudes. If the oscillation amplitude is large enough, it can destabilize the laminar Couette flow, Taylor vortices appears. The vortices direction can be deduced from the sign of the instantaneous wall shear rate time evolution.


Soft Matter ◽  
2018 ◽  
Vol 14 (36) ◽  
pp. 7401-7419 ◽  
Author(s):  
Huilin Ye ◽  
Zhiqiang Shen ◽  
Ying Li

The shape effect of micro-particles is examined by comparing the margination behaviors of sphere-like, oblate-like and prolate-like micro-particles under different wall shear rates in blood flow.


1987 ◽  
Vol 109 (1) ◽  
pp. 60-64 ◽  
Author(s):  
L. Talbot ◽  
J. J. Steinert

The frequency response of surface-mounted electrochemical mass transfer probes used to deduce wall shear rates has been investigated experimentally for the case of fully developed laminar pulsatile flow in a straight tube. Generally good agreement is found with the asymptotic results obtained by Lighthill’s methods. The significance of the results with regard to the investigation of models of pulsatile flows of physiological interest is discussed. It is concluded that the frequency-dependent phase and amplitude corrections required to obtain accurate wall shear measurements are of such magnitudes as to render impractical the use of electrochemical probes to determine wall shear rates in these flows.


2003 ◽  
Vol 26 (4) ◽  
pp. 331-338 ◽  
Author(s):  
S. Körfer ◽  
S. Klaus ◽  
K. Mottaghy

Background Artificial organs, implants and extracorporeal circulation affect the physiological flow characteristics of blood as a liquid organ. These artificial systems consist of a wide variety of biomaterials with different geometries and, therefore, with their own flow properties. Secondary flow also occurs in extra – as well as in intracorporeal circulation. Methods In order to investigate the influence of vortical flow conditions a modified Taylor-Couette system was introduced. It consisted of two coaxial cylinders whose surfaces were the target of investigation. The annular gap was filled with donor blood shear and secondary flows were produced by rotating the inner cylinder. Platelet activation and protein adsorption were investigated as markers for thrombogenicity. Results At shear rates high enough to establish stable Taylor vortices (G ≥ 550 s −1) significant differences between vortical Taylor flow and steady laminar flow were detected. At shear rates of G ≥ 550 s −1 laminar flow caused a significantly higher platelet drop and PF4 release when compared to Taylor vortex flow. Also protein adsorption per square unit was significantly higher for laminar flow. Conclusions Based on the present data we conclude that vortical flow patterns lead to an accumulation of platelets and plasma proteins in the vortex center and therefore to a decreased probability of contact between platelets and material surfaces. It can be concluded that a preactivation of the platelets circulating in extracorporeal circuits can be manifested downstream in other geometrical configurations and flow conditions.


Blood ◽  
1985 ◽  
Vol 65 (1) ◽  
pp. 85-90 ◽  
Author(s):  
HV Stel ◽  
KS Sakariassen ◽  
PG de Groot ◽  
JA van Mourik ◽  
JJ Sixma

Abstract A monoclonal antibody directed against the von Willebrand factor moiety (vWF) of factor VIII-von Willebrand factor (FVIII-vWF), which blocks ristocetin-induced platelet aggregation as well as the binding of FVIII- vWF to platelets in the presence of ristocetin, inhibited platelet adherence to human artery subendothelium when present in normal flowing blood. This monoclonal antibody, CLB-RAg 35, inhibited platelet adherence as a function of the shear rate. At wall shear rates below 500 s-1, platelet adherence was not affected, but at higher shear rates platelet adherence was gradually inhibited, reaching an average of 11% of the normal value at 2,500 s-1. Indirect immunofluorescence established the reactivity of CLB-RAg 35 with vWF present in artery subendothelium. Pretreatment of normal vessel walls with this antibody inhibited adherence of platelets in blood from a patient with severe homozygous von Willebrand's disease and in blood from normal individuals. The inhibition was shear-rate dependent and significant at high shear rates (2,500 s-1). By adding increasing amounts of purified FVIII-vWF to normal blood, the inhibition was gradually overcome. These data indicate that vWF present in the vessel wall contributes appreciably to platelet adherence. At high wall shear rates, platelet adherence is mediated virtually completely by both plasma FVIII-vWF and vWF in the vessel wall. At low wall shear rates (below 500 s-1), platelet adherence occurs independent of FVIII-vWF in plasma and vWF in the vessel wall.


1994 ◽  
Vol 116 (4) ◽  
pp. 446-451 ◽  
Author(s):  
John M. Siegel ◽  
Christos P. Markou ◽  
David N. Ku ◽  
S. R. Hanson

Atherosclerosis of the human arterial system produces major clinical symptoms when the plaque advances to create a high-grade stenosis. The hemodynamic shear rates produced in high-grade stenoses are important in the understanding of atheromatous plaque rupture and thrombosis. This study was designed to quantify the physiologic stress levels experienced by endothelial cells and platelets in the region of vascular stenoses. The steady hemodynamic flow field was solved for stenoses with percent area reductions of 50, 75, and 90 percent over a range of physiologic Reynolds numbers (100–400). The maximum wall shear rate in the throat region can be shown to vary by the square root of the Reynolds number. The shear rate results can be generalized to apply to a range of stenosis lengths and flow rates. Using dimensions typical for a human carotid or coronary artery, wall shear rates were found to vary from a maximum of 20,000 s−1 upstream of the throat to a minimum of −630 s−1 in the recirculation zone for a 90 percent stenosis. An example is given which illustrates how these values can be used to understand the relationship between hemodynamic shear and platelet deposition.


Author(s):  
Ravi Arora ◽  
Eric Daymo ◽  
Anna Lee Tonkovich ◽  
Laura Silva ◽  
Rick Stevenson ◽  
...  

Emulsion formation within microchannels enables smaller mean droplet sizes for new commercial applications such as personal care, medical, and food products among others. When operated at a high flow rate per channel, the resulting emulsion mixture creates a high wall shear stress along the walls of the narrow microchannel. This high fluid-wall shear stress of continuous phase material past a dispersed phase, introduced through a permeable wall, enables the formation of small emulsion droplets — one drop at a time. A challenge to the scale-up of this technology has been to understand the behavior of non-Newtonian fluids under high wall shear stress. A further complication has been the change in fluid properties with composition along the length of the microchannel as the emulsion is formed. Many of the predictive models for non-Newtonian emulsion fluids were derived at low shear rates and have shown excellent agreement between predictions and experiments. The power law relationship for non-Newtonian emulsions obtained at low shear rates breaks down under the high shear environment created by high throughputs in small microchannels. The small dimensions create higher velocity gradients at the wall, resulting in larger apparent viscosity. Extrapolation of the power law obtained in low shear environment may lead to under-predictions of pressure drop in microchannels. This work describes the results of a shear-thinning fluid that generates larger pressure drop in a high-wall shear stress microchannel environment than predicted from traditional correlations.


1994 ◽  
Vol 116 (3) ◽  
pp. 361-368 ◽  
Author(s):  
Ray S. Fatemi ◽  
Stanley E. Rittgers

Atherosclerosis, thrombosis, and intimal hyperplasia are major forms of cardiovascular diseases in the United States. Previous studies indicate a significant correlation between hemodynamics, in particular, wall shear rate, and pathology of the arterial walls. While results of these studies implicate morphologic and functional changes related to wall shear rate magnitude, a standard technique for wall shear rate measurement has not been established. In this study, theoretical and in-vitro experimental fully developed steady and physiologic pulsatile flow waveforms have been used to obtain velocity profiles in the near-wall region. The estimated wall shear rates from these results are compared to the theoretical value to assess the accuracy of the approximating technique. Experimentally obtained results from LDA suggest that in order to minimize the error in velocity data, and subsequently, the wall shear rate, the first measured velocity has to be 500 μm away from the wall. While a linear approximation did not produce errors larger than 16.4 percent at peak systole, these errors substantially increased as the velocity magnitudes decreased during late systole and diastole. Overall, a third degree polynomial curve fit using four points produced the most accurate estimation of wall shear rate through out the cardiac cycle. Results of higher degree curve-fitting functions can be unpredictable due to potential oscillations of the function near the wall. Hence, based on the results of this study, use of a linear approximation is not recommended; a third degree curve-fitting polynomial, using four points provided the most accurate approximation for these flow waveforms.


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