scholarly journals An (inverse) Pieri formula for Macdonald polynomials of type C

2010 ◽  
Vol 15 (1) ◽  
pp. 154-183 ◽  
Author(s):  
Michel Lassalle
2006 ◽  
Vol 202 (2) ◽  
pp. 289-325 ◽  
Author(s):  
Michel Lassalle ◽  
Michael Schlosser

2009 ◽  
Vol DMTCS Proceedings vol. AK,... (Proceedings) ◽  
Author(s):  
Cristian Lenart

International audience A breakthrough in the theory of (type $A$) Macdonald polynomials is due to Haglund, Haiman and Loehr, who exhibited a combinatorial formula for these polynomials in terms of fillings of Young diagrams. Recently, Ram and Yip gave a formula for the Macdonald polynomials of arbitrary type in terms of the corresponding affine Weyl group. In this paper, we show that a Haglund-Haiman-Loehr type formula follows naturally from the more general Ram-Yip formula, via compression. Then we extend this approach to the Hall-Littlewood polynomials of type $C$, which are specializations of the corresponding Macdonald polynomials at $q=0$. We note that no analog of the Haglund-Haiman-Loehr formula exists beyond type $A$, so our work is a first step towards finding such a formula.


Author(s):  
NIELS DISVELD ◽  
TOM H. KOORNWINDER ◽  
JASPER V. STOKMAN

AbstractNonsymmetric interpolation Laurent polynomials in n variables are introduced, with the interpolation points depending on q and on a n-tuple of parameters τ = (τ1, …, τn). When τi = stn − 1, Okounkov’s 3-parameter BCn-type interpolation Macdonald polynomials are recovered from the nonsymmetric interpolation Laurent polynomials through Hecke algebra symmetrisation with respect to a type Cn Hecke algebra action. In the Appendix we give some conjectures about extra vanishing, based on Mathematica computations in rank two.


Author(s):  
D.C. Hixson ◽  
J.C. Chan ◽  
J.M. Bowen ◽  
E.F. Walborg

Several years ago Karasaki (1) reported the production of type C virus particles by Novikoff ascites hepatocarcinoma cells. More recently, Weinstein (2) has reported the presence of type C virus particles in cell cultures derived from transplantable and primary hepatocellular carcinomas. To date, the biological function of these virus and their significance in chemically induced hepatocarcinogenesis are unknown. The present studies were initiated to determine a possible role for type C virus particles in chemically induced hepatocarcinogenesis. This communication describes results of studies on the biological and surface properties of type C virus associated with Novikoff hepatocarcinoma cells.Ecotropic and xenotropic murine leukemia virus (MuLV) activity in ascitic fluid of Novikoff tumor-bearing rats was assayed in murine sarcoma virus transformed S+L- mouse cells and S+L- mink cells, respectively. The presence of sarcoma virus activity was assayed in non-virus-producing normal rat kidney (NRK) cells. Ferritin conjugates of concanavalin A (Fer-Con wheat germ agglutinin (Fer-WGA), and Ricinus communis agglutinins I and II (Fer-RCAI and Fer-RCAII) were used to probe the structure and topography of saccharide determinants present on the viral envelope.


Author(s):  
L. Z. de Tkaczevski ◽  
E. de Harven ◽  
C. Friend

Despite extensive studies, the correlation between the morphology and pathogenicity of murine leukemia viruses (MLV) has not yet been clarified. The virus particles found in the plasma of leukemic mice belong to 2 distinct groups, 1 or 2% of them being enveloped A particles and the vast majority being of type C. It is generally believed that these 2 types of particles represent different phases in the development of the same virus. Particles of type A have been thought to be an earlier form of type C particles. One of the tissue culture lines established from Friend leukemia solid tumors has provided the material for the present study. The supernatant fluid of the line designated C-1A contains an almost pure population of A particles as illustrated in Figure 1. The ratio is, therefore, the reverse of what is unvariably observed in the plasma of leukemic mice where C particles predominate.


Author(s):  
Elizabeth S. Priori ◽  
T. Shigematsu ◽  
B. Myers ◽  
L. Dmochowski

Spontaneous release of type C virus particles in long-term cultures of mouse embryo cells as well as induction of similar particles in mouse embryo cell cultures with IUDR or BUDR have been reported. The presence of type C virus particles in cultures of normal rat embryos has not been reported.NB-1, a culture derived from embryos of a New Zealand Black (NB) rat (rats obtained from Mr. Samuel M. Poiley, N.C.I., Bethesda, Md.) and grown in McCoy's 5A medium supplemented with 20% fetal calf serum was passaged weekly. Extracellular virus particles similar to murine leukemia particles appeared in the 22nd subculture. General appearance of cells in passage 23 is shown in Fig. 1. Two budding figures and one immature type C virus particle may be seen in Fig. 2. The virus particles and budding were present in all further passages examined (currently passage 39). Various stages of budding are shown in Figs. 3a,b,c,d. Appearance of a mature virus particle is shown in Fig. 4.


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