Repeated crack cocaine administration alters panic-related responses and delta FosB immunoreactivity in panic-modulating brain regions

2021 ◽  
Vol 239 (4) ◽  
pp. 1179-1191
Author(s):  
Barbara dos Anjos Rosário ◽  
Maria de Fátima Santana de Nazaré ◽  
Jéssica Alves Lemes ◽  
José Simões de Andrade ◽  
Regina Barbosa da Silva ◽  
...  
Keyword(s):  
Crack Cocaine ◽  
Brain Regions ◽  
Cocaine Administration ◽  
Delta Fosb

scholarly journals Delta FosB and AP-1-mediated transcription modulate cannabinoid CB1 receptor signaling and desensitization in striatal and limbic brain regions

2014 ◽  
Vol 91 (3) ◽  
pp. 380-389 ◽  
Author(s):  
Matthew F. Lazenka ◽  
Bethany G. David ◽  
Aron H. Lichtman ◽  
Eric J. Nestler ◽  
Dana E. Selley ◽  
...  
Keyword(s):  
Brain Regions ◽  
Cb1 Receptor ◽  
Receptor Signaling ◽  
Cannabinoid Cb1 Receptor ◽  
Delta Fosb

Brain blood flow abnormalities associated with oral cocaine use

1997 ◽  
Vol 14 (2) ◽  
pp. 72-73 ◽  
Author(s):  
Bankole Johnson ◽  
Lamk Lamki ◽  
Bruce Barron ◽  
Ralph Spiga ◽  
Richard Chen ◽  
...  
Keyword(s):  
Blood Flow ◽  
Crack Cocaine ◽  
Single Photon ◽  
Photon Emission ◽  
Brain Regions ◽  
Single Photon Emission ◽  
Cocaine User ◽  
Cocaine Use ◽  
The Brain ◽  
Crack Users

AbstractSingle photon emission tomography (SPECT) is the prototypical tool for measuring cerebral blood flow (CBF) in discrete areas of the brain. Compared with when a male ‘crack’ cocaine user received placebo, oral cocaine (1mg/kg) ingestion was associated with non-uniformity of overall CBF with hypoperfusion of discrete brain regions, particularly of the frontal, temporo-parietal, basal ganglia, and thalamic areas. While these results should be viewed as preliminary, they do suggest that oral cocaine use may be associated with CBF abnormalities in ‘crack’ users.

Repeated cocaine administration does not affect 5-HT receptor subtypes (5-HT1A, 5-HT2) in several rat brain regions

1993 ◽  
Vol 238 (2-3) ◽  
pp. 425-429 ◽  
Author(s):  
Javaid I. Javaid ◽  
Sanjeev K. Sahni ◽  
Subhash C. Pandey ◽  
John M. Davis
Keyword(s):  
Rat Brain ◽  
Brain Regions ◽  
Receptor Subtypes ◽  
Cocaine Administration ◽  
Rat Brain Regions

Alterations of corticotropin-releasing factor-like immunoreactivity in different brain regions after acute cocaine administration in rats

1993 ◽  
Vol 616 (1-2) ◽  
pp. 315-319 ◽  
Author(s):  
Zoltán Sarnyai ◽  
Éva Bíró ◽  
János Gardi ◽  
Miklós Vecsernyés ◽  
János Julesz ◽  
...  
Keyword(s):  
Brain Regions ◽  
Corticotropin Releasing Factor ◽  
Cocaine Administration

Environmental enrichment decreases avoidance responses in the elevated T-maze and delta FosB immunoreactivity in anxiety-related brain regions

2018 ◽  
Vol 344 ◽  
pp. 65-72 ◽  
Author(s):  
Danielle A. Lopes ◽  
Thaissa M.O. Souza ◽  
José S. de Andrade ◽  
Mariana F.S. Silva ◽  
Hanna K.M. Antunes ◽  
...  
Keyword(s):  
Environmental Enrichment ◽  
Brain Regions ◽  
Delta Fosb ◽  
Avoidance Responses

scholarly journals Dopamine D1-D2 receptor heteromer expression in key brain regions of rat and higher species: Upregulation in rat striatum after cocaine administration

2020 ◽  
Vol 143 ◽  
pp. 105017
Author(s):  
Ahmed Hasbi ◽  
Meenalochani Sivasubramanian ◽  
Marija Milenkovic ◽  
Kristina Komarek ◽  
Bertha K. Madras ◽  
...  
Keyword(s):  
D2 Receptor ◽  
Brain Regions ◽  
Rat Striatum ◽  
Cocaine Administration

Synaptic organization of the gustatory NST

1994 ◽  
Vol 52 ◽  
pp. 150-151
Author(s):  
M. C. Whitehead
Keyword(s):  
Central Nervous System ◽  
Dendritic Spines ◽  
Fundamental Problem ◽  
Cell Types ◽  
Brain Regions ◽  
Excitatory Synapses ◽  
Solitary Tract ◽  
Synaptic Organization ◽  
Synaptic Junctions ◽  
Afferent Terminals

A fundamental problem in taste research is to determine how gustatory signals are processed and disseminated in the mammalian central nervous system. An important first step toward understanding information processing is the identification of cell types in the nucleus of the solitary tract (NST) and their synaptic relationships with oral primary afferent terminals. Facial and glossopharyngeal (LIX) terminals in the hamster were labelled with HRP, examined with EM, and characterized as containing moderate concentrations of medium-sized round vesicles, and engaging in asymmetrical synaptic junctions. Ultrastructurally the endings resemble excitatory synapses in other brain regions.Labelled facial afferent endings in the RC subdivision synapse almost exclusively with distal dendrites and dendritic spines of NST cells. Most synaptic relationships between the facial synapses and the dendrites are simple. However, 40% of facial endings engage in complex synaptic relationships within glomeruli containing unlabelled axon endings particularly ones termed "SP" endings. SP endings are densely packed with small, pleomorphic vesicles and synapse with both the facial endings and their postsynaptic dendrites by means of nearly symmetrical junctions.

Musical Anhedonia

2020 ◽  
Vol 31 (2) ◽  
pp. 62-68
Author(s):  
Sara E. Holm ◽  
Alexander Schmidt ◽  
Christoph J. Ploner
Keyword(s):  
Quality Of Life ◽  
Nucleus Accumbens ◽  
Brain Damage ◽  
Parietal Cortex ◽  
Neurological Disorders ◽  
Brain Regions ◽  
Precise Information ◽  
Exact Localization ◽  
High Prevalence

Abstract. Some people, although they are perfectly healthy and happy, cannot enjoy music. These individuals have musical anhedonia, a condition which can be congenital or may occur after focal brain damage. To date, only a few cases of acquired musical anhedonia have been reported in the literature with lesions of the temporo-parietal cortex being particularly important. Even less literature exists on congenital musical anhedonia, in which impaired connectivity of temporal brain regions with the Nucleus accumbens is implicated. Nonetheless, there is no precise information on the prevalence, causes or exact localization of both congenital and acquired musical anhedonia. However, the frequent involvement of temporo-parietal brain regions in neurological disorders such as stroke suggest the possibility of a high prevalence of this disorder, which leads to a considerable reduction in the quality of life.

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