Role of saturated and unsaturated fatty acids on dicarbonyl–albumin derived advanced glycation end products in vitro

Amino Acids ◽  
2021 ◽  
Author(s):  
Brock Peake ◽  
Maulik Ghetia ◽  
Cobus Gerber ◽  
Maurizio Costabile ◽  
Permal Deo
Keyword(s):  
Fatty Acids ◽  
Advanced Glycation End Products ◽  
Unsaturated Fatty Acids ◽  
Advanced Glycation ◽  
Glycation End Products ◽  
End Products

scholarly journals Advanced glycation end products and strontium ranelate promote osteogenic differentiation of vascular smooth muscle cells in vitro: Preventive role of vitamin D

2017 ◽  
Vol 450 ◽  
pp. 94-104 ◽  
Author(s):  
María Silvina Molinuevo ◽  
Juan Manuel Fernández ◽  
Ana María Cortizo ◽  
Antonio Desmond McCarthy ◽  
León Schurman ◽  
...  
Keyword(s):  
Vitamin D ◽  
Smooth Muscle ◽  
Vascular Smooth Muscle Cells ◽  
Advanced Glycation End Products ◽  
Advanced Glycation ◽  
Glycation End Products ◽  
End Products ◽  
Preventive Role

Role of maltol in advanced glycation end products and free radicals: in-vitro and in-vivo studies

2008 ◽  
Vol 60 (4) ◽  
pp. 445-452 ◽  
Author(s):  
Ki Sung Kang ◽  
Noriko Yamabe ◽  
Hyun Young Kim ◽  
Takako Yokozawa
Keyword(s):  
Free Radicals ◽  
Advanced Glycation End Products ◽  
In Vivo Studies ◽  
Advanced Glycation ◽  
Glycation End Products ◽  
End Products

scholarly journals In Vitro Methodologies to Study the Role of Advanced Glycation End Products (AGEs) in Neurodegeneration

Nutrients ◽  
2022 ◽  
Vol 14 (2) ◽  
pp. 363
Author(s):  
Marialena Chrysanthou ◽  
Ignacio Miro Estruch ◽  
Ivonne M. C. M. Rietjens ◽  
Harry J. Wichers ◽  
Tamara Hoppenbrouwers
Keyword(s):  
Neurodegenerative Diseases ◽  
Advanced Glycation End Products ◽  
In Vitro Models ◽  
Research Field ◽  
Advanced Glycation ◽  
Glycation End Products ◽  
End Products ◽  
Specific Receptors

Advanced glycation end products (AGEs) can be present in food or be endogenously produced in biological systems. Their formation has been associated with chronic neurodegenerative diseases such as Alzheimer’s disease, Parkinson’s disease, multiple sclerosis, and amyotrophic lateral sclerosis. The implication of AGEs in neurodegeneration is related to their ability to bind to AGE-specific receptors and the ability of their precursors to induce the so-called “dicarbonyl stress”, resulting in cross-linking and protein damage. However, the mode of action underlying their role in neurodegeneration remains unclear. While some research has been carried out in observational clinical studies, further in vitro studies may help elucidate these underlying modes of action. This review presents and discusses in vitro methodologies used in research on the potential role of AGEs in neuroinflammation and neurodegeneration. The overview reveals the main concepts linking AGEs to neurodegeneration, the current findings, and the available and advisable in vitro models to study their role. Moreover, the major questions regarding the role of AGEs in neurodegenerative diseases and the challenges and discrepancies in the research field are discussed.

Sulforaphane inhibits formation of advanced glycation end products in vitro

2014 ◽  
Vol 1 (e1) ◽  
pp. 001-001 ◽  
Author(s):  
Kei Fukami ◽  
Takanori Matsui ◽  
Sho-ichi Yamagishi
Keyword(s):  
Advanced Glycation End Products ◽  
Advanced Glycation ◽  
Glycation End Products ◽  
End Products
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