Temporal relationship of subjective perception and the neuronal events induced by peripheral stimulation

1985 ◽  
Vol 61 (3) ◽  
pp. S238-S239
Author(s):  
W. Massing
Clinics ◽  
2013 ◽  
Vol 68 (7) ◽  
pp. 1034-1038 ◽  
Author(s):  
FJ Guzmán-de la Garza ◽  
JM Ibarra-Hernández ◽  
P Cordero-Pérez ◽  
P Villegas-Quintero ◽  
CI Villarreal-Ovalle ◽  
...  

2013 ◽  
Vol 19 (4) ◽  
pp. 233-239 ◽  
Author(s):  
Chad Brodt ◽  
Jill D. Siegfried ◽  
Mark Hofmeyer ◽  
Jose Martel ◽  
Evadnie Rampersaud ◽  
...  

Author(s):  
David Locke

The musical rhythm of Agbadza is analyzed as dynamic and multideterminant, that is, as emerging from the interaction of many different musical factors including dance, metric structure, and the accentuation and grouping of the parts in the drum ensemble (bell, handclap, rattle, and support drum). Response drum and lead drum parts are analyzed for accentuation and rhythmic motion; the singing of Agbadza is studied in terms of melodic motion, design of phrases, and call-and-response form. The temporal relationship of songs to instrumental music is examined. By integrating the performance modalities of dance, song, and drumming within a holistic, ethnographically informed analysis of musical rhythm, the chapter models a method for documenting and understanding traditions of African performance art.


1998 ◽  
Vol 275 (2) ◽  
pp. R502-R508 ◽  
Author(s):  
Xianzhong Meng ◽  
Lihua Ao ◽  
Daniel R. Meldrum ◽  
Brian S. Cain ◽  
Brian D. Shames ◽  
...  

Exogenous tumor necrosis factor-α (TNF-α) induces delayed myocardial depression in vivo but promotes rapid myocardial depression in vitro. The temporal relationship between endogenous TNF-α and endotoxemic myocardial depression is unclear, and the role of TNF-α in this myocardial disorder remains controversial. Using a rat model of endotoxemia not complicated by shock, we sought to determine 1) the temporal relationship of changes in circulating and myocardial TNF-α with myocardial depression, 2) the influences of protein synthesis inhibition or immunosuppression on TNF-α production and myocardial depression, and 3) the influence of neutralization of TNF-α on myocardial depression. Rats were treated with lipopolysaccharide (LPS, 0.5 mg/kg ip). Circulating and myocardial TNF-α increased at 1 and 2 h, whereas myocardial contractility was depressed at 4 and 6 h. Pretreatment with cycloheximide or dexamethasone abolished the increase in circulating and myocardial TNF-α and preserved myocardial contractile function. Similarly, treatment with TNF binding protein immediately after LPS prevented myocardial depression. We conclude that endogenous TNF-α mediates delayed myocardial depression in endotoxemic rats and that inhibition of TNF-α production or neutralization of TNF-α preserves myocardial contractile function in endotoxemia.


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