scholarly journals Which echocardiographic method used for calculation of left ventricular mass best predicts depressed flow reserve in hypertensive heart disease?

1990 ◽  
Vol 15 (2) ◽  
pp. A143
Author(s):  
Jan L. Houghton ◽  
L.Michael Prisant ◽  
Albert A. Carr ◽  
Thomas W. von Dohlen ◽  
Martin J. Frank
Keyword(s):  
Heart Disease ◽  
Left Ventricular Mass ◽  
Hypertensive Heart Disease ◽  
Left Ventricular ◽  
Flow Reserve ◽  
Ventricular Mass

Left ventricular hypertrophy in athletes: How to differentiate between hypertensive heart disease and athlete’s heart

2020 ◽  
pp. 204748732091185 ◽  
Author(s):  
Flavio D’Ascenzi ◽  
Caterina Fiorentini ◽  
Francesca Anselmi ◽  
Sergio Mondillo
Keyword(s):  
Heart Disease ◽  
Left Ventricular Mass ◽  
Wall Thickness ◽  
Hypertensive Heart Disease ◽  
Clinical Information ◽  
Left Ventricular ◽  
Athlete’S Heart ◽  
Ventricular Mass ◽  
Competitive Athletes ◽  
Athlete's Heart

Athlete’s heart is typically accompanied by a remodelling of the cardiac chambers induced by exercise. However, although competitive athletes are commonly considered healthy, they can be affected by cardiac disorders characterised by an increase in left ventricular mass and wall thickness, such as hypertension. Unfortunately, training-induced increase in left ventricular mass, wall thickness, and atrial and ventricular dilatation observed in competitive athletes may mimic the pathological remodelling of pathological hypertrophy. As a consequence, distinguishing between athlete’s heart and hypertension can sometimes be challenging. The present review aimed to focus on the differential diagnosis between hypertensive heart disease and athlete’s heart, providing clinical information useful to distinguish between physiological and pathological remodelling.

scholarly journals Prognostic Value of CTA-Derived Left Ventricular Mass in Neonates with Congenital Heart Disease

Diagnostics ◽  
2021 ◽  
Vol 11 (7) ◽  
pp. 1215
Author(s):  
Stephan Ellmann ◽  
Julie-Marie Nickel ◽  
Rafael Heiss ◽  
Nouhayla El Amrani ◽  
Wolfgang Wüst ◽  
...  
Keyword(s):  
Congenital Heart Disease ◽  
Heart Disease ◽  
Left Ventricular Mass ◽  
Congenital Heart ◽  
Left Ventricular ◽  
Surgical Approaches ◽  
Pulmonary Artery Stenosis ◽  
Operating Characteristics ◽  
Ventricular Mass ◽  
Therapeutic Decisions

For therapeutic decisions regarding uni- or biventricular surgical repair in congenital heart disease (CHD), left ventricular mass (LVM) is an important factor. The aim of this retrospective study was to determine the LVM of infants with CHD in thoracic computed tomography angiographies (CTAs) and to evaluate its usefulness as a prognostic parameter, with special attention paid to hypoplastic left heart (HLH) patients. Manual segmentation of the left ventricular endo- and epicardial volumes was performed in CTAs of 132 infants. LVMs were determined from these volumes and normalized to body surface area. LVMs of patients with different types of CHD were compared to each other using analyses of variances (ANOVA). An LVM cutoff for discrimination between uni- and biventricular repair was determined using receiver operating characteristics. Survival rates were calculated using Kaplan–Meier statistics. Patients with a clinical diagnosis of an HLH had significantly lower mean LVM (21.88 g/m2) compared to patients without applicable disease (50.22 g/m2; p < 0.0001) and compared to other CHDs, including persistent truncus arteriosus, left ventricular outflow tract obstruction, transposition of the great arteries, pulmonary artery stenosis or atresia, and double-outlet right ventricle (all, p < 0.05). The LVM cutoff for uni- vs. biventricular surgery was 33.9 g/m2 (sensitivity: 82.3%; specificity: 73.7%; PPV: 94.9%). In a subanalysis of HLH patients, a sensitivity of 50.0%, specificity of 100%, PPV of 100%, and NPV of 83.3% was determined. Patient survival was not significantly different between the surgical approaches or between patients with LVM above or below the cutoff. LVM can be measured in chest CTA of newborns with CHD and can be used as a prognostic factor.

Effects of exercise training on left ventricular mass in patients with ischemic heart disease

1981 ◽  
Vol 101 (6) ◽  
pp. 701-706 ◽  
Author(s):  
Roy V. Ditchey ◽  
John Watkins ◽  
M.Dan McKirnan ◽  
Victor Froelicher
Keyword(s):  
Heart Disease ◽  
Exercise Training ◽  
Ischemic Heart Disease ◽  
Left Ventricular Mass ◽  
Left Ventricular ◽  
Ischemic Heart ◽  
Ventricular Mass

scholarly journals Aldosterone synthase gene polymorphism in patients with hypertension combined ischemic heart disease with in various left ventricular mass

2014 ◽  
Vol 11 (1) ◽  
pp. 16-20
Author(s):  
D A Yahontov ◽  
D A Derisheva ◽  
L F Gulyaeva
Keyword(s):  
Coronary Artery Disease ◽  
Heart Disease ◽  
Left Ventricular Hypertrophy ◽  
Left Ventricular Mass ◽  
Ventricular Hypertrophy ◽  
Left Ventricular ◽  
Aldosterone Synthase ◽  
Ventricular Mass ◽  
Artery Disease ◽  
The Relationship

Study purpose - assessment of the relationship of levels of aldosterone and natriuretic peptide plasma aldosterone synthase gene polymorphism variants in patients with coronary heart disease (CHD) in combination with arterial hypertension (AH) at different left ventricular mass.Materials and methods. The study involved 63 male with hypertension and CHD; there were 32 men with signs of left ventricular hypertrophy (LVH) and 31 men with coronary artery disease with hypertension with a normal index of left ventricular mass. Evaluated the concentrations of aldosterone and plasma natriuretic peptide (NUP) in the relationship with characteristic of aldosterone synthase gene polymorphism.Results. It is established that the genotype CYP11В2 T/С of is associated with increased levels of aldosterone and signs of left ventricular hypertrophy. The level of LVC in the signs of LVH group was 2,1 fold higher than in patients without LVH. For patients with evidence of left ventricular hypertrophy is characterized by: the genotype of CYP11В2 T/C (62,5% of cases), the identification of the T allele (1,3 times more likely than patients without evidence of LVH) and more rarely than with normal index of left ventricular mass occurs variant CYP11В2 T/Т and more rarely prevalent allele C. Conclusion. In patients with coronary artery disease and hypertension, it is reasonable to implement in practice population genetic analysis to assess the probability the formation of left ventricular hypertrophy in the early stages of the disease.

Sign in / Sign up

Export Citation Format

Share Document