Efficient costimulatory signal for T cell proliferation induced by human lymphoma cells transduced with B7-1 and B7-2 molecules by recombinant adeno-associated virus (AAV) vectors

1995 ◽  
Vol 31 ◽  
pp. S26
Author(s):  
Elisabeth Mangold ◽  
Clemens M. Wendtner ◽  
Bertold Emmerich ◽  
Ernst-Ludwig Winnacker ◽  
Michael Hallek
Keyword(s):  
Cell Proliferation ◽  
T Cell ◽  
T Cell Proliferation ◽  
Lymphoma Cells ◽  
Adeno Associated Virus ◽  
Costimulatory Signal ◽  
Human Lymphoma

SATB1 overexpression promotes malignant T-cell proliferation in cutaneous CD30+ lymphoproliferative disease by repressing p21

Blood ◽  
2014 ◽  
Vol 123 (22) ◽  
pp. 3452-3461 ◽  
Author(s):  
Yang Wang ◽  
Xiaoguang Gu ◽  
Gaolei Zhang ◽  
Lin Wang ◽  
Tingting Wang ◽  
...  
Keyword(s):  
Cell Cycle ◽  
Cell Proliferation ◽  
T Cell ◽  
Transcriptional Repression ◽  
Lymphoproliferative Disease ◽  
T Cell Proliferation ◽  
Lymphoma Cells ◽  
Cell Cycle Inhibitor ◽  
Key Points

Key Points SATB1 is specifically overexpressed in the CD30+ lymphoma cells in cutaneous CD30+ lymphoproliferative disease. SATB1 promotes proliferation of CD30+ lymphoma cells by direct transcriptional repression of cell cycle inhibitor p21.

The co-receptor BTLA negatively regulates human Vγ9Vδ2 T-cell proliferation: a potential way of immune escape for lymphoma cells

Blood ◽  
2013 ◽  
Vol 122 (6) ◽  
pp. 922-931 ◽  
Author(s):  
Julie Gertner-Dardenne ◽  
Cyril Fauriat ◽  
Florence Orlanducci ◽  
Marie-Laure Thibult ◽  
Sonia Pastor ◽  
...  
Keyword(s):  
Cell Proliferation ◽  
T Cell ◽  
Immune Escape ◽  
T Cell Proliferation ◽  
Lymphoma Cells ◽  
Key Points

Key Points BTLA-HVEM interaction negatively regulates the proliferation of LTγδ. BTLA-HVEM interaction appears as a new possible mechanism of immune escape by lymphoma cells.

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