Angiosarcoma of the Thoracoabdominal Aorta Presenting with Systemic Hypertension, Anemia, and Visceral Ischemia

2008 ◽  
Vol 22 (3) ◽  
pp. 459-464 ◽  
Author(s):  
Tara Karamlou ◽  
Melissa K. Li ◽  
W. Kent Williamson ◽  
Lloyd Heller ◽  
John W. Wiest
Keyword(s):  
Systemic Hypertension ◽  
Thoracoabdominal Aorta ◽  
Visceral Ischemia

scholarly journals Endovascular Treatment of Acquired Atheromatous Postsubclavian Aortic Coarctation

2021 ◽  
Vol 48 (5) ◽  
Author(s):  
Bhushan S. Sonawane ◽  
Sreeja Pavithran ◽  
Kothandam Sivakumar
Keyword(s):  
Heart Failure ◽  
Coral Reef ◽  
Endovascular Treatment ◽  
Aortic Coarctation ◽  
Covered Stent ◽  
Endovascular Intervention ◽  
Systemic Hypertension ◽  
Extraanatomic Bypass ◽  
Thoracoabdominal Aorta ◽  
Obstructive Disease

Coral reef aorta is a rare calcifying obstructive disease that involves the thoracoabdominal aorta. Similar presentations in the postsubclavian aorta may result in acquired atheromatous aortic coarctation leading to systemic hypertension and heart failure. The associated calcification makes surgical anatomic or extraanatomic bypass and thromboendarterectomy challenging. Extensive circumferential calcification often precludes endovascular intervention. We present the case of a 25-year-old man with an acquired atheromatous coarctation of the postsubclavian aorta who underwent successful endovascular treatment with use of a balloon-expandable covered stent.

Coarctation of the abdominal aorta: Demonstration of a rare anomaly using contrast-enhanced MR-angiography

VASA ◽  
2009 ◽  
Vol 38 (1) ◽  
pp. 66-71 ◽  
Author(s):  
Schubert
Keyword(s):  
Abdominal Aorta ◽  
Mr Angiography ◽  
Parallel Imaging ◽  
Left Renal Vein ◽  
Systemic Hypertension ◽  
Contrast Enhanced ◽  
Younger Age ◽  
Maximum Intensity Projections ◽  
Collateral Vessels ◽  
First Time

We describe a case of aortic coarctation at the level of the infrarenal abdominal aorta which is encountered in less than six individuals in one million. In contrast to aortic narrowing above or including the renal arteries, this seems to be a relatively benign anomaly without systemic hypertension or impaired renal function. For the first time in this type of anomaly, contrast-enhanced MR angiography (ce-MRA) on a multi-receiver channel MR system, with an 8-channel phased array coil and parallel imaging was used. Ce-MRA displayed a tortuous, narrowed aortic segment that was found to be associated with mesenteric artery stenosis and compression of the orthotopic left renal vein, also known as the nutcracker phenomenon. All major aortic branches could be depicted using 3D surface-shaded displays and subvolume maximum intensity projections (MIPs). Collateral vessels of the abdominal wall were identified using whole-volume MIPs. Since the majority of aortic malformations are diagnosed at a younger age, and many suffer from renal insufficiency, we conclude that ce-MRA will eventually place conventional DSA as the modality of choice in malformations of the abdominal aorta.

Hybrid Endovascular Repair of Thoracoabdominal Aorta: Early Results

2014 ◽  
Vol 17 (3) ◽  
pp. 146
Author(s):  
Osman Tansel Darcin ◽  
Mehmet Kalender ◽  
Ayse Gul Kunt ◽  
Okay Guven Karaca ◽  
Ata Niyazi Ecevit ◽  
...  
Keyword(s):  
Endovascular Repair ◽  
Therapeutic Option ◽  
Complete Recovery ◽  
Aortic Aneurysms ◽  
Thoracoabdominal Aorta ◽  
Mortality And Morbidity ◽  
Early Results ◽  
The Mean ◽  
Procedural Success

<p><b>Background:</b> Thoracoabdominal aortic aneurysms (TAAA) present a significant clinical challenge, as they are complex and require invasive surgery. In an attempt to prevent considerably high mortality and morbidity in open repair, hybrid endovascular repair has been developed by many authors. In this study, we evaluated the early-term results obtained from this procedure.</p><p><b>Methods:</b> From November 2010 to February 2013, we performed thoracoabdominal hybrid aortic repair in 18 patients. The mean age was 68 years (12 men, 6 women). All of the patients had significant comorbidities. Follow-up computed tomography (CT) scans were performed at 1 week, 3 months, 6 months, and annually thereafter.</p><p><b>Results:</b> All patients were operated on in a staged procedure and stent graft deployment was achieved. Procedural success was achieved in all cases. All patients were discharged with complete recovery. No endoleaks weres detected in further CT examination.</p><p><b>Conclusion:</b> Our results suggests that hybrid debranching and endovascular repair of extensive thoracoabdominal aneurysms represents a suitable therapeutic option to reduce the morbidity and mortality of TAAA repair, particularly in those typically considered at high risk for standard repair.</p>

Spontaneous intracerebral hemorrhage in a child with systemic hypertension and adrenal adenoma

2007 ◽  
Vol 107 (4) ◽  
pp. 328-329
Author(s):  
Oszkar Szentirmai ◽  
Ken R. Winston ◽  
Paul Jedlicka ◽  
Gary M. Lum
Keyword(s):  
Intracerebral Hemorrhage ◽  
Adrenal Adenoma ◽  
Systemic Hypertension ◽  
Spontaneous Intracerebral Hemorrhage

Epicardial Coronary Artery Remodeling in Systemic Hypertension

2005 ◽  
Vol 12 (3) ◽  
pp. 164
Author(s):  
M. Koz??kov?? ◽  
M. Paterni ◽  
F. Bartolomucci ◽  
C. Morizzo ◽  
C. Palombo
Keyword(s):  
Coronary Artery ◽  
Systemic Hypertension ◽  
Epicardial Coronary Artery ◽  
Artery Remodeling

Cardiovascular Changes in Menopause

2020 ◽  
Vol 16 ◽  
Author(s):  
Anjana R Nair ◽  
Aiswarya J Pillai ◽  
Nandini Nair
Keyword(s):  
Cardiovascular Risk ◽  
Structural Changes ◽  
Systemic Hypertension ◽  
Metabolic Dysfunction ◽  
Review Of Literature ◽  
Coronary Anatomy ◽  
Complex Interactions ◽  
Fat Redistribution ◽  
Current Therapies ◽  
Cardiovascular Aging

: Menopause is associated with changes consistent with cardiovascular aging. The effects on cardiac disease is multifaceted affecting endothelial function, coronary artery physiology and metabolic dysfunction leading to structural changes in the coronary anatomy. A systematic review of literature from 1986 to 2019 was conducted using PubMed and Google Scholar. The search was directed to retrieve papers that addressed the changes in cardiovascular physiology in menopause and the current therapies available to treat cardiovascular manifestations of menopause. The metabolic and clinical factors secondary to menopause such as dyslipidemia, insulin resistance, fat redistribution and systemic hypertension contribute to the accelerated risk for cardiovascular aging and disease. Atherosclerosis appears to be the end result of the interaction between cardiovascular risk factors and their accentuation during the perimenopausal period. Additionally, complex interactions between oxidative stress and levels of L-arginine and ADMA may also influence endothelial dysfunction in menopause. The increased cardiovascular risk in menopause stems from the exaggerated effects of changing physiology on the cardiovascular system affecting peripheral, cardiac and cerebrovascular beds. The differential effects of menopause on cardiovascular disease at the subclinical, biochemical and molecular levels form the highlights of this review.

scholarly journals Is hypertensive left ventricular hypertrophy a cause of sustained ventricular arrhythmias in humans?

2021 ◽  
Author(s):  
R. Nadarajah ◽  
P. A. Patel ◽  
M. H. Tayebjee
Keyword(s):  
Left Ventricular Hypertrophy ◽  
Ventricular Arrhythmias ◽  
Ventricular Hypertrophy ◽  
Left Ventricular ◽  
Systemic Hypertension ◽  
Electrolyte Disturbance ◽  
Electrocardiographic Monitoring ◽  
Homogenous Distribution ◽  
Artery Disease ◽  
Asymptomatic Coronary Artery Disease

AbstractSudden cardiac death (SCD) is most commonly secondary to sustained ventricular arrhythmias (VAs). This review aimed to evaluate if left ventricular hypertrophy (LVH) secondary to systemic hypertension in humans is an isolated risk factor for ventricular arrhythmogenesis. Animal models of hypertensive LVH have shown changes in ion channel function and distribution, gap junction re-distribution and fibrotic deposition. Clinical data has consistently exhibited an increase in prevalence and complexity of non-sustained VAs on electrocardiographic monitoring. However, there is a dearth of trials suggesting progression to sustained VAs and SCD, with extrapolations being confounded by presence of co-existent asymptomatic coronary artery disease (CAD). Putatively, this lack of data may be due to the presence of more homogenous distribution of pathophysiological changes seen in those with hypertensive LVH versus known pro-arrhythmic conditions such as HCM and myocardial infarction. The overall impression is that sustained VAs in the context of hypertensive LVH are most likely to be precipitated by other causes such as CAD or electrolyte disturbance.

Sign in / Sign up

Export Citation Format

Share Document