scholarly journals Overexpression of Orail Contributes to Abnormal Cell Proliferation in Esophageal Squamous Carcinoma Cancer

2011 ◽  
Vol 100 (3) ◽  
pp. 83a
Author(s):  
Hua Zhu ◽  
Mingzhu Fang ◽  
Chung S. Yang ◽  
Zui Pan
Keyword(s):  
Cell Proliferation ◽  
Squamous Carcinoma ◽  
Abnormal Cell ◽  
Esophageal Squamous Carcinoma ◽  
Abnormal Cell Proliferation

Carvacrol Induces Cell Apoptosis in Human Esophageal Squamous Carcinoma the Human Esophageal Squamous Carcinoma Cell Line (KYSE-150) Cells

2021 ◽  
Vol 11 (9) ◽  
pp. 1844-1847
Author(s):  
DaoBin Lin ◽  
Zhangkai Yang ◽  
YiHua Su ◽  
YaWei Cheng
Keyword(s):  
Cell Proliferation ◽  
Esophageal Cancer ◽  
Cell Apoptosis ◽  
Squamous Carcinoma ◽  
Mrna Levels ◽  
Dependent Manner ◽  
Anti Cancer ◽  
Esophageal Squamous Carcinoma Cell ◽  
Esophageal Squamous Carcinoma ◽  
Squamous Carcinoma Cell

Carvacrol is a natural phenol with antioxidant, antimicrobials, and anti-cancer activities. The present study aimed to explore the anti-cancer activity of carvacrol in human esophageal squamous carcinoma KYSE-150 cells. Cell proliferation and apoptosis were measured using WST-1 assay and cell death detection ELISA kit respectively. Caspase activities were determined with caspase fluorometric assay kits. WST-1 assay revealed that carvacrol suppressed KYSE-150 cell proliferation in both dose and time-dependent manner. Carvacrol could increase cell apoptosis in a dose-dependent manner through caspase-3 and caspase-9 activation. In addition, Bcl-2 mRNA levels were significantly decreased by carvacrol treatment. This study delivers that carvacrol exerts a favorable anti-cancer action against esophageal cancer via inhibiting cell proliferation and inducing cell apoptosis. These discoveries suggest that carvacrol is deserved to be further studied and developed as a chemotherapeutic agent for esophageal cancer.

Abnormal cell proliferation and p52/p35-CSK expression in the colons of aging rats

1995 ◽  
Vol 30 (5) ◽  
pp. 495-503 ◽  
Author(s):  
Peter R. Holt ◽  
Paul J. Higgins ◽  
Evren Atillasoy ◽  
Paul J. Davis ◽  
Martin Lipkin
Keyword(s):  
Cell Proliferation ◽  
Aging Rats ◽  
Abnormal Cell ◽  
Abnormal Cell Proliferation

scholarly journals NF-κB is required for H-ras oncogene induced abnormal cell proliferation and tumorigenesis

Oncogene ◽  
2000 ◽  
Vol 19 (7) ◽  
pp. 841-849 ◽  
Author(s):  
Hakryul Jo ◽  
Rong Zhang ◽  
Hong Zhang ◽  
Timothy A McKinsey ◽  
Jinyi Shao ◽  
...  
Keyword(s):  
Cell Proliferation ◽  
Ras Oncogene ◽  
Abnormal Cell ◽  
Abnormal Cell Proliferation
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