scholarly journals Interactions of oxygen with intrinsic defects in L10 γ-TiAl in presence of substitutional solutes: Influence on diffusion kinetics

2022 ◽  
Vol 201 ◽  
pp. 110933
Author(s):  
Camille Thenot ◽  
Rémy Besson ◽  
Pierre Sallot ◽  
Jean-Philippe Monchoux ◽  
Damien Connétable
Keyword(s):  
Diffusion Kinetics ◽  
Intrinsic Defects

DIFFUSION KINETICS IN GOLD-AMORPHOUS GeTe4THIN FILMS

1981 ◽  
Vol 42 (C4) ◽  
pp. C4-975-C4-978 ◽  
Author(s):  
J. M. Mackowski ◽  
M. Bendali ◽  
P. Normandon ◽  
P. Kumurdjian
Keyword(s):  
Diffusion Kinetics

Na, K, Rb, and Cs exchange in heulandite single crystals; diffusion kinetics

1997 ◽  
Vol 82 (5-6) ◽  
pp. 517-525 ◽  
Author(s):  
Ping Yang ◽  
Jano Stolz ◽  
Thomas Armbruster ◽  
Mickey E. Gunter
Keyword(s):  
Single Crystals ◽  
Diffusion Kinetics

Kinetics of Methane-Oxidizing Biofilms for Degradation of Toxic Organics

1988 ◽  
Vol 20 (11-12) ◽  
pp. 167-173 ◽  
Author(s):  
S. E. Strand ◽  
R. M. Seamons ◽  
M. D. Bjelland ◽  
H. D. Stensel
Keyword(s):  
Chlorinated Hydrocarbons ◽  
Enzymatic Oxidation ◽  
Diffusion Kinetics ◽  
Toxic Compound ◽  
Biofilm Model ◽  
Uptake Rates ◽  
Substrate Diffusion ◽  
Toxic Organics ◽  
Competitive Substrate ◽  
Kinetics Of

The kinetics of methane-oxidizing bioreactors for the degradation of toxic organics are modeled. Calculations of the fluxes of methane and toxic chlorinated hydrocarbons were made using a biofilm model. The model simulated the effects of competition by toxics and mediane on their enzymatic oxidation by the methane monooxygenase. Dual-competitive-substrate/diffusion kinetics were used to model biofilm co-metabolism, integrating equations of the following form:where S1 and S2 are the local concentrations of methane and toxic compound, respectively, and r and K are the maximum uptake rates and Monod coefficients, and x is the distance into the biofilm.

Hydrogen diffusion kinetics under different conditions applied to VT6 alloy

2020 ◽  
pp. 98-107
Author(s):  
E. I. Maslikova ◽  
V. D. Andreeva ◽  
E. L. Alekseeva ◽  
Yu. A. Yakovlev
Keyword(s):  
Heat Treatment ◽  
Oxide Film ◽  
Titanium Alloys ◽  
Hydrogen Diffusion ◽  
Heat Treating ◽  
Protective Atmosphere ◽  
Diffusion Kinetics ◽  
Hydrogen Charging ◽  
Hydride Formation ◽  
Different Types

Research of hydrogen diffusion in VT6 alloy is carried out considering different types of heat treating and hydrogen charging. The influence of microalloying on the susceptibility to hydride formation and embrittlement of titanium alloys is analyzed, and also effects of an oxide film on hydrogen charging during heat treatment without protective atmosphere, are studied.

Highly electroconductive and uniform WS2 film growth by sulfurization of W film using diethyl sulfide

2021 ◽  
Author(s):  
Yoobeen Lee ◽  
Jin Won Jung ◽  
Jin Seok Lee
Keyword(s):  
Transition Metal ◽  
Grain Boundaries ◽  
High Performance ◽  
Film Growth ◽  
Transition Metal Dichalcogenides ◽  
Electronic Systems ◽  
Intrinsic Defects ◽  
Diethyl Sulfide ◽  
Metal Dichalcogenides

The reduction of intrinsic defects, including vacancies and grain boundaries, remains one of the greatest challenges to produce high-performance transition metal dichalcogenides (TMDCs) electronic systems. A deeper comprehension of the...

scholarly journals The Interplay between Drivers of Erythropoiesis and Iron Homeostasis in Rare Hereditary Anemias: Tipping the Balance

2021 ◽  
Vol 22 (4) ◽  
pp. 2204
Author(s):  
Simon Grootendorst ◽  
Jonathan de Wilde ◽  
Birgit van Dooijeweert ◽  
Annelies van Vuren ◽  
Wouter van Solinge ◽  
...  
Keyword(s):  
Iron Overload ◽  
Iron Metabolism ◽  
Iron Homeostasis ◽  
Significant Problem ◽  
Blood Transfusions ◽  
Intrinsic Defects ◽  
Chronic Anemia ◽  
Erythroid Development ◽  
Disease Specific

Rare hereditary anemias (RHA) represent a group of disorders characterized by either impaired production of erythrocytes or decreased survival (i.e., hemolysis). In RHA, the regulation of iron metabolism and erythropoiesis is often disturbed, leading to iron overload or worsening of chronic anemia due to unavailability of iron for erythropoiesis. Whereas iron overload generally is a well-recognized complication in patients requiring regular blood transfusions, it is also a significant problem in a large proportion of patients with RHA that are not transfusion dependent. This indicates that RHA share disease-specific defects in erythroid development that are linked to intrinsic defects in iron metabolism. In this review, we discuss the key regulators involved in the interplay between iron and erythropoiesis and their importance in the spectrum of RHA.

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