Fuzhuan brick tea extract prevents diet-induced obesity via stimulation of fat browning in mice

2022 ◽  
pp. 132006
Author(s):  
Ahyoung Yoo ◽  
Min Jung Kim ◽  
Jiyun Ahn ◽  
Chang Hwa Jung ◽  
Hyo Deok Seo ◽  
...  
Keyword(s):  
Diet Induced Obesity ◽  
Tea Extract ◽  
Stimulation Of

scholarly journals White tea (Camellia sinensis) extract reduces oxidative stress and triacylglycerols in obese mice

2012 ◽  
Vol 32 (4) ◽  
pp. 733-741 ◽  
Author(s):  
Lílian Gonçalves Teixeira ◽  
Priscilla Ceci Lages ◽  
Tatianna Lemos Jascolka ◽  
Edenil Costa Aguilar ◽  
Fabíola Lacerda Pires Soares ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Body Weight ◽  
Adipose Tissue ◽  
Camellia Sinensis ◽  
Reduce Food Intake ◽  
Obese Mice ◽  
White Tea ◽  
Diet Induced Obesity ◽  
Tea Extract ◽  
The Relationship

White tea is an unfermented tea made from young shoots of Camellia sinensis protected from sunlight to avoid polyphenol degradation. Although its levels of catechins are higher than those of green tea (derived from the same plant), there are no studies addressing the relationship between this tea and obesity associated with oxidative stress.The objective of this study was to evaluate the effect of white tea on obesity and its complications using a diet induced obesity model. Forty male C57BL/6 mice were fed a high-fat diet to induce obesity (Obese group) or the same diet supplemented with 0.5% white tea extract (Obese + WTE) for 8 weeks. Adipose tissue, serum lipid profile, and oxidative stress were studied. White tea supplementation was not able to reduce food intake, body weight, or visceral adiposity. Similarly, there were no changes in cholesterol rich lipoprotein profile between the groups. A reduction in blood triacylglycerols associated with increased cecal lipids was observed in the group fed the diet supplemented with white tea. White tea supplementation also reduced oxidative stress in liver and adipose tissue. In conclusion, white tea extract supplementation (0.5%) does not influence body weight or adiposity in obese mice. Its benefits are restricted to the reduction in oxidative stress associated with obesity and improvement of hypertriacylglycerolemia.

scholarly journals Beneficial effects of tea catechins on diet-induced obesity: stimulation of lipid catabolism in the liver

2002 ◽  
Vol 26 (11) ◽  
pp. 1459-1464 ◽  
Author(s):  
T Murase ◽  
A Nagasawa ◽  
J Suzuki ◽  
T Hase ◽  
I Tokimitsu
Keyword(s):  
Tea Catechins ◽  
Beneficial Effects ◽  
Lipid Catabolism ◽  
Diet Induced Obesity ◽  
Stimulation Of

scholarly journals Novel discovery of Averrhoa bilimbi ethanolic leaf extract in the stimulation of brown fat differentiation program in combating diet-induced obesity

2019 ◽  
Vol 19 (1) ◽  
Author(s):  
Wai Kwan Lau ◽  
Nur Adelina Ahmad Noruddin ◽  
Abdul Hadi Ariffin ◽  
Muhd Zulkarnain Mahmud ◽  
Mohd Hasnan Mohd Noor ◽  
...  
Keyword(s):  
Leaf Extract ◽  
Brown Fat ◽  
Diet Induced Obesity ◽  
Ethanolic Leaf Extract ◽  
Stimulation Of

Mo1970 UNIQUE MECHANISM OF STIMULATION OF GLUCOSE AND NACL ABSORPTION IN DIET-INDUCED OBESITY

2020 ◽  
Vol 158 (6) ◽  
pp. S-996
Author(s):  
Balasubramanian Palaniappan ◽  
RAJA SINGH PAULRAJ ◽  
Niraj Nepal ◽  
Subha Arthur ◽  
Uma Sundaram
Keyword(s):  
Nacl Absorption ◽  
Diet Induced Obesity ◽  
Unique Mechanism ◽  
Stimulation Of

Effects of 5-5'-Diphenyl-2-thiohydantoin (DPTH) and Thyroxine on the Ultrastructure and Chemical Composition of Rat Liver

1971 ◽  
Vol 29 ◽  
pp. 570-571
Author(s):  
E. A. Elfont ◽  
R. B. Tobin ◽  
D. G. Colton ◽  
M. A. Mehlman
Keyword(s):  
Chemical Composition ◽  
Rat Liver ◽  
Future Study ◽  
Mode Of Action ◽  
Liver Mitochondria ◽  
Rat Liver Mitochondria ◽  
Effective Inhibitor ◽  
Biochemical Effects ◽  
Glycerophosphate Dehydrogenase ◽  
Stimulation Of

Summary5,-5'-diphenyl-2-thiohydantoin (DPTH) is an effective inhibitor of thyroxine (T4) stimulation of α-glycerophosphate dehydrogenase in rat liver mitochondria. Because this finding indicated a possible tool for future study of the mode of action of thyroxine, the ultrastructural and biochemical effects of DPTH and/or thyroxine on rat liver mere investigated.Rats were fed either standard or DPTH (0.06%) diet for 30 days before T4 (250 ug/kg/day) was injected. Injection of T4 occurred daily for 10 days prior to sacrifice. After removal of the liver and kidneys, part of the tissue was frozen at -50°C for later biocheailcal analyses, while the rest was prefixed in buffered 3.5X glutaraldehyde (390 mOs) and post-fixed in buffered 1Z OsO4 (376 mOs). Tissues were embedded in Araldlte 502 and the sections examined in a Zeiss EM 9S.Hepatocytes from hyperthyroid rats (Fig. 2) demonstrated enlarged and more numerous mitochondria than those of controls (Fig. 1). Glycogen was almost totally absent from the cytoplasm of the T4-treated rats.

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