Characterization of an NBS-LRR resistance gene homologue from soybean

2004 ◽  
Vol 161 (7) ◽  
pp. 815-822 ◽  
Author(s):  
Bangjun Wang ◽  
Yongjun Wang ◽  
Qiang Wang ◽  
Guangzuo Luo ◽  
Zhigang Zhang ◽  
...  
2004 ◽  
Vol 54 (2) ◽  
pp. 533-545 ◽  
Author(s):  
Nienke Westerink ◽  
Bas F. Brandwagt ◽  
Pierre J. G. M. De Wit ◽  
Matthieu H. A. J. Joosten

Genetics ◽  
1999 ◽  
Vol 153 (4) ◽  
pp. 1929-1948 ◽  
Author(s):  
Fusheng Wei ◽  
Karin Gobelman-Werner ◽  
Shaun M Morroll ◽  
Joachim Kurth ◽  
Long Mao ◽  
...  

Abstract Powdery mildew of barley, caused by Erysiphe graminis f. sp. hordei, is a model system for investigating the mechanism of gene-for-gene interaction between large-genome cereals and obligate-fungal pathogens. A large number of loci that confer resistance to this disease are located on the short arm of chromosome 5(1H). The Mla resistance-gene cluster is positioned near the telomeric end of this chromosome arm. AFLP-, RAPD-, and RFLP-derived markers were used to saturate the Mla region in a high-resolution recombinant population segregating for the (Mla6 + Mla14) and (Mla13 + Ml-Ru3) resistance specificities. These tightly linked genetic markers were used to identify and develop a physical contig of YAC and BAC clones spanning the Mla cluster. Three distinct NBS-LRR resistance-gene homologue (RGH) families were revealed via computational analysis of low-pass and BAC-end sequence data derived from Mla-spanning clones. Genetic and physical mapping delimited the Mla-associated, NBS-LRR gene families to a 240-kb interval. Recombination within the RGH families was at least 10-fold less frequent than between markers directly adjacent to the Mla cluster.


2018 ◽  
Vol 44 (4) ◽  
pp. 473 ◽  
Author(s):  
Zhan-Wang ZHU ◽  
Deng-An XU ◽  
Shun-He CHENG ◽  
Chun-Bao GAO ◽  
Xian-Chun XIA ◽  
...  

1991 ◽  
Vol 55 (7) ◽  
pp. 1913-1918
Author(s):  
Ryutaro Utsumi ◽  
Tadashi Yagi ◽  
Satoshi Katayama ◽  
Kiyonori Katsuragi ◽  
Kouji Tachibana ◽  
...  

Plant Science ◽  
2003 ◽  
Vol 164 (1) ◽  
pp. 71-75 ◽  
Author(s):  
Maria Rosaria Cortese ◽  
Elena Fanelli ◽  
Carla De Giorgi

1997 ◽  
Vol 41 (2) ◽  
pp. 314-318 ◽  
Author(s):  
E Hannecart-Pokorni ◽  
F Depuydt ◽  
L de wit ◽  
E van Bossuyt ◽  
J Content ◽  
...  

The amikacin resistance gene aac(6')-Im [corrected] from Citrobacter freundii Cf155 encoding an aminoglycoside 6'-N-acetyltransferase was characterized. The gene was identified as a coding sequence of 521 bp located down-stream from the 5' conserved segment of an integron. The sequence of this aac(6')-Im [corrected] gene corresponded to a protein of 173 amino acids which possessed 64.2% identity in a 165-amino-acid overlap with the aac(6')-Ia gene product (F.C. Tenover, D. Filpula, K.L. Phillips, and J. J. Plorde, J. Bacteriol. 170:471-473, 1988). By using PCR, the aac(6')-Im [corrected] gene could be detected in 8 of 86 gram-negative clinical isolates from two Belgian hospitals, including isolates of Citrobacter, Klebsiella spp., and Escherichia coli. PCR mapping of the aac(6')-Im [corrected] gene environment in these isolates indicated that the gene was located within a sulI-type integron; the insert region is 1,700 bases long and includes two genes cassettes, the second being ant (3")-Ib.


Sign in / Sign up

Export Citation Format

Share Document