scholarly journals Left thoracoscopic sympathectomy for cardiac denervation in patients with life-threatening ventricular arrhythmias

2014 ◽  
Vol 147 (1) ◽  
pp. 404-411 ◽  
Author(s):  
Sophie C. Hofferberth ◽  
Frank Cecchin ◽  
Dan Loberman ◽  
Francis Fynn-Thompson
EP Europace ◽  
2017 ◽  
Vol 19 (suppl_3) ◽  
pp. iii196-iii196
Author(s):  
S. Termosesov ◽  
I. Khamnagadaev ◽  
R. Garipov ◽  
Y. Volkova ◽  
R. Ildarova ◽  
...  

Medicina ◽  
2021 ◽  
Vol 57 (3) ◽  
pp. 205
Author(s):  
Nicola Tarantino ◽  
Domenico G. Della Rocca ◽  
Nicole S. De Leon De La Cruz ◽  
Eric D. Manheimer ◽  
Michele Magnocavallo ◽  
...  

A recent surveillance analysis indicates that cardiac arrest/death occurs in ≈1:50,000 professional or semi-professional athletes, and the most common cause is attributable to life-threatening ventricular arrhythmias (VAs). It is critically important to diagnose any inherited/acquired cardiac disease, including coronary artery disease, since it frequently represents the arrhythmogenic substrate in a substantial part of the athletes presenting with major VAs. New insights indicate that athletes develop a specific electro-anatomical remodeling, with peculiar anatomic distribution and VAs patterns. However, because of the scarcity of clinical data concerning the natural history of VAs in sports performers, there are no dedicated recommendations for VA ablation. The treatment remains at the mercy of several individual factors, including the type of VA, the athlete’s age, and the operator’s expertise. With the present review, we aimed to illustrate the prevalence, electrocardiographic (ECG) features, and imaging correlations of the most common VAs in athletes, focusing on etiology, outcomes, and sports eligibility after catheter ablation.


2017 ◽  
Vol 36 (4) ◽  
pp. S432 ◽  
Author(s):  
J.G. Jungschleger ◽  
M.M. Mydin ◽  
A. Woods ◽  
M. Urban ◽  
N. Robinson ◽  
...  

2020 ◽  
Vol 22 (12) ◽  
Author(s):  
Lisa D. Wilsbacher

Abstract Purpose of Review Dilated cardiomyopathy (DCM) frequently involves an underlying genetic etiology, but the clinical approach for genetic diagnosis and application of results in clinical practice can be complex. Recent Findings International sequence databases described the landscape of genetic variability across populations, which informed guidelines for the interpretation of DCM gene variants. New evidence indicates that loss-of-function mutations in filamin C (FLNC) contribute to DCM and portend high risk of ventricular arrhythmia. Summary A clinical framework aids in referring patients for DCM genetic testing and applying results to patient care. Results of genetic testing can change medical management, particularly in a subset of genes that increase risk for life-threatening ventricular arrhythmias, and can influence decisions for defibrillator therapy. Clinical screening and cascade genetic testing of family members should be diligently pursued to identify those at risk of developing DCM.


1995 ◽  
Vol 28 ◽  
pp. 71-73
Author(s):  
Labros A. Karagounis ◽  
Kenneth M. Stein ◽  
Tami Bair ◽  
Derrick Albright ◽  
Jeffrey L. Anderson

2021 ◽  
Vol 5 (6) ◽  
Author(s):  
Kyunghee Lim ◽  
Jong Sung Park ◽  
Byeol-A Yoon ◽  
Song-Hee Han

Abstract Background Necrotizing autoimmune myopathy is a rare subtype of idiopathic inflammatory myopathy; however, it can be associated with fatal cardiac manifestations. Case summary A 58-year-old female patient was referred for congestive heart failure with dysrhythmia. Electrocardiograms showed ventricular arrhythmias of various QRS complex morphologies and coupling intervals with beat-to-beat differences. Despite optimal medical therapy for heart failure, the patient was admitted for the progression of dyspnoea and generalized motor weakness. The burden of non-sustained ventricular tachycardia gradually increased, and ventricular fibrillation eventually occurred. In view of a differential diagnosis of an inflammatory myocardial diseases such as sarcoidosis, a cardiac biopsy was performed. However, pathologic examinations revealed only necrotic muscle fibres without granuloma. Further examinations revealed proximal dominant motor weakness, an elevated serum creatinine-phosphokinase level, myogenic potentials on needle electromyography, and biceps muscle biopsy findings that were compatible with necrotizing autoimmune myopathy. High-dose steroid therapy improved the patient’s motor weakness, including her respiratory impairment, and successfully suppressed ventricular arrhythmias. Discussion This case suggests that intensive immunosuppressive therapy with high-dose steroid could be useful in the necrotizing autoimmune myopathy manifested as congestive heart failure and life-threatening ventricular arrhythmias.


2021 ◽  
Vol 15 (2) ◽  
pp. 207
Author(s):  
CamilaBiazussi Damasceno ◽  
MarinaAyres Delgado ◽  
PaulaAlves Pinheiro ◽  
IsadoraMegale Quadros

1984 ◽  
Vol 4 (5) ◽  
pp. 952-957 ◽  
Author(s):  
Declan D. Sugrue ◽  
David R. Holmes ◽  
Bernard J. Gersh ◽  
William D. Edwards ◽  
Christopher J. McLaran ◽  
...  

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