Effect of high intensity training on anaerobic capacity of middle gluteal muscle in Thoroughbred horses

2004 ◽  
Vol 76 (2) ◽  
pp. 139-144 ◽  
Author(s):  
D. Eto ◽  
S. Yamano ◽  
K. Mukai ◽  
T. Sugiura ◽  
T. Nasu ◽  
...  
2009 ◽  
Vol 6 (04) ◽  
pp. 171-175 ◽  
Author(s):  
Yu Kitaoka ◽  
Yuki Wakasugi ◽  
Daisuke Hoshino ◽  
Kazutaka Mukai ◽  
Atsushi Hiraga ◽  
...  

2010 ◽  
Vol 42 ◽  
pp. 87
Author(s):  
Yu Kitaoka ◽  
Yukari Endo ◽  
Kazutaka Mukai ◽  
Atsushi Hiraga ◽  
Tohru Takemasa ◽  
...  

2003 ◽  
Vol 94 (2) ◽  
pp. 733-743 ◽  
Author(s):  
M. J. Truijens ◽  
H. M. Toussaint ◽  
J. Dow ◽  
B. D. Levine

The objective of this study was to test the hypothesis that high-intensity hypoxic training improves sea-level performances more than equivalent training in normoxia. Sixteen well-trained collegiate and Masters swimmers (10 women, 6 men) completed a 5-wk training program, consisting of three high-intensity training sessions in a flume and supplemental low- or moderate-intensity sessions in a pool each week. Subjects were matched for gender, performance level, and training history, and they were assigned to either hypoxic [Hypo; inspired O2 fraction (Fi O2 ) = 15.3%, equivalent to a simulated altitude of 2,500 m] or normoxic (Norm; Fi O2 = 20.9%) interval training in a randomized, double-blind, placebo-controlled design. All pool training occurred under Norm conditions. The primary performance measures were 100- and 400-m freestyle time trials. Laboratory outcomes included maximal O2 uptake (V˙o 2 max), anaerobic capacity (accumulated O2 deficit), and swimming economy. Significant ( P = 0.02 and <0.001 for 100- and 400-m trials, respectively) improvements were found in performance on both the 100- [Norm: −0.7 s (95% confidence limits: +0.2 to −1.7 s), −1.2%; Hypo: −0.8 s (95% confidence limits: −0.1 to −1.5 s), −1.1%] and 400-m freestyle [Norm: −3.6 s (−1.8 to −5.5 s), −1.2%; Hypo: −5.3 s (−2.3 to −8.3 s), −1.7%]. There was no significant difference between groups for either distance (ANOVA interaction, P = 0.91 and 0.36 for 100- and 400-m trials, respectively). V˙o 2 max was improved significantly (Norm: 0.16 ± 0.23 l/min, 6.4 ±8.1%; Hypo: 0.11 ± 0.18 l/min, 4.2 ± 7.0%). There was no significant difference between groups ( P = 0.58). We conclude that 5 wk of high-intensity training in a flume improves sea-level swimming performances andV˙o 2 max in well-trained swimmers, with no additive effect of hypoxic training.


2021 ◽  
Vol 11 (1) ◽  
Author(s):  
Eugenia Murawska-Ciałowicz ◽  
Gilmara Gomes de Assis ◽  
Filipe Manuel Clemente ◽  
Yuri Feito ◽  
Petr Stastny ◽  
...  

AbstractThis study examined the effects of a nine-week intervention of four different high-intensity training modalities [high-intensity functional training (HIFT), high-intensity interval training (HIIT), high-intensity power training (HIPT), and high-intensity endurance training (HIET)] on the resting concentration of brain-derived neurotropic factor (BDNF). In addition, we evaluated the BDNF responses to Graded Exercise Test (GXT) and Wingate Anaerobic Test (WAnT) in men. Thirty-five healthy individuals with body mass index 25.55 ± 2.35 kg/m2 voluntarily participated in this study and were randomly assigned into four training groups. During nine-weeks they completed three exercise sessions per week for one-hour. BDNF was analyzed before and after a GXT and WAnT in two stages: (stage 0—before training and stage 9—after nine weeks of training). At stage 0, an increase in BDNF concentration was observed in HIFT (33%; p < 0.05), HIPT (36%; p < 0.05) and HIIT (38%; p < 0.05) after GXT. Even though HIET showed an increase in BDNF (10%) this was not statistically significant (p > 0.05). At stage 9, higher BDNF levels after GXT were seen only for the HIFT (30%; p < 0.05) and HIIT (18%; p < 0.05) groups. Reduction in BDNF levels were noted after the WAnT in stage 0 for HIFT (− 47%; p < 0.01), HIPT (− 49%; p < 0.001), HIET (− 18%; p < 0.05)], with no changes in the HIIT group (− 2%). At stage 9, BDNF was also reduced after WAnT, although these changes were lower compared to stage 0. The reduced level of BDNF was noted in the HIFT (− 28%; p < 0.05), and HIPT (− 19%;p < 0.05) groups. Additionally, all groups saw an improvement in VO2max (8%; p < 0.001), while BDNF was also correlated with lactate and minute ventilation and selected WAnT parameters. Our research has shown that resting values of BDNF after nine weeks of different forms of high-intensity training (HIT) have not changed or were reduced. Resting BDNF measured at 3th (before GXT at stage 9) and 6th day after long lasting HITs (before WAnT at stage 9) did not differed (before GXT), but in comparison to the resting value before WAnT at the baseline state, was lower in three groups. It appears that BDNF levels after one bout of exercise is depended on duration time, intensity and type of test/exercise.


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