Relationship between pregnancy-induced hypertension and placenta previa: A population-based study

Background: Grand multiparity considered as a risk factor of obstetrics because of the recorded complications linked to the condition. Grand multiparity typically considered as the distinctive reason for the raised, maternal and fetal morbidity and mortality because of expanded incidence of adverse outcome during pregnancy and birth. Objective: To determine frequency of antenatal complications in grand multipara. Study Design: Cross-sectional study. Settings: Department of Obstetrics and Gynecology, Jinnah Postgraduate Medical Centre, Karachi Pakistan. Duration: Study duration was six months from March 2016 September 2016. Methodology: Total 212 patients were included in this study. Anemia was taken as Hb of 11g/dl, PIH was taken as BP of >140/90mmHg after 20 weeks of gestation with or without proteinuria on two or more occasion 6 hours apart and placenta previa was confirmed via ultrasonography. All the information was collected via study proforma. Results: Patients mean age was 34.90±3.51 years. Most of the patients 96.7% had parity 5-9. Anemia was found 69.8% and pregnancy induced hypertension was 22.2%, while placenta previa was found to be 18.9%. Antenatal complications including anemia, pregnancy induced hypertension and placenta previa were found to be statistically insignificant according to age, parity and BMI, (p->0.05). Conclusion: Most common antenatal complication in this study was anemia followed by hypertension and placenta previa. Grand multiparity is at a greater risk of antenatal complications.

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Relationship between placenta previa and preeclampsia: A population-based study

American Journal of Obstetrics and Gynecology ◽

10.1016/s0002-9378(97)80419-x ◽

1997 ◽

Vol 176 (1) ◽

pp. S106

Author(s):

CV Ananth ◽

WA Bowes ◽

DA Savitz ◽

ER Luther

Keyword(s):

Placenta Previa ◽

Population Based ◽

Population Based Study

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Placental Infarcts, Intervillous Fibrin Plaques, and Intervillous Thrombi: Incidences, Cooccurrences, and Epidemiological Associations

Pediatric and Developmental Pathology ◽

10.1007/s10024-003-4032-3 ◽

2004 ◽

Vol 7 (1) ◽

pp. 26-34 ◽

Cited By ~ 23

Author(s):

David M.O. Becroft ◽

John M. D. Thompson ◽

Edwin A. Mitchell

Keyword(s):

Gestational Age ◽

Case Control Study ◽

Population Based ◽

Pregnancy Induced Hypertension ◽

Related Factors ◽

Induced Hypertension ◽

Appropriate For Gestational Age ◽

Pathogenetic Factor ◽

Clinical Associations ◽

Control Study

The incidences, cooccurrences and epidemiological associations at term of the three common focal macroscopic placental lesions, infarcts, intervillous fibrin plaques (IVFP), and intervillous thrombi (IVT) were investigated as part of a population-based case-control study of small-for-gestational age (SGA) infants. Five hundred and nine placentas from women delivering SGA infants (10th percentile or less for gestational age) and 529 placentas from women delivering infants with birth-weights appropriate for gestational age were examined using fixed protocols for identification of macroscopic lesions and microscopic diagnoses. One or more of these lesions were found in 280 placentas (28%), including infarcts in 150 (15%), IVFP in 132 (13%), and IVT in 64 (6%). Macroscopic misidentifications, particularly of IVFP as infarcts, emphasize the need for microscopic diagnoses. There were strong associations between the occurrence of any one type of lesion and cooccurrence of either of the other two, and these associations were site-dependent: between central (nonmarginal) infarcts and central IVFP ( P = 0.0023); marginal infarcts and marginal IVFP ( P < 0.0001); and between IVT (all central) and marginal infarcts ( P < 0.0001) and marginal IVFP ( P = 0.012). However, a study of associations between the incidences of placentas bearing each of the three lesions and 31 socio-demographic and pregnancy-related factors showed no associations in common. IVFP, an IVFP variant termed “labyrinthine,” and IVT did not show any of the independent associations demonstrated between infarcts and SGA, pregnancy-induced hypertension, nonsmoking, age at first pregnancy, and ethnicity. IVFP had no significant associations, and IVT were associated only with male gender. The study has shown that IVFP and IVT do not share the important clinical associations demonstrated for infarcts, but has not identified the pathogenetic factor or factors responsible for the frequent cooccurrence of these lesions. The maternal thrombophilias may have such a role.

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Association Between Pregnancy Induced Hypertension and Low Birth Weight; A Population Based Case-Control Study

Asia Pacific Journal of Public Health ◽

10.1177/1010539507311553 ◽

2008 ◽

Vol 20 (2) ◽

pp. 152-158 ◽

Cited By ~ 17

Author(s):

Latifah A. Rahman ◽

Noran N. Hairi ◽

Nooriah Salleh

Keyword(s):

Birth Weight ◽

Low Birth Weight ◽

Case Control Study ◽

Previous History ◽

Population Based ◽

Case Control ◽

Pregnancy Induced Hypertension ◽

Low Birth Weight Babies ◽

Induced Hypertension ◽

Control Study

The purpose of this study was to investigate the association between pregnancy-induced hypertension and low birth weight. A population-based case control study was conducted. Antenatal mothers who attended the government health centers in the district of Kuala Muda, Kedah, Malaysia from June 2003 to May 2004 were recruited. Cases were 312 mothers who delivered low birth weight babies, and controls were 312 mothers who delivered normal birth weight babies. Face-to-face interviews using a structured questionnaire and a review of medical records were carried out. After controlling for important confounders such as gestational age at delivery, maternal age, ethnicity, education, parity, and previous history of abortion, pregnancy-induced hypertension was found to be an independent risk factor (adjusted odds ratio = 5.06; 95% confidence interval: 2.63, 9.71) for low birth weight. There was a significant association of pregnancy-induced hypertension with low birth weight. Women who delivered low birth weight babies were 5 times more likely to have had pregnancy-induced hypertension.

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