Su1478 - Vitamin D Deficiency and Metabolic Bone Disease is Highly Prevalent in Patients with Cirrhosis- a Hospital Based Study

2018 ◽  
Vol 154 (6) ◽  
pp. S-533
Author(s):  
Indu Grover ◽  
Deepak Gunjan ◽  
Namrata Singh ◽  
Ravinder Goswami ◽  
lakshmy Ramakrishnan ◽  
...  
Keyword(s):  
Vitamin D ◽  
Vitamin D Deficiency ◽  
Bone Disease ◽  
Metabolic Bone Disease ◽  
Metabolic Bone

scholarly journals Are There Hallmarks of Child Abuse? I. Osseous Injuries

2016 ◽  
Vol 6 (4) ◽  
pp. 568-590
Author(s):  
Alfredo Walker ◽  
Charis Kepron ◽  
Christopher M. Milroy
Keyword(s):  
Vitamin D ◽  
Child Abuse ◽  
Young Children ◽  
Vitamin D Deficiency ◽  
Bone Disease ◽  
Metabolic Bone Disease ◽  
Rib Fractures ◽  
Metabolic Bone ◽  
Deficiency Rickets ◽  
Metaphyseal Fractures

Fractures are commonly found in cases regarded as child abuse. The most commonly encountered fractures are to the ribs and the metaphyses. This paper examines the specificity of the classical metaphyseal lesion (CML) and rib fractures as hallmarks of child abuse. Recently, vitamin D deficiency (rickets) has been proposed as an alternative cause for the appearances typically described in CML. The literature in this area is examined. Rib fractures have also been highly associated with child abuse, particularly posterior rib fractures. As well as metabolic bone disease, resuscitation has been examined as a cause of rib fractures in young children. The current literature remains strongly supportive of rib fractures and metaphyseal fractures being indicators of child abuse.

Osteomalacia With Hyperostosis in Captive Lesser Hedgehog Tenrecs (Echinops telfairi)

2020 ◽  
Vol 57 (6) ◽  
pp. 885-888
Author(s):  
Elise E. B. LaDouceur ◽  
Brian G. Murphy ◽  
Michael M. Garner ◽  
Andrew N. Cartoceti
Keyword(s):  
Vitamin D ◽  
Vitamin D Deficiency ◽  
Bone Disease ◽  
Metabolic Bone Disease ◽  
Dietary Vitamin ◽  
Dietary History ◽  
Metabolic Bone ◽  
Dietary Vitamin D

Four captive, lesser hedgehog tenrecs ( Echinops telfairi) were euthanized for soft bones that prevented normal mastication and/or ambulation. Antemortem radiographs (available in 2 cases) revealed osteopenia. Antemortem bloodwork (available in 2 cases) revealed hypophosphatemia. Dietary history (available in 2 cases) indicated the animals were eating only insects. Histologically, all examined bones had wide osteoid seams that caused expansion of the cortices. Osteoid deposition was exuberant and it partially filled marrow cavities (hyperostosis). Nondecalcified sections of bone (available in 2 cases) revealed that osteoid was poorly mineralized, consistent with osteomalacia. Insects are poor dietary sources of vitamin D, and dietary vitamin D deficiency is considered the most likely cause for metabolic bone disease in these animals.

Vitamin D levels and signs of metabolic bone disease in adolescents with idiopathic scoliosis

2013 ◽  
Author(s):  
Adodra Annika ◽  
Kouklinos Andreas ◽  
Julies Priscilla ◽  
Shaw Mathew ◽  
Jacobs Benjamin
Keyword(s):  
Vitamin D ◽  
Idiopathic Scoliosis ◽  
Bone Disease ◽  
Metabolic Bone Disease ◽  
Metabolic Bone ◽  
Vitamin D Levels

Hypervitaminosis D After Prolonged Feeding With a Premature Formula

PEDIATRICS ◽  
1993 ◽  
Vol 92 (6) ◽  
pp. 862-864
Author(s):  
YASUSHI NAKO ◽  
NAOBUMI FUKUSHIMA ◽  
TAKESHI TOMOMASA ◽  
KANJI NAGASHIMA ◽  
TAKAYOSHI KUROUME
Keyword(s):  
Vitamin D ◽  
Premature Infant ◽  
Premature Infants ◽  
Bone Disease ◽  
Calcium Intake ◽  
Metabolic Bone Disease ◽  
Hypervitaminosis D ◽  
Severe Hypercalcemia ◽  
Metabolic Bone ◽  
Prolonged Feeding

Hypervitaminosis D is one of the causes of severe hypercalcemia in children. Most cases of hypervitaminosis D during childhood are due to an excessive supplementation of vitamin D by physicians or parents.1,2 To prevent metabolic bone disease of prematurity (rickets of prematurity), formulas designed for premature infants ("premature formulas"), which contain more calcium and vitamin D than standard formulas, are given to premature infants in addition to human milk.1 In some cases, separate vitamin D products are also given to these infants, although requirements for vitamin D and calcium intake in the premature infant and the formerly premature infant have not been fully estimated.1

Metabolic Bone Disease in the CAPD Patient

1983 ◽  
Vol 3 (1_suppl) ◽  
pp. 24-26 ◽  
Author(s):  
Francisco Llach
Keyword(s):  
Vitamin D ◽  
Renal Osteodystrophy ◽  
Bone Disease ◽  
Metabolic Bone Disease ◽  
Osteitis Fibrosa ◽  
Prospective Evaluation ◽  
Metabolic Bone ◽  
Capd Patient

It seems that CAPD may improve some patients with osteomalacia but may be similar to hemodialysis in regard to osteitis fibrosa. However, long-term prospective evaluation of the incidence of bone disease in CAPD patients is necessary before we can determine how CAPD may alter the incidence and expression of renal osteodystrophy. We need more information before we can conclude that CAPD may improve pure osteomalacia. Finally, the data available are insufficient to clarify the role of vitamin D analogues in these patients.

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