scholarly journals ATF2 partly mediated the expressions of proliferative factors and inhibited pro-inflammatory factors’ secretion in arsenite-treated human uroepithelial cells

2017 ◽  
Vol 6 (4) ◽  
pp. 468-476
Author(s):  
Shengnan Liu ◽  
Fei Wang ◽  
Jieyu Liu ◽  
Peiyu Jin ◽  
Xiaoyan Wang ◽  
...  
Keyword(s):  
Transcription Factor ◽  
Epithelial Cell ◽  
Urinary Bladder ◽  
Cell Line ◽  
Inflammatory Factors ◽  
Epithelial Cell Line ◽  
Bladder Epithelial Cell ◽  
Human Urinary Bladder ◽  
Uroepithelial Cells ◽  
Activating Transcription Factor

Activating transcription factor 2 (ATF2) was involved in the expressions of proliferative factors in arsenite treated human urinary bladder epithelial cell line (SV-HUC-1 cells).

Turtle urinary bladder epithelial cell line

1989 ◽  
Vol 92 (1) ◽  
pp. 21-29
Author(s):  
Jose A.L. Arruda ◽  
Harry J. Lubansky ◽  
Kathy Thomasson
Keyword(s):  
Epithelial Cell ◽  
Urinary Bladder ◽  
Cell Line ◽  
Epithelial Cell Line ◽  
Bladder Epithelial Cell

scholarly journals Baicalin attenuates LPS-induced alveolar type II epithelial cell A549 injury by attenuation of the FSTL1 signaling pathway via increasing miR-200b-3p expression

2021 ◽  
pp. 175342592110138
Author(s):  
Xin-ya Duan ◽  
Yang Sun ◽  
Zhu-feng Zhao ◽  
Yao-qing Shi ◽  
Xun-yan Ma ◽  
...  
Keyword(s):  
Epithelial Cell ◽  
Cell Line ◽  
Alveolar Epithelial Cell ◽  
Inflammatory Factors ◽  
Epithelial Cell Line ◽  
Protective Mechanism ◽  
Alveolar Type ◽  
Dependent Manner ◽  
Type Ii ◽  
Alveolar Epithelial

In China, baicalin is the main active component of Scutellaria baicalensis, which has been used in the treatment of inflammation-related diseases, such as inflammation-induced acute lung injury. However, its specific mechanism remains unclear. This study examined the protective effect of baicalin on LPS-induced inflammation injury of alveolar epithelial cell line A549 and explored its protective mechanism. Compared with the LPS-induced group, the proliferation inhibition rates of alveolar type II epithelial cell line A549 intervened by different concentrations of baicalin decreased significantly, as did the levels of inflammatory factors IL-6, IL-1β, prostaglandin 2 and TNF-α in the supernatant. The expression levels of inflammatory proteins inducible NO synthase (iNOS), NF-κB65, phosphorylated ERK (p-ERK1/2), and phosphorylated c-Jun N-terminal kinase (p-JNK1) significantly decreased, as did the protein expression of follistatin-like protein 1 (FSTL1). In contrast, expression of miR-200b-3p significantly increased in a dose-dependent manner. These results suggested that baicalin could significantly inhibit the expression of inflammation-related proteins and improve LPS-induced inflammatory injury in alveolar type II epithelial cells. The mechanism may be related to the inhibition of ERK/JNK inflammatory pathway activation by increasing the expression of miR-200b-3p. Thus, FSTL1 is the regulatory target of miR-200b-3p.

An Established Epithelial Cell Line (NPC/HK1) from a Nasopharyngeal Carcinoma - II. A Sem Study

1982 ◽  
Vol 40 ◽  
pp. 224-225
Author(s):  
Li C.L. ◽  
Chew E.C. ◽  
Huang D.P. ◽  
Ho H.C. ◽  
Mak L.S. ◽  
...  
Keyword(s):  
Nasopharyngeal Carcinoma ◽  
Epithelial Cell ◽  
Surface Morphology ◽  
Cell Line ◽  
Epithelial Cell Line ◽  
Present Communication ◽  
Cells In Culture ◽  
Sem Study ◽  
Well Differentiated

An epithelial cell line, NPC/HK1, has recently been successfully established from a nasopharyngeal carcinoma of the moderately to well differentiated squamous type. The present communication reports on the surface morphology of the NPC/HK1 cells in culture.

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