scholarly journals Catheter Cryoablation of the Atrioventricular Node in Patients with Atrial Fibrillation: A Novel Technology for Ablation of Cardiac Arrhythmias

2001 ◽  
Vol 12 (4) ◽  
pp. 439-444 ◽  
Author(s):  
MARC DUBUC ◽  
PAUL KHAIRY ◽  
ANGEL RODRIGUEZ-SANTIAGO ◽  
MARIO TALAJIC ◽  
JEAN-CLAUDE TARDIF ◽  
...  
Keyword(s):  
Atrial Fibrillation ◽  
Cardiac Arrhythmias ◽  
Atrioventricular Node ◽  
Novel Technology

scholarly journals Cardiac Arrhythmias – Part III Narrow Complex Tachycardia

2005 ◽  
Vol 4 (2) ◽  
pp. 51-57
Author(s):  
DW Davies ◽  
◽  
MD O’Neill ◽  
Keyword(s):  
Atrial Fibrillation ◽  
Cardiac Arrhythmias ◽  
Sinus Tachycardia ◽  
Sinus Node ◽  
Atrioventricular Node ◽  
Supraventricular Arrhythmia ◽  
Common Causes ◽  
Atrioventricular Reciprocating Tachycardia ◽  
Narrow Complex Tachycardia ◽  
Wolff Parkinson White Syndrome

Narrow complex tachycardia usually refers to an abnormality of cardiac rhythm involving the tissues of the sinus node, atrial tissue, the atrioventricular node or an accessory atrioventricular communication. Although atrial fibrillation is the most common supraventricular arrhythmia, the term “supraventricular tachycardia” conventionally refers to the group of rhythm disturbances encompassing sinus tachycardia (appropriate and inappropriate), atrial tachycardia, atrial flutter, atrioventricular nodal reciprocating tachycardia (AVNRT) and atrioventricular reciprocating tachycardia (AVRT) including the Wolff Parkinson White syndrome (WPW). Atrial fibrillation is beyond the scope of this article which focuses on the diagnosis and acute management of the patient presenting with one of these common causes of a regular, narrow complex tachycardia.

scholarly journals 10-year trend analysis of atrioventricular node ablation in patient with atrial fibrillation: 2005–2014 United State hospitalization

2020 ◽  
Vol 41 (Supplement_2) ◽  
Author(s):  
L Wu ◽  
B Narasimhan ◽  
A.N Shah ◽  
Y.Y Zheng ◽  
K Bhatia ◽  
...  
Keyword(s):  
Atrial Fibrillation ◽  
Atrioventricular Node ◽  
Health Care Research ◽  
Progressive Increase ◽  
Pharmacological Therapy ◽  
Af Ablation ◽  
Diagnosis Codes ◽  
Increasing Trend ◽  
Quality Guideline ◽  
Pacemaker Insertion

Abstract Introduction Atrial fibrillation (AF) ablation and Atrioventricular Node (AVN) ablation are both important non-pharmacological therapy of AF. In spite of increased availability of AF ablation data, that of AVN ablation per se is limited. Method AF ablation was identified using ICD-9 procedure code with principle diagnosis of AF from United States National Inpatient Sample database 2005–2014. From procedure and diagnosis codes of pacemaker insertion followed by ablation, the cohort who underwent AVN ablation was identified. Patients hospitalization with any diagnosis of other type of arrythmia or epicardial ablation were excluded. Complications were defined as per the Agency for Health Care Research and Quality guideline. Results Total AF ablation was noted to increase from 2005- 2011, and declined steadily from 2011–2014. In contrast, the number of AVN ablations increased from 4505 cases to 5175 (Figure 1). AVN ablation were mainly performed in elderly patient (mean age 72), and increasingly in patient with higher Charlson Commobidity index (0.9 to 1.7)and higher CHA2DS2-VASc score (2.8 to 3.7) (Table 1). An increasing trend in procedure complications but no significant change in mortalitywere observed with AVN ablation. Progressive increase in the length of stay and the hospitalization cost were also observed over the years with AVN ablation. Conclusion AVN ablation is being performed at a steady volume, and increasingly in patients with multiple comorbidities. This trend although was not associated with increased mortality, it was associated with increased hospital complications. Funding Acknowledgement Type of funding source: None

Smartwatch-based detection of cardiac arrhythmias Beyond the differentiation between sinus rhythm and atrial fibrillation

Heart Rhythm ◽  
2021 ◽  
Author(s):  
Marc Strik ◽  
Sylvain Ploux ◽  
F Daniel Ramirez ◽  
Saer Abu-Alrub ◽  
Pierre Jais ◽  
...  
Keyword(s):  
Atrial Fibrillation ◽  
Sinus Rhythm ◽  
Cardiac Arrhythmias

Reentrant excitation as a cause of cardiac arrhythmias

1978 ◽  
Vol 235 (1) ◽  
pp. H1-H17 ◽  
Author(s):  
A. L. Wit ◽  
P. F. Cranefield
Keyword(s):  
Myocardial Infarction ◽  
Atrial Fibrillation ◽  
Experimental Evidence ◽  
Cardiac Arrhythmias ◽  
Fiber Bundles ◽  
Atrial Arrhythmias ◽  
Ventricular Muscle ◽  
Av Node ◽  
Reentrant Excitation ◽  
Longitudinal Dissociation

Mechanisms that cause reentry were defined in rings of tissue cut from jellyfish as early as 1906 by Mayer. The concepts were developed by Mines and Garrey during the next 10 years. Lewis then tried to demonstrate that reentry caused atrial flutter. Lewis, Garrey, and later Moe also proposed that atrial fibrillation was caused by reentry. Rosenblueth provided additional experimental evidence that reentry could cause atrial arrhythmias after crushing the intercaval bridge of atrial muscle. Recent studies by Allessie using microelectrodes have provided detailed evidence for reentry in atrial tissue. Mines in 1913 also proposed that reentry could occur in the AV node. Scherf then introduced the concept of functional longitudinal dissociation as a cause of return extrasystoles and this was later shown to happen in the node by Moe and his colleagues. Reentry can also occur between atria and ventricles utilizing accessory connecting pathways. Schmitt and Erlanger in 1913 were the first to do experiments which indicated that reentry can also occur in the ventricles. Subsequently it was shown that reentry can occur in Purkinje fiber bundles. Reentry in ventricular muscle may also cause some of the arrhythmias that occur after myocardial infarction.

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