scholarly journals WWOX, the common fragile site FRA16D gene product, regulates ATM activation and the DNA damage response

2014 ◽  
Vol 111 (44) ◽  
pp. E4716-E4725 ◽  
Author(s):  
M. Abu-Odeh ◽  
Z. Salah ◽  
C. Herbel ◽  
T. G. Hofmann ◽  
R. I. Aqeilan
Keyword(s):  
Dna Damage ◽  
Gene Product ◽  
Dna Damage Response ◽  
Fragile Site ◽  
Common Fragile Site ◽  
Damage Response ◽  
The Common

Functional link of BRCA1 and ataxia telangiectasia gene product in DNA damage response

Nature ◽  
2000 ◽  
Vol 406 (6792) ◽  
pp. 210-215 ◽  
Author(s):  
Shang Li ◽  
Nicholas S. Y. Ting ◽  
Lei Zheng ◽  
Phang-Lang Chen ◽  
Yael Ziv ◽  
...  
Keyword(s):  
Dna Damage ◽  
Gene Product ◽  
Dna Damage Response ◽  
Ataxia Telangiectasia ◽  
Functional Link ◽  
Damage Response

Role of Retinoblastoma Gene Product in p53-Mediated DNA Damage Response

1994 ◽  
Vol 215 (2) ◽  
pp. 386-389 ◽  
Author(s):  
Martin L. Smith ◽  
Qimin Zhan ◽  
Insoo Bae ◽  
Albert J. Fornace
Keyword(s):  
Dna Damage ◽  
Gene Product ◽  
Dna Damage Response ◽  
Retinoblastoma Gene ◽  
Damage Response ◽  
Retinoblastoma Gene Product

scholarly journals Mitotic and DNA Damage Response Proteins: Maintaining the Genome Stability and Working for the Common Good

2021 ◽  
Vol 9 ◽  
Author(s):  
Fernando Luna-Maldonado ◽  
Marco A. Andonegui-Elguera ◽  
José Díaz-Chávez ◽  
Luis A. Herrera
Keyword(s):  
Dna Damage ◽  
Dna Damage Response ◽  
Chromosome Segregation ◽  
Common Good ◽  
Genome Stability ◽  
Spindle Assembly Checkpoint ◽  
Cellular Mechanisms ◽  
Damage Response ◽  
The Common ◽  
Maintain Genome Stability

Cellular function is highly dependent on genomic stability, which is mainly ensured by two cellular mechanisms: the DNA damage response (DDR) and the Spindle Assembly Checkpoint (SAC). The former provides the repair of damaged DNA, and the latter ensures correct chromosome segregation. This review focuses on recently emerging data indicating that the SAC and the DDR proteins function together throughout the cell cycle, suggesting crosstalk between both checkpoints to maintain genome stability.

2118-P: Regulation of Ataxia-Telangiectasia and Rad3-Related (ATR)–Dependent DNA Damage Response by Nitric Oxide in ß-Cells

Diabetes ◽  
2020 ◽  
Vol 69 (Supplement 1) ◽  
pp. 2118-P
Author(s):  
CHAY TENG YEO ◽  
BRYNDON OLESON ◽  
JOHN A. CORBETT ◽  
JAMIE K. SCHNUCK
Keyword(s):  
Nitric Oxide ◽  
Dna Damage ◽  
Dna Damage Response ◽  
Ataxia Telangiectasia ◽  
Damage Response
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