scholarly journals SP351THE 'SUBLIME' APPROACH: COST-EFFICACY OF A NOVEL SELF-MANAGEMENT APPROACH FOR DIETARY SODIUM RESTRICTION IN CKD PATIENTS

2016 ◽  
Vol 31 (suppl_1) ◽  
pp. i206-i207 ◽  
Author(s):  
J. K. Humalda ◽  
G. Klaassen ◽  
H. De Vries ◽  
Y. Meuleman ◽  
G. D. Laverman ◽  
...  
Keyword(s):  
Dietary Sodium ◽  
Self Management ◽  
Management Approach ◽  
Sodium Restriction ◽  
Cost Efficacy ◽  
Dietary Sodium Restriction

scholarly journals A Self-management Approach for Dietary Sodium Restriction in Patients With CKD: A Randomized Controlled Trial

2020 ◽  
Vol 75 (6) ◽  
pp. 847-856 ◽  
Author(s):  
Jelmer K. Humalda ◽  
Gerald Klaassen ◽  
Hanne de Vries ◽  
Yvette Meuleman ◽  
Lara C. Verschuur ◽  
...  
Keyword(s):  
Randomized Controlled Trial ◽  
Controlled Trial ◽  
Dietary Sodium ◽  
Self Management ◽  
Management Approach ◽  
Sodium Restriction ◽  
Randomized Controlled ◽  
Dietary Sodium Restriction

scholarly journals The Effects of Short-Term Changes in Sodium Intake on Plasma Marinobufagenin Levels in Patients with Primary Salt-Sensitive and Salt-Insensitive Hypertension

Nutrients ◽  
2021 ◽  
Vol 13 (5) ◽  
pp. 1502
Author(s):  
Katarzyna Łabno-Kirszniok ◽  
Agata Kujawa-Szewieczek ◽  
Andrzej Wiecek ◽  
Grzegorz Piecha
Keyword(s):  
Blood Pressure ◽  
Diastolic Blood Pressure ◽  
Sodium Intake ◽  
Left Ventricular ◽  
Primary Hypertension ◽  
Dietary Sodium ◽  
Sodium Restriction ◽  
Short Term ◽  
Salt Loading ◽  
Dietary Sodium Restriction

Increased marinobufagenin (MBG) synthesis has been suggested in response to high dietary salt intake. The aim of this study was to determine the effects of short-term changes in sodium intake on plasma MBG levels in patients with primary salt-sensitive and salt-insensitive hypertension. In total, 51 patients with primary hypertension were evaluated during acute sodium restriction and sodium loading. Plasma or serum concentrations of MBG, natriuretic pro-peptides, aldosterone, sodium, potassium, as well as hematocrit (Hct) value, plasma renin activity (PRA) and urinary sodium and potassium excretion were measured. Ambulatory blood pressure monitoring (ABPM) and echocardiography were performed at baseline. In salt-sensitive patients with primary hypertension plasma MBG correlated positively with diastolic blood pressure (ABPM) and serum NT-proANP concentration at baseline and with serum NT-proANP concentration after dietary sodium restriction. In this subgroup plasma MBG concentration decreased during sodium restriction, and a parallel increase of PRA was observed. Acute salt loading further decreased plasma MBG concentration in salt-sensitive subjects in contrast to salt insensitive patients. No correlation was found between plasma MBG concentration and left ventricular mass index. In conclusion, in salt-sensitive hypertensive patients plasma MBG concentration correlates with 24-h diastolic blood pressure and dietary sodium restriction reduces plasma MBG levels. Decreased MBG secretion in response to acute salt loading may play an important role in the pathogenesis of salt sensitivity.

Effect of dietary sodium restriction on body water, blood pressure, and inflammation in hemodialysis patients: a prospective randomized controlled study

2013 ◽  
Vol 46 (1) ◽  
pp. 91-97 ◽  
Author(s):  
Lidiane Silva Rodrigues Telini ◽  
Gabriela de Carvalho Beduschi ◽  
Jacqueline Costa Teixeira Caramori ◽  
João Henrique Castro ◽  
Luis Cuadrado Martin ◽  
...  
Keyword(s):  
Blood Pressure ◽  
Hemodialysis Patients ◽  
Body Water ◽  
Dietary Sodium ◽  
Randomized Controlled Study ◽  
Controlled Study ◽  
Sodium Restriction ◽  
Randomized Controlled ◽  
Dietary Sodium Restriction

Changes in the circadian rhythm of blood pressure in primary aldosteronism in response to dietary sodium restriction and adrenalectomy

1998 ◽  
Vol 16 (12) ◽  
pp. 1745-1748 ◽  
Author(s):  
Takashi Uzu ◽  
Masataka Nishimura ◽  
Takashi Fujii ◽  
Masanobu Takeji ◽  
Setsuko Kuroda ◽  
...  
Keyword(s):  
Blood Pressure ◽  
Circadian Rhythm ◽  
Primary Aldosteronism ◽  
Dietary Sodium ◽  
Sodium Restriction ◽  
Dietary Sodium Restriction

Dietary sodium restriction and development of hypertension in spontaneously hypertensive rats

1983 ◽  
Vol 245 (6) ◽  
pp. H1081-H1084 ◽  
Author(s):  
C. B. Toal ◽  
F. H. Leenen
Keyword(s):  
Blood Pressure ◽  
Control Diet ◽  
Dietary Sodium ◽  
Sodium Restriction ◽  
Spontaneously Hypertensive ◽  
Basal Blood Pressure ◽  
Blood Pressures ◽  
Dietary Sodium Restriction ◽  
Wistar Kyoto ◽  
And Control

Blood pressure and body weight of conscious spontaneously hypertensive (SHR) and Wistar-Kyoto (WKY) rats were measured up to 16 wk of age in animals started at birth on five different sodium-containing diets. SHR on 9 mumol sodium/g food did not show a rise in basal blood pressure; however, when stressed the SHR still exhibited slightly higher blood pressures than WKY. In SHR on 17 mumol sodium/g food the development of hypertension was blunted compared with that of control (101 mumol/g) diet animals. SHR on 26 or 44 mumol sodium/g diet exhibited a development of hypertension similar to that of SHR on control diet. The 26 mumol/g, 44 mumol/g, and control sodium diet groups, regardless of strain, had similar growth rates. By contrast, on 17 mumol sodium/g food both SHR and WKY showed a substantially reduced growth rate, and all animals on 9 mumol sodium/g diet were severely retarded in growth. The results indicate that dietary sodium restriction can ameliorate the development of hypertension in SHR, but only when the sodium levels are so low as to affect overall growth.

Further evidence for hepatic control of salt intake in rats

1987 ◽  
Vol 253 (3) ◽  
pp. R444-R449 ◽  
Author(s):  
M. G. Tordoff ◽  
J. Schulkin ◽  
M. I. Friedman
Keyword(s):  
Portal Vein ◽  
Salt Intake ◽  
Dietary Sodium ◽  
Sodium Restriction ◽  
Hepatic Portal Vein ◽  
Dietary Sodium Restriction ◽  
Reduced Salt ◽  
Hepatic Portal

We examined the contributions of oral and hepatic factors to the control of salt intake in rats made "salt hungry" by combined dietary sodium restriction and furosemide treatment. A 7.5-ml preload of 0.5 M NaCl, which was either voluntarily ingested or slowly infused (7.5 ml/90 min) into the hepatic portal vein, decreased subsequent 0.5 M NaCl intake by approximately 7.5 ml in both cases. Hepatic portal infusions were probably acting in the liver because slow jugular infusions (7.5 ml/90 min) did not influence salt drinking, although rapid (7.5 ml/5 min) infusions by both routes reduced salt intake. If salt was ingested but drained through a gastric cannula, rats showed little sign of reducing salt in a 2-h test. These results suggest that the quantity of salt ingested by salt-hungry rats is regulated by a postabsorptive mechanism that is probably located in the liver.

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