scholarly journals Internal and external radiative widths in the combined R -matrix and potential-model formalism

2017 ◽  
Vol 95 (2) ◽  
Author(s):  
A. M. Mukhamedzhanov ◽  
Shubhchintak ◽  
C. A. Bertulani ◽  
T. V. Nhan Hao
Author(s):  
Abdul Kabir ◽  
Jameel-Un Nabi

Abstract Radiative capture p+9Be → 10B+γ at energies bearing astrophysical importance is a key process for the spectroscopic study of 10B. In this work, we consider the radiative capture cross-section for the 9Be(p, γ)10B within the framework of the potential model and the R-matrix method for the multi-entrance channel cases. In certain cases, when the potential fails, therefore, the R-matrix approach is better to use for the description of partial components of the cross-section that have sharp or broad resonances. For all possible electric and magnetic dipole transitions, partial components of the astrophysical S-factor are computed. The computed value of the total S-factor at zero energy is consistent with the reported results.


2008 ◽  
Vol 17 (10) ◽  
pp. 2165-2170
Author(s):  
PIERRE DESCOUVEMONT

General properties of low-energy cross sections and of reaction rates are presented. We describe different models used in nuclear astrophysics: microscopic models, the potential model, and the R-matrix method. Two important reactions, 7 Be ( p ,γ)8 B and 12 C (α,γ)16 O , are then briefly discussed.


Author(s):  
Nguyen Hoang Phuc ◽  
Nguyen Tri Toan Phuc ◽  
Do Cong Cuong

We apply the Lagrange-mesh [Formula: see text]-matrix method to calculate the [Formula: see text]-factor for the [Formula: see text]C[Formula: see text]N and [Formula: see text]O[Formula: see text]F direct radiative capture reactions. By comparing the astrophysical [Formula: see text]-factors calculated with nonlocal and local potentials, we investigate the nonlocality effects coming from the nuclear potentials in the direct capture reactions. Our calculations are in good agreement with the experimental data and indicate a nonnegligible difference in the results of local and nonlocal potentials. The use of small diffuseness narrow potentials also provides a remarkably good fit in the case with multiple broad resonances. Our findings suggest that the nonlocal potential improves the calculated results although the difference between the local and nonlocal potentials is smaller than uncertainties from other sources. We propose the nonlocality potential should be used in the potential model calculation of future astrophysics rates evaluation.


2009 ◽  
Vol 47 (09) ◽  
Author(s):  
K Hochrath ◽  
B Rathkolb ◽  
K Butuzova ◽  
W Hans ◽  
H Fuchs ◽  
...  

2008 ◽  
Vol 31 (4) ◽  
pp. 5
Author(s):  
Brett Burstein ◽  
Kunihiro Nishida ◽  
Philippe Comtois ◽  
Louis Villenuve ◽  
Yung-Hsin Yeh ◽  
...  

Background: Connexin alterations occur in various atrial fibrillation (AF) paradigms, but their functional significance remains unclear. No data are available regarding the effects of CHF on atrial connexin expression and phosphorylation. We therefore analyzed connexin changes and their contribution to the AF substrate during the development and reversal ofCHF. Methods and Results: Dogs were allocated to three groups: CHF induced by 2-week ventricular tachypacing (CHF, n=15); CHF dogs allowed to recover for 4 weeks after 2-week tachypacing (REC, n=15) and non-paced shams (CTL, n=11). Left ventricular end-diastolic pressure increased with CHF (14.5±1.0*** vs.3.7±0.7, ***P < 0.001 vs. CTL) and normalized upon CHF recovery (5.1±1.0^†††, ^††† P < 0.001 vs. CHF). Real-time PCR and Western-blot analyses revealed connexin43 (Cx43) and connexin40 (Cx40) mRNA and protein expression to be unchanged by CHF and REC. However, CHF caused Cx43 dephosphorylation(by ~73%***) and increased Cx40/Cx43 protein ratio (by ~35%***), with both alterations completely reversing in REC. Immunofluorescent confocal microscopy confirmed connexin protein trends, with a reduction in phosphorylated Cx43 (by ~68%*** in CHF) that returned to control in REC. CHF caused conduction abnormalities (phasedelay-range and heterogeneity index, both P < 0.01) and burst pacing-induced AF prolongation (CTL 22±7s, CHF 1100±171s***, REC 884±220s***) which persisted in the recovery period, along with residual fibrosis (CTL 3.6±0.7%, CHF 14.7±1.5%***, REC13.3±2.3%***). Fibrosis physically interrupted muscle bundle continuity and anionically-based action potential model of canine atrium showed that fibrosiswas able to account for the observed conduction abnormalities. Conclusions: CHF causes connexin-dephosphorylation and Cx40/Cx43ratio increases. With CHF reversal, atrial connexin alterations recover completely, but tissue fibrosis, conduction abnormalities and a substrate forAF remain with fibrosis accounting for conduction abnormalities. Thus, althougha trial connexin changes occur with CHF, they are not essential for conduction disturbances and AF promotion, which appear rather to be related primarily tofibrotic interruption of muscle-bundle continuity.


Author(s):  
The Anh Nguyen ◽  
Frank Thomas Seifried

Sign in / Sign up

Export Citation Format

Share Document