Chlorogenic Acid Oxidation and Its Reaction with Sunflower Proteins to Form Green-Colored Complexes

Tobacco mosaic virus (TMV) infection has been studied in several investigations of Nicotiana tabacum leaf tissue. Earlier studies have suggested that TMV infection does not have precise infective selectivity vs. specific types of tissues. Also, such tissue conditions as vein banding, vein clearing, liquification and suberization may result from causes other than direct TMV infection. At the present time, it is thought that the plasmodesmata, ectodesmata and perhaps the plasmodesmata of the basal septum may represent the actual or more precise sites of TMV infection.TMV infection has been implicated in elevated levels of oxidative metabolism; also, TMV infection may have a major role in host resistance vs. concentration levels of phenolic-type enzymes. Therefore, enzymes such as polyphenol oxidase, peroxidase and phenylalamine ammonia-lyase may show an increase in activity in response to TMV infection. It has been reported that TMV infection may cause a decrease in o-dihydric phenols (chlorogenic acid) in some tissues.

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Peroxisome proliferation: Organelles or activity as an endpoint?

Proceedings, annual meeting, Electron Microscopy Society of America ◽

10.1017/s0424820100147818 ◽

1993 ◽

Vol 51 ◽

pp. 396-397

Author(s):

Catherine A. Taylor ◽

Bruce M. Jarnot

Keyword(s):

Acid Oxidation ◽

Peroxisome Proliferation ◽

Post Dose ◽

Oxidation Activity ◽

Wasting Syndrome ◽

Perfluorodecanoic Acid ◽

Hypolipidemic Agent ◽

Commercially Important ◽

Acyl Coa Oxidase ◽

Mitochondrial Disruption

Peroxisome induction can be expressed as an increase in peroxisome area (proliferation) or as an increase in peroxisomal fatty acid oxidation (activity). This study compares proliferation and activity as endpoints for hepatic peroxisome induction by perfluorodecanoic acid (PFDA). Fluorocarboxylic acids such as PFDA represent a class of compounds possessing commercially important surfactant properties. A single 50 mg/Kg ip. dose of PFDA produces a characteristic “wasting syndrome” in male F-344 rats. Symptoms include hypophagia, weight loss, hepatomegaly, and delayed lethality. Hepatic studies reveal changes similar to those seen with the hypolipidemic agent clofibrate. These include mitochondrial disruption, endoplasmic reticulum and peroxisome proliferation, and increased peroxisomal acyl-CoA oxidase activity.Male Fisher-344 rats received a single ip. dose of 2, 20, or 50mg/Kg PFDA dissolved in 1:1 propylene glycol/water and were sacrificed 8 days post-dose. All control rats received an equal volume of vehicle ip. Animals were provided food and water ad libitum, except pair-fed controls which received the same restrictive food intake consumed by their weight-paired dosed partners (50mg/Kg PFDA group) to simulate the hypophagia associated with PFDA.

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Antioxidant activity of some Scorzonera species and quantitative analysis of chlorogenic acid

Planta Medica ◽

10.1055/s-0030-1264437 ◽

2010 ◽

Vol 76 (12) ◽

Cited By ~ 1

Author(s):

O Bahadir ◽

G Saltan Citoglu ◽

T Coban

Keyword(s):

Antioxidant Activity ◽

Quantitative Analysis ◽

Chlorogenic Acid

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