Up‐regulation of cofilin‐1 in cell senescence associates with morphological change and p27 kip1 ‐mediated growth delay

10.1111/acel.13288 ◽

2020 ◽

Author(s):

Cheng‐Han Tsai ◽

Chun‐Yuan Chang ◽

Bing‐Ze Lin ◽

Yu‐Lou Wu ◽

Meng‐Hsiu Wu ◽

...

Keyword(s):

Morphological Change ◽

Cell Senescence ◽

Growth Delay ◽

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Faculty Opinions recommendation of Degradation of p27(Kip1) at the G(0)-G(1) transition mediated by a Skp2-independent ubiquitination pathway.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1003339.40606 ◽

2002 ◽

Author(s):

Michele Pagano

Keyword(s):

Ubiquitination Pathway ◽

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Faculty Opinions recommendation of Degradation of p27(Kip1) at the G(0)-G(1) transition mediated by a Skp2-independent ubiquitination pathway.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1003339.30657 ◽

2001 ◽

Author(s):

Hiroto Okayama

Keyword(s):

Ubiquitination Pathway ◽

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Faculty Opinions recommendation of 14-3-3 suppresses the nuclear localization of threonine 157-phosphorylated p27(Kip1).

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1019191.216499 ◽

2004 ◽

Author(s):

Michael B Yaffe

Keyword(s):

Nuclear Localization ◽

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Faculty Opinions recommendation of Effect of p27(KIP1) on cell cycle and apoptosis in gastric cancer cells.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1031750.367857 ◽

2006 ◽

Author(s):

Esther Bullitt

Keyword(s):

Gastric Cancer ◽

Cancer Cells ◽

Gastric Cancer Cells ◽

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Faculty Opinions recommendation of Downregulation of Wnt signaling is a trigger for formation of facultative heterochromatin and onset of cell senescence in primary human cells.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1089432.544670 ◽

2007 ◽

Author(s):

Peter Scambler

Keyword(s):

Wnt Signaling ◽

Human Cells ◽

Cell Senescence ◽

Facultative Heterochromatin ◽

Primary Human Cells

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Faculty Opinions recommendation of Argyrin a reveals a critical role for the tumor suppressor protein p27(kip1) in mediating antitumor activities in response to proteasome inhibition.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1115518.571555 ◽

2008 ◽

Author(s):

Michele Pagano

Keyword(s):

Tumor Suppressor ◽

Critical Role ◽

Proteasome Inhibition ◽

Tumor Suppressor Protein ◽

Antitumor Activities ◽

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Faculty Opinions recommendation of Pulmonary artery smooth muscle cell senescence is a pathogenic mechanism for pulmonary hypertension in chronic lung disease.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.12312957.13487055 ◽

2011 ◽

Author(s):

Paul Corris

Keyword(s):

Pulmonary Hypertension ◽

Smooth Muscle ◽

Pulmonary Artery ◽

Smooth Muscle Cell ◽

Lung Disease ◽

Muscle Cell ◽

Chronic Lung Disease ◽

Cell Senescence ◽

Pathogenic Mechanism ◽

Pulmonary Artery Smooth Muscle

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Faculty Opinions recommendation of Inhibition of IGF-1R prevents ionizing radiation-induced primary endothelial cell senescence.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.718169014.793488998 ◽

2013 ◽

Author(s):

John Lazo

Keyword(s):

Endothelial Cell ◽

Ionizing Radiation ◽

Cell Senescence ◽

Radiation Induced

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Faculty Opinions recommendation of Maternal care in Acanthosomatinae (Insecta: Heteroptera: Acanthosomatidae)--correlated evolution with morphological change.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.725950239.793511996 ◽

2015 ◽

Author(s):

Joël Meunier

Keyword(s):

Morphological Change ◽

Maternal Care ◽

Correlated Evolution

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Faculty Opinions recommendation of Evaluating the role of p38 MAPK in the accelerated cell senescence of werner syndrome fibroblasts.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.731249479.793537088 ◽

2017 ◽

Author(s):

Vilhelm Bohr

Keyword(s):

P38 Mapk ◽

Werner Syndrome ◽

Cell Senescence

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