Mast cells after capsaicin application in normal human skin

1990 ◽  
Vol 123 (s37) ◽  
pp. 119-120
Author(s):  
C.B. Bunker ◽  
R. Cerio ◽  
H.A. Bull ◽  
J. Evans ◽  
J.C. Foreman ◽  
...  
1991 ◽  
Vol 33 (1-2) ◽  
pp. 195-196 ◽  
Author(s):  
C. B. Bunker ◽  
R. Cerio ◽  
H. A. Bull ◽  
J. Evans ◽  
P. M. Dowd ◽  
...  

1997 ◽  
Vol 289 (5) ◽  
pp. 256-260 ◽  
Author(s):  
T. E. Damsgaard ◽  
Anne Braae Olesen ◽  
Flemming Brandt Sørensen ◽  
Kristian Thestrup-Pedersen ◽  
Peter Oluf Schiøtz

1994 ◽  
Vol 286 (7) ◽  
pp. 380-385 ◽  
Author(s):  
K. Hamann ◽  
J. Grabbe ◽  
P. Welker ◽  
N. Haas ◽  
B. Algermissen ◽  
...  

1984 ◽  
Vol 12 (2) ◽  
pp. 89-97
Author(s):  
Graham R. Elliott ◽  
H.E. Amos ◽  
James W. Bridges

The rate of growth of normal human skin fibroblasts was inhibited in a dose related, reversible, fashion by practolol (N-4-(2-hydroxy)-3 (1-methyl)-aminopropoxyphenylacetamine) (ID50 1.35 ± 0.14 x 10-3M), propranolol (1-(isopropylamino)-3(1-naphthyl-oxy)-2-propranolol) (ID50 0.145 ± 0.02 x 10-3M) and paracetamol (N-(4-hydroxyphenyl) acetamide) (ID50 0.85 ± 0.2 x 10-3M). Skin fibroblasts isolated from a psoriasis patient were more sensitive towards practolol (ID50 0.48 ± 0.14 x 10-3M) and propranolol (ID50 0.032 ± 0.002 x 10-3M), but less sensitive towards paracetamol (ID50 1.3 ± 0.07 x 10-3M). In vitro generated metabolites of practolol, using normal or Arochlor 1254-pretreated hamster liver preparations, and structural analogues of practolol had no effect upon the growth of either cell type.


1987 ◽  
Vol 117 (4) ◽  
pp. 419-428 ◽  
Author(s):  
C.R. LOVELL ◽  
K.A. SMOLENSKI ◽  
V.C. DUANCE ◽  
N.D. LIGHT ◽  
S. YOUNG ◽  
...  

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