Influence of Successive Badminton Matches on Muscle Strength, Power, and Body-Fluid Balance in Elite Players

2014 ◽  
Vol 9 (4) ◽  
pp. 689-694 ◽  
Author(s):  
Javier Abian-Vicen ◽  
Adrián Castanedo ◽  
Pablo Abian ◽  
Cristina Gonzalez-Millan ◽  
Juan José Salinero ◽  
...  

The aim was to analyze the influence of competitive round on muscle strength, body-fluid balance, and renal function in elite badminton players during a real competition. Body mass, jump height during a countermovement jump, handgrip force, and urine samples were obtained from 13 elite badminton players (6 men and 7 women) before and after the 2nd-round and quarterfinal matches of the national Spanish badminton championship. Sweat rate was determined by using prematch-to-postmatch body-mass change and by weighing individually labeled fluid bottles. Sweat rates were 1.04 ± 0.62 and 0.98 ± 0.43 L/h, while rehydration rate was 0.69 ± 0.26 and 0.91 ± 0.52 L/h for the 2nd round and quarterfinals, respectively. Thus, dehydration was 0.47% ± 1.03% after the 2nd round and 0.23% ± 0.43% after the quarterfinals. There were no differences in prematch-to-postmatch jump height, but jump height was reduced from 37.51 ± 8.83 cm after the 2nd-round game to 34.82 ± 7.37 cm after the quarterfinals (P < .05). No significant differences were found in handgrip force when comparing prepost matches or rounds, although there were significant differences between dominant and nondominant hands (P < .05). The succession of rounds caused the appearance of proteinuria, hematuria, glycosuria, and higher nitrite and ketone concentrations in urine. Rehydration patterns during a real badminton competition were effective to prevent dehydration. A badminton match did not affect jump height or handgrip force, but jump height was progressively reduced by the competitive round. Badminton players’ renal responses reflected diminished renal flux due to the high-intensity nature of this racket sport.

1990 ◽  
Vol 68 (7) ◽  
pp. 889-892 ◽  
Author(s):  
Bareket Falk ◽  
Ruth Burstein ◽  
Josef Rosenblum ◽  
Yair Shapiro ◽  
E. Zylber-Katz ◽  
...  

This study investigated the effects of caffeine supplementation on thermoregulation and body fluid balance during prolonged exercise in a thermoneutral environment (25 °C, 50% RH). Seven trained male subjects exercised on a treadmill at an intensity of 70–75% of maximal oxygen consumption to self-determined exhaustion. Subjects exercised once after caffeine and once after placebo ingestion, given in a double-blind crossover design. Five milligrams per kilogram body weight of caffeine followed by 2.5 mg∙kg−1 of caffeine were given 2 and 0.5 h before exercise, respectively. Rectal temperature was recorded and venous blood samples were withdrawn every 15 min. Water loss and sweat rate were calculated from the difference between pre- and post-exercise body weight, corrected for liquid intake. Following caffeine ingestion, when compared with placebo, no significant difference in final rectal temperature or in percent change in plasma volume were found. No significant differences were observed in total water loss (1376 ± 154 vs. 1141 ± 158 mL, respectively), sweat rate (12.4 ± 1.1 vs. 10.9 ± 0.7 g∙m−2 min−1, respectively), rise in rectal temperature (2.1 ± 0.3 vs. 1.5 ± 0.4 °C, respectively), nor in the calculated rate of heat storage during exercise (134.4 ± 17.7 vs. 93.5 ± 22.5 W, respectively). Thus, in spite of the expected rise in oxygen uptake, caffeine ingestion under the conditions of this study does not seem to disturb body fluid balance or affect thermoregulation during exercise performance.Key words: caffeine, physical exercise, thermoregulation, fluid balance, dehydration.


2011 ◽  
Vol 1383 ◽  
pp. 179-186 ◽  
Author(s):  
Rie Asami ◽  
Kentaro Ono ◽  
Osamu Nakanishi ◽  
Kiyotoshi Inenaga

Nutrients ◽  
2020 ◽  
Vol 12 (5) ◽  
pp. 1276 ◽  
Author(s):  
Colleen X. Muñoz ◽  
Evan C. Johnson ◽  
Laura J. Kunces ◽  
Amy L. McKenzie ◽  
Michael Wininger ◽  
...  

We investigated the impact of nutrient intake on hydration biomarkers in cyclists before and after a 161 km ride, including one hour after a 650 mL water bolus consumed post-ride. To control for multicollinearity, we chose a clustering-based, machine learning statistical approach. Five hydration biomarkers (urine color, urine specific gravity, plasma osmolality, plasma copeptin, and body mass change) were configured as raw- and percent change. Linear regressions were used to test for associations between hydration markers and eight predictor terms derived from 19 nutrients merged into a reduced-dimensionality dataset through serial k-means clustering. Most predictor groups showed significant association with at least one hydration biomarker: (1) Glycemic Load + Carbohydrates + Sodium, (2) Protein + Fat + Zinc, (3) Magnesium + Calcium, (4) Pinitol, (5) Caffeine, (6) Fiber + Betaine, and (7) Water; potassium + three polyols, and mannitol + sorbitol showed no significant associations with any hydration biomarker. All five hydration biomarkers were associated with at least one nutrient predictor in at least one configuration. We conclude that in a real-life scenario, some nutrients may serve as mediators of body water, and urine-specific hydration biomarkers may be more responsive to nutrient intake than measures derived from plasma or body mass.


2001 ◽  
Vol 281 (5) ◽  
pp. R1637-R1646 ◽  
Author(s):  
D. J. Sly ◽  
M. J. McKinley ◽  
B. J. Oldfield

This study was undertaken to determine if neurons in the lamina terminalis, previously identified as projecting to the kidney (35), were responsive to alterations in stimuli associated with fluid balance homeostasis. Neurons in the lamina terminalis projecting to the kidney were identified by the retrograde transynaptic transport of Bartha's strain of pseudorabies virus in anesthetized rats. Rats were also exposed to 24-h water deprivation, intravenous hypertonic saline, or intracerebroventricular ANG II. To determine if “kidney-directed” neurons were activated following each stimulus, brain sections that included the lamina terminalis were examined immunohistochemically for viral antigen and Fos protein. With the exception of ANG II in the subfornical organ, all regions of the lamina terminalis contained neurons that were significantly activated by water deprivation, hypertonic saline, and ANG II. These results provide evidence for a neural substrate, which may underpin some of the effects of hypertonic saline and ANG II on renal function thought to be mediated through the lamina terminalis.


2005 ◽  
Vol 37 (Supplement) ◽  
pp. S104???S105
Author(s):  
Simone D. Henkin ◽  
M??rcio Martins Silveira ◽  
Henrique Lannerhirt ◽  
Fl??via Meyer ◽  
Luis Fernando Martins Kruel

Author(s):  
Karen Soo ◽  
Geraldine Naughton

This study investigated the hydration profile of high-performance female cricket players competing at a national tournament. The profile comprised hydration monitoring (n = 18) and a questionnaire (n = 20). Our objectives were to 1) advance the understanding of fluid losses in cricket sessions across a tournament and 2) assess the hydration knowledge and practices in female cricket players. Body mass before and after each game inning was recorded in order to estimate sweat rate, sweat loss, and percentage body-mass loss. Comparisons were made between groups categorized according to level of activity during each inning. When sweat rates were estimated according to actual activity time, results were in the range of those in other female team sports but less than results from male cricket players. A range of knowledge of hydration issues was also observed. This study supports the need for individualized hydration recommendations and provides direction for further hydration education in women’s cricket.


1994 ◽  
Vol 55 (1) ◽  
pp. 163-167 ◽  
Author(s):  
Tetsuya Yoshida ◽  
Tadashi Okuno ◽  
Takashi Kawabata ◽  
Taketoshi Morimoto

2011 ◽  
Vol 21 (6) ◽  
pp. 492-500 ◽  
Author(s):  
Matthew J.E. Lott ◽  
Stuart D.R. Galloway

This study assessed fluid balance, sodium losses, and effort intensity during indoor tennis match play (17 ± 2 °C, 42% ± 9% relative humidity) over a mean match duration of 68.1 ± 12.8 min in 16 male tennis players. Ad libitum fluid intake was recorded throughout the match. Sweat loss from change in nude body mass; sweat electrolyte content from patches applied to the forearm, calf, and thigh, and back of each player; and electrolyte balance derived from sweat, urine, and daily food-intake analysis were measured. Effort intensity was assessed from on-court heart rate compared with data obtained during a maximal treadmill test. Sweat rate (M ± SD) was 1.1 ± 0.4 L/hr, and fluid-ingestion rate was 1.0 ± 0.6 L/hr (replacing 93% ± 47% of fluid lost), resulting in only a small mean loss in body mass of 0.15% ± 0.74%. Large interindividual variabilities in sweat rate (range 0.3–2.0 L/hr) and fluid intake (range 0.31–2.52 L/hr) were noted. Whole-body sweat sodium concentration was 38 ± 12 mmol/L, and total sodium losses during match play were 1.1 ± 0.4 g (range 0.5–1.8 g). Daily sodium intake was 2.8 ± 1.1 g. Indoor match play largely consisted of low-intensity exercise below ventilatory threshold (mean match heart rate was 138 ± 24 beats/min). This study shows that in moderate indoor temperature conditions players ingest sufficient fluid to replace sweat losses. However, the wide range in data obtained highlights the need for individualized fluid-replacement guidance.


1996 ◽  
Vol 28 (Supplement) ◽  
pp. 157
Author(s):  
R. Soler ◽  
M. A. Rivera ◽  
A. Rivera-Brown ◽  
W. R. Frontera ◽  
E. Lopategui

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