scholarly journals Effects of ischemic preconditioning on short-duration cycling performance

2016 ◽  
Vol 41 (8) ◽  
pp. 825-831 ◽  
Author(s):  
Rogério Santos de Oliveira Cruz ◽  
Rafael Alves de Aguiar ◽  
Tiago Turnes ◽  
Amadeo Félix Salvador ◽  
Fabrizio Caputo
Keyword(s):  
Skeletal Muscle ◽  
Blood Lactate ◽  
Ischemic Preconditioning ◽  
Power Output ◽  
Muscle Activation ◽  
Endurance Performance ◽  
Cycling Performance ◽  
Oxygen Deficit ◽  
Mean Power ◽  
Lactate Kinetics

It has been demonstrated that ischemic preconditioning (IPC) improves endurance performance. However, the potential benefits during anaerobic events and the mechanism(s) underlying these benefits remain unclear. Fifteen recreational cyclists were assessed to evaluate the effects of IPC of the upper thighs on anaerobic performance, skeletal muscle activation, and metabolic responses during a 60-s sprint performance. After an incremental test and a familiarization visit, subjects were randomly submitted in visits 3 and 4 to a performance protocol preceded by intermittent bilateral cuff inflation (4 × (5 min of blood flow restriction + 5 min reperfusion)) at either 220 mm Hg (IPC) or 20 mm Hg (control). To increase data reliability, each intervention was replicated, which was also in a random manner. In addition to the mean power output, the pulmonary oxygen uptake, blood lactate kinetics, and quadriceps electromyograms (EMGs) were analyzed during performance and throughout 45 min of passive recovery. After IPC, performance was improved by 2.1% compared with control (95% confidence intervals of 0.8% to 3.3%, P = 0.001), followed by increases in (i) the accumulated oxygen deficit, (ii) the amplitude of blood lactate kinetics, (iii) the total amount of oxygen consumed during recovery, and (iv) the overall EMG amplitude (P < 0.05). In addition, the ratio between EMG and power output was higher during the final third of performance after IPC (P < 0.05). These results suggest an increased skeletal muscle activation and a higher anaerobic contribution as the ultimate responses of IPC on short-term exercise performance.

scholarly journals Is There an Optimal Ischemic-Preconditioning Dose to Improve Cycling Performance?

2018 ◽  
Vol 13 (3) ◽  
pp. 274-282 ◽  
Author(s):  
Scott Cocking ◽  
Mathew G. Wilson ◽  
David Nichols ◽  
N. Timothy Cable ◽  
Daniel J. Green ◽  
...  
Keyword(s):  
Blood Lactate ◽  
Ischemic Preconditioning ◽  
Perceived Exertion ◽  
Time Trial ◽  
Endurance Performance ◽  
Cycling Performance ◽  
Performance Outcomes ◽  
Rating Of Perceived Exertion ◽  
Cycling Time Trial ◽  
Number Of Cycles

Introduction: Ischemic preconditioning (IPC) may enhance endurance performance. No previous study has directly compared distinct IPC protocols for optimal benefit. Purpose: To determine whether a specific IPC protocol (ie, number of cycles, amount of muscle tissue, and local vs remote occlusion) elicits greater performance outcomes. Methods: Twelve cyclists performed 5 different IPC protocols 30 min before a blinded 375-kJ cycling time trial (TT) in a laboratory. Responses to traditional IPC (4 × 5-min legs) were compared with those to 8 × 5-min legs and sham (dose cycles), 4 × 5-min unilateral legs (dose tissue), and 4 × 5-min arms (remote). Rating of perceived exertion and blood lactate were recorded at each 25% TT completion. Power (W), heart rate (beats/min), and oxygen uptake () (mL · kg−1 · min−1) were measured continuously throughout TTs. Magnitude-based-inference statistics were employed to compare variable differences to the minimal practically important difference. Results: Traditional IPC was associated with a 17-s (0, 34) faster TT time than sham. Applying more dose cycles (8 × 5 min) had no impact on performance. Traditional IPC was associated with likely trivial higher blood lactate and possibly beneficial lower responses vs sham. Unilateral IPC was associated with 18-s (−11, 48) slower performance than bilateral (dose tissue). TT times after remote and local IPC were not different (0 [−16, 16] s). Conclusion: The traditional 4 × 5-min (local or remote) IPC stimulus resulted in the fastest TT time compared with sham; there was no benefit of applying a greater number of cycles or employing unilateral IPC.

scholarly journals The Ingestion of 39 or 64 g·hr−1 of Carbohydrate is Equally Effective at Improving Endurance Exercise Performance in Cyclists

2015 ◽  
Vol 25 (3) ◽  
pp. 285-292 ◽  
Author(s):  
Michael L. Newell ◽  
Angus M. Hunter ◽  
Claire Lawrence ◽  
Kevin D. Tipton ◽  
Stuart D. R. Galloway
Keyword(s):  
Power Output ◽  
Statistical Significance ◽  
Time Trial ◽  
Cycling Performance ◽  
Constant Load ◽  
Peak Power Output ◽  
Task Completion ◽  
Intake Rate ◽  
Mean Power ◽  
Pacing Strategy

In an investigator-blind, randomized cross-over design, male cyclists (mean± SD) age 34.0 (± 10.2) years, body mass 74.6 (±7.9) kg, stature 178.3 (±8.0) cm, peak power output (PPO) 393 (±36) W, and VO2max 62 (±9) ml·kg−1min−1 training for more than 6 hr/wk for more than 3y (n = 20) completed four experimental trials. Each trial consisted of a 2-hr constant load ride at 95% of lactate threshold (185 ± 25W) then a work-matched time trial task (~30min at 70% of PPO). Three commercially available carbohydrate (CHO) beverages, plus a control (water), were administered during the 2-hr ride providing 0, 20, 39, or 64g·hr−1 of CHO at a fluid intake rate of 1L·hr−1. Performance was assessed by time to complete the time trial task, mean power output sustained, and pacing strategy used. Mean task completion time (min:sec ± SD) for 39g·hr−1 (34:19.5 ± 03:07.1, p = .006) and 64g·hr−1 (34:11.3 ± 03:08.5 p = .004) of CHO were significantly faster than control (37:01.9 ± 05:35.0). The mean percentage improvement from control was −6.1% (95% CI: −11.3 to −1.0) and −6.5% (95% CI: −11.7 to −1.4) in the 39 and 64g·hr−1 trials respectively. The 20g·hr−1 (35:17.6 ± 04:16.3) treatment did not reach statistical significance compared with control (p = .126) despite a mean improvement of −3.7% (95% CI −8.8−1.5%). No further differences between CHO trials were reported. No interaction between CHO dose and pacing strategy occurred. 39 and 64g·hr−1 of CHO were similarly effective at improving endurance cycling performance compared with a 0g·hr−1 control in our trained cyclists.

scholarly journals Anaerobic energy provision does not limit Wingate exercise performance in endurance-trained cyclists

2003 ◽  
Vol 94 (2) ◽  
pp. 668-676 ◽  
Author(s):  
J. A. L. Calbet ◽  
J. A. De Paz ◽  
N. Garatachea ◽  
S. Cabeza de Vaca ◽  
J. Chavarren
Keyword(s):  
Exercise Performance ◽  
Power Output ◽  
Acute Hypoxia ◽  
Lactate Concentration ◽  
Oxygen Deficit ◽  
Peak Power Output ◽  
Fatigue Index ◽  
Mean Power ◽  
Anaerobic Energy ◽  
Mean Power Output

The aim of this study was to evaluate the effects of severe acute hypoxia on exercise performance and metabolism during 30-s Wingate tests. Five endurance- (E) and five sprint- (S) trained track cyclists from the Spanish National Team performed 30-s Wingate tests in normoxia and hypoxia (inspired O2 fraction = 0.10). Oxygen deficit was estimated from submaximal cycling economy tests by use of a nonlinear model. E cyclists showed higher maximal O2 uptake than S (72 ± 1 and 62 ± 2 ml · kg−1 · min−1, P < 0.05). S cyclists achieved higher peak and mean power output, and 33% larger oxygen deficit than E ( P< 0.05). During the Wingate test in normoxia, S relied more on anaerobic energy sources than E ( P < 0.05); however, S showed a larger fatigue index in both conditions ( P < 0.05). Compared with normoxia, hypoxia lowered O2 uptake by 16% in E and S ( P < 0.05). Peak power output, fatigue index, and exercise femoral vein blood lactate concentration were not altered by hypoxia in any group. Endurance cyclists, unlike S, maintained their mean power output in hypoxia by increasing their anaerobic energy production, as shown by 7% greater oxygen deficit and 11% higher postexercise lactate concentration. In conclusion, performance during 30-s Wingate tests in severe acute hypoxia is maintained or barely reduced owing to the enhancement of the anaerobic energy release. The effect of severe acute hypoxia on supramaximal exercise performance depends on training background.

scholarly journals Acute ibuprofen ingestion does not attenuate fatigue during maximal intermittent knee extensor or all-out cycling exercise

2019 ◽  
Vol 44 (2) ◽  
pp. 208-215 ◽  
Author(s):  
Paul T. Morgan ◽  
Anni Vanhatalo ◽  
Joanna L. Bowtell ◽  
Andrew M. Jones ◽  
Stephen J. Bailey
Keyword(s):  
Exercise Performance ◽  
Power Output ◽  
Knee Extensor ◽  
Cycling Performance ◽  
Similar Rate ◽  
Cycle Ergometer ◽  
Knee Extensors ◽  
Mean Power ◽  
Healthy Humans ◽  
The Mean

Recent research suggests that acute consumption of pharmacological analgesics can improve exercise performance, but the ergogenic potential of ibuprofen (IBP) administration is poorly understood. This study tested the hypothesis that IBP administration would enhance maximal exercise performance. In one study, 13 physically active males completed 60 × 3-s maximal voluntary contractions (MVCs) of the knee extensors interspersed with 2-s passive recovery periods, on 2 occasions, with the critical torque (CT) estimated as the mean torque over the last 12 contractions (part A). In another study, 16 active males completed two 3-min all-out tests against a fixed resistance on an electronically braked cycle ergometer, with the critical power estimated from the mean power output over the final 30 s of the test (part B). All tests were completed 60 min after ingestion of maltodextrin (placebo, PL) or 400 mg of IBP. Peripheral nerve stimulation was administered at regular intervals and electromyography was measured throughout. For part A, mean torque (IBP: 60% ± 13% of pre-exercise MVC; PL: 58% ± 14% of pre-exercise MVC) and CT (IBP: 41% ± 16% of pre-exercise MVC; PL: 40% ± 15% of pre-exercise MVC) were not different between conditions (P > 0.05). For part B, end-test power output (IBP: 292 ± 28 W; PL: 288 ± 31 W) and work done (IBP: 65.9 ± 5.9 kJ; PL: 65.4 ± 6.4 kJ) during the 3-min all-out cycling tests were not different between conditions (all P > 0.05). For both studies, neuromuscular fatigue declined at a similar rate in both conditions (P > 0.05). In conclusion, acute ingestion of 400 mg of IBP does not improve single-leg or maximal cycling performance in healthy humans.

Combined Glucose Ingestion and Mouth Rinsing Improves Sprint Cycling Performance

2014 ◽  
Vol 24 (6) ◽  
pp. 605-612 ◽  
Author(s):  
Edwin Chong ◽  
Kym J. Guelfi ◽  
Paul A. Fournier
Keyword(s):  
Blood Glucose ◽  
Power Output ◽  
Cycling Performance ◽  
Cycle Ergometer ◽  
Peak Power Output ◽  
Mean Power ◽  
Glucose Ingestion ◽  
Sprint Cycling ◽  
Double Blinded ◽  
Mouth Rinsing

This study investigated whether combined ingestion and mouth rinsing with a carbohydrate solution could improve maximal sprint cycling performance. Twelve competitive male cyclists ingested 100 ml of one of the following solutions 20 min before exercise in a randomized double-blinded counterbalanced order (a) 10% glucose solution, (b) 0.05% aspartame solution, (c) 9.0% maltodextrin solution, or (d) water as a control. Fifteen min after ingestion, repeated mouth rinsing was carried out with 11 × 15 ml bolus doses of the same solution at 30-s intervals. Each participant then performed a 45-s maximal sprint effort on a cycle ergometer. Peak power output was significantly higher in response to the glucose trial (1188 ± 166 W) compared with the water (1036 ± 177 W), aspartame (1088 ± 128 W) and maltodextrin (1024 ± 202W) trials by 14.7 ± 10.6, 9.2 ± 4.6 and 16.0 ± 6.0% respectively (p < .05). Mean power output during the sprint was significantly higher in the glucose trial compared with maltodextrin (p < .05) and also tended to be higher than the water trial (p = .075). Glucose and maltodextrin resulted in a similar increase in blood glucose, and the responses of blood lactate and pH to sprinting did not differ significantly between treatments (p > .05). These findings suggest that combining the ingestion of glucose with glucose mouth rinsing improves maximal sprint performance. This ergogenic effect is unlikely to be related to changes in blood glucose, sweetness, or energy sensing mechanisms in the gastrointestinal tract.

Physiological Profile of an Uphill Time Trial in Elite Cyclists

2018 ◽  
Vol 13 (3) ◽  
pp. 268-273 ◽  
Author(s):  
Ana B. Peinado ◽  
Nuria Romero-Parra ◽  
Miguel A. Rojo-Tirado ◽  
Rocío Cupeiro ◽  
Javier Butragueño ◽  
...  
Keyword(s):  
Power Output ◽  
Perceived Exertion ◽  
Lactate Concentration ◽  
Time Trial ◽  
Cycling Performance ◽  
Mean Power ◽  
Road Cycling ◽  
And Performance ◽  
Mean Power Output ◽  
Elite Cyclists

Context: While a number of studies have researched road-cycling performance, few have attempted to investigate the physiological response in field conditions. Purpose: To describe the physiological and performance profile of an uphill time trial (TT) frequently used in cycling competitions. Methods: Fourteen elite road cyclists (mean ± SD age 25 ± 6 y, height 174 ± 4.2 cm, body mass 64.4 ± 6.1 kg, fat mass 7.48% ± 2.82%) performed a graded exercise test to exhaustion to determine maximal parameters. They then completed a field-based uphill TT in a 9.2-km first-category mountain pass with a 7.1% slope. Oxygen uptake (VO2), power output, heart rate (HR), lactate concentration, and perceived-exertion variables were measured throughout the field-based test. Results: During the uphill TT, mean power output and velocity were 302 ± 7 W (4.2 ± 0.1 W/kg) and 18.7 ± 1.6 km/h, respectively. Mean VO2 and HR were 61.6 ± 2.0 mL · kg−1 · min−1 and 178 ± 2 beats/min, respectively. Values were significantly affected by the 1st, 2nd, 6th, and final kilometers (P < .05). Lactate concentration and perceived exertion were 10.87 ± 1.12 mmol/L and 19.1 ± 0.1, respectively, at the end of the test, being significantly different from baseline measures. Conclusion: The studied uphill TT is performed at 90% of maximum HR and VO2 and 70% of maximum power output. To the authors’ knowledge, this is the first study assessing cardiorespiratory parameters combined with measures of performance, perceived exertion, and biochemical variables during a field-based uphill TT in elite cyclists.

AMP deaminase deficiency is associated with lower sprint cycling performance in healthy subjects

2007 ◽  
Vol 103 (1) ◽  
pp. 315-322 ◽  
Author(s):  
Heléne Fischer ◽  
Mona Esbjörnsson ◽  
Richard L. Sabina ◽  
Anna Strömberg ◽  
Myriam Peyrard-Janvid ◽  
...  
Keyword(s):  
Skeletal Muscle ◽  
Exercise Performance ◽  
Adenine Nucleotide ◽  
Cycling Performance ◽  
The Other ◽  
Small Subset ◽  
Amp Deaminase ◽  
Mean Power ◽  
Significant Difference ◽  
Deficient Group

AMP deaminase (AMPD) deficiency is an inherited disorder of skeletal muscle found in ∼2% of the Caucasian population. Although most AMPD-deficient individuals are asymptomatic, a small subset has exercise-related cramping and pain without any other identifiable neuromuscular complications. This heterogeneity has raised doubts about the physiological significance of AMPD in skeletal muscle, despite evidence for disrupted adenine nucleotide catabolism during exercise in deficient individuals. Previous studies have evaluated the effect of AMPD deficiency on exercise performance with mixed results. This study was designed to circumvent the perceived limitations in previous reports by measuring exercise performance during a 30-s Wingate test in 139 healthy, physically active subjects of both sexes, with different AMPD1 genotypes, including 12 AMPD-deficient subjects. Three of the deficient subjects were compound heterozygotes characterized by the common c.34C>T mutation in one allele and a newly discovered AMPD1 mutation, c.404delT, in the other. While there was no significant difference in peak power across AMPD1 genotypes, statistical analysis revealed a faster power decrease in the AMPD-deficient group and a difference in mean power across the genotypes ( P = 0.0035). This divergence was most striking at 15 s of the 30-s cycling. Assessed by the fatigue index, the decrease in power output at 15 s of exercise was significantly greater in the deficient group compared with the other genotypes ( P = 0.0006). The approximate 10% lower mean power in healthy AMPD-deficient subjects during a 30-s Wingate cycling test reveals a functional role for the AMPD1 enzyme in sprint exercise.

scholarly journals Neuromuscular Adjustments Following Sprint Training with Ischemic Preconditioning in Endurance Athletes: Preliminary Data

Sports ◽  
2021 ◽  
Vol 9 (9) ◽  
pp. 124
Author(s):  
Stéphan Bouffard ◽  
Pénélope Paradis-Deschênes ◽  
François Billaut
Keyword(s):  
Root Mean Square ◽  
Ischemic Preconditioning ◽  
Power Output ◽  
Wingate Test ◽  
Peak Power Output ◽  
Mean Square ◽  
Sprint Training ◽  
Mean Power ◽  
Root Mean Square Amplitude ◽  
Mean Power Frequency

This preliminary study examined the effect of chronic ischemic preconditioning (IPC) on neuromuscular responses to high-intensity exercise. In a parallel-group design, twelve endurance-trained males (VO2max 60.0 ± 9.1 mL·kg−1·min−1) performed a 30-s Wingate test before, during, and after 4 weeks of sprint-interval training. Training consisted of bi-weekly sessions of 4 to 7 supra-maximal all-out 30-s cycling bouts with 4.5 min of recovery, preceded by either IPC (3 × 5-min of compression at 220 mmHg/5-min reperfusion, IPC, n = 6) or placebo compressions (20 mmHg, PLA, n = 6). Mechanical indices and the root mean square and mean power frequency of the electromyographic signal from three lower-limb muscles were continuously measured during the Wingate tests. Data were averaged over six 5-s intervals and analyzed with Cohen’s effect sizes. Changes in peak power output were not different between groups. However, from mid- to post-training, IPC improved power output more than PLA in the 20 to 25-s interval (7.6 ± 10.0%, ES 0.51) and the 25 to 30-s interval (8.8 ± 11.2%, ES 0.58), as well as the fatigue index (10.0 ± 2.3%, ES 0.46). Concomitantly to this performance difference, IPC attenuated the decline in frequency spectrum throughout the Wingate (mean difference: 14.8%, ES range: 0.88–1.80). There was no difference in root mean square amplitude between groups. These preliminary results suggest that using IPC before sprint training may enhance performance during a 30-s Wingate test, and such gains occurred in the last 2 weeks of the intervention. This improvement may be due, in part, to neuromuscular adjustments induced by the chronic use of IPC.

No additive effect of acetaminophen when co-ingested with caffeine on cycling performance in well-trained young men

2021 ◽  
Author(s):  
Søren Jessen ◽  
Kasper Eibye ◽  
Peter Møller Christensen ◽  
Morten Hostrup ◽  
Jens Bangsbo
Keyword(s):  
Power Output ◽  
Performance Test ◽  
Treatment Time ◽  
Muscle Protein ◽  
Cycling Performance ◽  
Peripheral Fatigue ◽  
Mean Power ◽  
Major Mechanism ◽  
Lower Treatment ◽  
Substrate Phosphorylation

We investigated the effect of caffeine and acetaminophen on power output during a 6-min performance-test, peripheral fatigue, and muscle protein kinase A (PKA) substrate-phosphorylation. Fourteen men (age(mean±SD): 26±6 years; V̇O2max: 63.9±5.0 mL∙min-1∙kg-1) completed four randomized trials with acetaminophen (1500 mg), caffeine (5 mg∙kgbw-1), combined caffeine and acetaminophen (caffeine+acetaminophen) or placebo. Mean power output during the 6-min performance-test (placebo mean:312±41 W) was higher with caffeine (+5 W;95%CI: 1 to 9;P=0.017) and caffeine+acetaminophen (+6 W;95%CI: 0 to 12;P=0.049) than placebo, but not with acetaminophen (+1 W;95%CI: -4 to 7;P=0.529). Decline in quadriceps maximal isometric voluntary torque immediately after the performance-test was lower (treatment×time; P=0.035) with acetaminophen (-40 Nm;95%CI:-53 to -30;P<0.001) and caffeine+acetaminophen (-44 Nm;95%CI: -58 to -30;P<0.001) than placebo (-53 Nm;95%CI: -71 to -39;P<0.001) but was similar with caffeine (-54 Nm;95%CI: -69 to -38;P<0.001). Muscle phosphocreatine content decreased more during the performance-test (treatment×time;P=0.036) with caffeine+acetaminophen (-55 mmol∙kgdw-1;95%CI: -65 to -46;P<0.001) than placebo (-40 mmol∙kgdw-1;95%CI: -52 to -24;P<0.001). Muscle net lactate accumulation was not different from placebo (+85 mmol∙kgdw-1;95%CI: 60 to 110;P<0.001) for any treatment (treatment×time;P=0.066), being +75 mmol∙kgdw-1 (95%CI: 51 to 99;P<0.001) with caffeine, +76 mmol∙kgdw-1 (95%CI: 58 to 96;P<0.001) with acetaminophen, and +103 mmol∙kgdw-1 (95%CI: 89 to 115;P<0.001) with caffeine+acetaminophen. Decline in muscle ATP and glycogen content and increase in PKA substrate-phosphorylation was not different between treatments (treatment×time;P>0.1). Thus, acetaminophen provides no additive performance enhancing effect to caffeine during 6-min maximal cycling. In addition, change in PKA activity is likely not a major mechanism of performance improvement with caffeine.

Immersion with menthol improves recovery between 2 cycling exercises in hot and humid environment

2018 ◽  
Vol 43 (9) ◽  
pp. 902-908 ◽  
Author(s):  
Kévin Rinaldi ◽  
Than Tran Trong ◽  
Florence Riera ◽  
Katharina Appel ◽  
Olivier Hue
Keyword(s):  
Power Output ◽  
Perceived Exertion ◽  
Cold Water ◽  
Thermal Sensation ◽  
Water Immersion ◽  
Cycling Performance ◽  
Cold Water Immersion ◽  
Mean Power ◽  
Humid Environment ◽  
Mean Power Output

Endurance exercise performance is impaired in a hot and humid environment. This study compared the effects of cold water immersion, with (CMWI) and without (CWI) menthol, on the recovery of cycling performance. Eight heat-acclimatized cyclists (age, 24.1 ± 4.4 years; mass, 65.3 ± 5.2 kg) performed 2 randomized sessions, each consisting of a 20-min cycling trial (T1) followed by 10 min of immersion during recovery and then a second 20-min cycling trial (T2). Mean power output and perceived exertion (RPE) were recorded for both trials. Rectal (Trec) and skin temperatures were measured before and immediately after T1, immersion, and T2. Perceived thermal sensation (TS) and comfort were measured immediately after T1 and T2. Power output was significantly improved in T2 compared with T1 in the CMWI condition (+15.6%). Performance did not change in the CWI condition. After immersion, Trec was lower in CWI (–1.17 °C) than in CMWI (–0.6 °C). TS decreased significantly after immersion in both conditions. This decline was significantly more pronounced in CMWI (5.9 ± 1 to 3.6 ± 0.5) than in CWI (5.6 ± 0.9 to 4.4 ± 1.2). In CMWI, RPE was significantly higher in T1 (6.57 ± 0.9) than in T2 (5.14 ± 1.25). However, there was no difference in TC. This study suggests that menthol immersion probably (i) improves the performance of a repeated 20-min cycling bout, (ii) decreases TS, and (iii) impairs thermoregulation processes.

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