Analytical Examinations of Negative Resistance Due to Double Injection Model

We report here on recent measurements made on our Pt–Cu2O–Cu diodes, annealed at an air pressure of 1 to 2 Torr, which we interpret as due to double injection, i.e. injection of holes from the platinum electrode and injection of electrons from the copper–Cu2O junction into the single crystal Cu2O region. The measurements discussed here include the forward I–V characteristics at various temperatures, current vs. thickness relationship at constant voltage, the effect of photo-memory on the I–V characteristics, and the discovery of a negative resistance regime at below room temperature at sufficiently high injection levels. The analysis of the temperature dependence of the current in the ohmic and the [Formula: see text] regimes, together with the effect of photomemory on the I–V characteristics enable us to identify the [Formula: see text] regime as the Ashley–Milnes regime with field dependent mobility.

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Avalanche Breakdown-Double Injection Induced Negative Resistance in Semiconductors

Journal of the Physical Society of Japan ◽

10.1143/jpsj.17.1729 ◽

1962 ◽

Vol 17 (11) ◽

pp. 1729-1736 ◽

Cited By ~ 25

Author(s):

M.C. Steele ◽

K. Ando ◽

M. A. Lampert

Keyword(s):

Negative Resistance ◽

Avalanche Breakdown ◽

Double Injection

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Vascular relaxation properties of calcitonin gene-related peptide and vasoactive intestinal polypeptide in subarachnoid hemorrhage

Journal of Neurosurgery ◽

10.3171/jns.1990.72.5.0792 ◽

1990 ◽

Vol 72 (5) ◽

pp. 792-797 ◽

Cited By ~ 17

Author(s):

Kazuhiko Nozaki ◽

Shinichiro Okamoto ◽

Yoshihiko Uemura ◽

Haruhiko Kikuchi ◽

Noboru Mizuno

Keyword(s):

Subarachnoid Hemorrhage ◽

Vasoactive Intestinal Polypeptide ◽

Basilar Artery ◽

Single Injection ◽

Calcitonin Gene Related Peptide ◽

Vascular Relaxation ◽

Double Injection ◽

Related Peptide ◽

Injection Model ◽

Calcitonin Gene

✓ The vascular relaxation effects of calcitonin gene-related peptide (CGRP) and vasoactive intestinal polypeptide (VIP) on the dog basilar artery after experimentally produced subarachnoid hemorrhage (SAH) were examined in vitro by an isometric tension recording method. Both CGRP and VIP induced dose-dependent relaxations in ring segments of the intact basilar artery of control dogs. The vasorelaxant action of CGRP was more potent than that of VIP. The single-injection model of SAH was produced by injection of fresh autologous arterial blood ( 1 ml/kg body weight) into the cisterna magna on Day 0 of the post-SAH period, and the double-injection model was produced by two injections of blood (0.5 ml/kg each) on Days 0 and 2. Narrowing of the basilar arteries on vertebral angiograms was most prominent on Day 3 or 7 in the single- or double-injection model, respectively. Relaxation of the basilar artery induced by CGRP and VIP was to some extent decreased on Days 3 and 7 of the post-SAH period in the single-injection model, and on Days 7 and 14 in the double-injection model. However, the vasorelaxant effects of CGRP and VIP were significantly enhanced on Day 14 of the post-SAH period in the single-injection model, and on Days 28 and 42 in the double-injection model. Subsequently, these effects returned to control levels by Days 28 or 63 in the single- or double-injection model, respectively.

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