Anticipatory, exercise, and recovery heart rates of girls as affected by four running events

1964 ◽  
Vol 19 (5) ◽  
pp. 853-856 ◽  
Author(s):  
Vera Skubic ◽  
Jane Hilgendorf

The heart rate response to running various distances was studied using five highly trained girls as subjects. A telemetering instrument was employed so that the testing could be done under actual sport conditions. The findings indicated that 1) the anticipatory heart rate just prior to exercise represented 59% of the total adjustment to exercise, 2) the heart rates during exercise were 2.5 times the resting values, and 3) heart rates observed at the end of the 220-, 440-, 880-yard, and mile events were simila cardiac function; exercise Submitted on October 22, 1963

1973 ◽  
Vol 36 (2) ◽  
pp. 639-645 ◽  
Author(s):  
G. Harley Hartung

Heart rates of 30 male Ss were monitored prior to, during, and in recovery from a standard treadmill walk. 10 Ss were trained distance runners and were considered trained in the specific work studied. 10 Ss were conditioned swimmers but were doing a type of work for which they were not trained. The runners had significantly better mean endurance times (time to heart rates 110, 130, 150, and 170) than the swimmers, even though both had previously undergone extensive endurance training. The swimmers did not perform significantly better than a group of nontrained Ss except to heart rate 130. In recovery to heart rate 130, there was no significant difference among the means of the three groups. The only significant difference among group means to final recovery heart rate was between the runners and the nontrained group.


1991 ◽  
Vol 62 (2) ◽  
pp. 43-47 ◽  
Author(s):  
A. J. Guthrie ◽  
Valerie M. Killeen ◽  
Maria S.G. Mülders ◽  
J. F.W. Grosskopf

The ratio of the cardiopulmonary blood volume to stroke volume is called the cardiopulmonary flow index (CPFI). The CPFI can be determined indirectly from the simultaneous recording of a radio cardiogram and an electrocardiogram. The CPFI and cardiac output were measured simultaneously in horses (n = 10) that were diagnosed as having cardiac disease. The diseased subjects were probably all exposed to feed contaminated with the ionophore, salinomycin, and all showed clinical signs indicative of chronic toxic myocarditis. The results obtained from these subjects were compared with those from control animals and significant differences (P 0,05) were found between the mean CPFI of the control horses and those with macroscopically visible myocardial fibrosis on post mortem examination. No significant differences were found between the means of the cardiac output measured in either of the groups of horses. The effect of pharmacological acceleration of the heart rate on the CPFI was also studied. Significant differences (P 0,05) were found between the mean CPFI and the slopes of the regression lines of CPFI on heart rate of the control and principal groups of horses. These differences were greatest at heart rates near to the resting heart rates of the individuals. The CPFI was found to be a more sensitive measure of cardiac function than cardiac output, in the horses.


1977 ◽  
Vol 42 (6) ◽  
pp. 824-832 ◽  
Author(s):  
H. L. Stone

Exercise training (T) was accomplished in 21 mongrel dogs. The animals were instrumented to measure ascending aortic flow, left ventricular pressure, and left atrial pressure. They were allowed to recover for 4 wk following surgery before accomplishing a standardized submaximal test (SMT). The exercise program alternated daily between sprint and endurance training. During T, the animals were tested while lying quietly on a laboratory table as well as during the SMT. In six animals, ventricular function curves (VFC) were obtained by rapid volume loading at similar time intervals as the SMT. Heart rate increased during the SMT but was found to be reduced in the T animals by an average of 20 beats/min. The maximum derivative of left ventricular pressure (P) increased during the SMT in T animals by an average of 2,200 Torr/s above the untrained animals. The VFC was lower in T animals than untrained animals because of a reduction in heart rate response. Results indicate a reflex adaptation of the nervous system with training to improve cardiac function.


2011 ◽  
Vol 2011 ◽  
pp. 1-10
Author(s):  
Lin Zhang ◽  
Yuan Yu ◽  
Zhen Song ◽  
Yun-Ying Wang ◽  
Zhi-Bin Yu

We hypothesized that the extent of frequency-dependent acceleration of relaxation (FDAR) would be less than that of isoproterenol-(ISO-)dependent acceleration of relaxation (IDAR) at the same increment of heart rates, and ISO may improve FDAR. Cardiac function and phosphorylation of PLB and cTnI were compared in pacing, ISO treatment, and combined pacing and ISO treatment in isolated working heart. The increase in cardiac output and the degree of relaxation was less in pacing than in ISO treatment at the same increment of heart rates. The increasing stimulation frequency induced more significant relaxant effect in ISO perfusion than that in physiological salt perfusion. The pacing only phosphorylated PLB at Thr17, but ISO induced phosphorylation of cTnI and PLB at Ser16 and Thr17. Those results suggest that the synergistic effects of PLB and cTnI induce higher degree of relaxation which makes a sufficient diastolic filling of the ventricle at higher heart rate.


1970 ◽  
Vol 27 (2) ◽  
pp. 651-658 ◽  
Author(s):  
Terry L. De Vietti ◽  
Paul B. Porter

36 rats were used to determine the effects of: (a) footshock before specific training, (b) CS duration (6 vs 60 sec.), and (c) form of aversive conditioning (CER vs CAR) on heart rate both during the presentation of the CS and between presentations. All three tested variables were effective in modifying the heart-rate response to the CS while only CS duration altered heart rates between CS presentations.


1978 ◽  
Vol 56 (5) ◽  
pp. 1207-1210 ◽  
Author(s):  
Aaron N. Moen ◽  
M. A. DellaFera ◽  
A. L. Hiller ◽  
B. A. Buxton

Weekly measurements of heart rate responses of a hand-reared white-tailed deer fawn to recorded wolf howls were completed through the age of 87 days, with occasional measurements of the responses of three other fawns. Alarm bradycardia was a very predictable initial response to the first howl through 45 days of age. Increased heart rates were observed in response to a chorus of howls 35 s into the 62-s sequence. A most interesting characteristic of the response was a highly significant (sign test) relationship between the direction of the heart rate response at 1-s intervals from one test to another, and from one week to the next, until the fawn was 45 days old. Heart rates varied from 86 to 225 beats per minute during the tests, within the ranges observed for transient stimuli such as horses, rain, machines, and other deer. The fawn remained bedded during the tests. A hiding response and alarm bradycardia in the first few weeks of life seems to be good adaptive strategy as the neonate has little chance to escape from a predator by running.


1957 ◽  
Author(s):  
Philip J. Bersh ◽  
Joseph M. Notterman ◽  
William N. Schoenfeld

1995 ◽  
Vol 133 (6) ◽  
pp. 723-728 ◽  
Author(s):  
Ettore C degli Uberti ◽  
Maria R Ambrosio ◽  
Marta Bondanelli ◽  
Giorgio Transforini ◽  
Alberto Valentini ◽  
...  

degli Uberti EC, Ambrosio MR, Bondanelli M, Trasforini G, Valentini A, Rossi R, Margutti A, Campo M. Effect of human galanin on the response of circulating catecholamines to hypoglycemia in man. Eur J Endocrinol 1995;133:723–8. ISSN 0804–4643 Human galanin (hGAL) is a neuropeptide with 30 amino acid residues that has been found in the peripheral and central nervous system, where it often co-exists with catecholamines. In order to clarify the possible role of hGAL in the regulation of sympathoadrenomedullary function, the effect of a 60 min infusion of hGAL (80 pmol·kg−1 · min−1) on plasma epinephrine and norepinephrine responses to insulin-induced hypoglycemia in nine healthy subjects was investigated. Human GAL administration significantly reduced both the release of basal norepinephrine and the response to insulin-induced hypoglycemia, whereas it attenuated the epinephrine response by 26%, with the hGAL-induced decrease in epinephrine release failing to achieve statistical significance. Human GAL significantly increased the heart rate in resting conditions and clearly exaggerated the heart rate response to insulin-induced hypoglycemia, whereas it had no effect on the blood pressure. We conclude that GAL receptor stimulation exerts an inhibitory effect on basal and insulin-induced hypoglycemia-stimulated release of norepinephrine. These findings provide further evidence that GAL may modulate sympathetic nerve activity in man but that it does not play an important role in the regulation of adrenal medullary function. Ettore C degli Uberti, Chair of Endocrinology, University of Ferrara, Via Savonarola 9, I-44100 Ferrara, Italy


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